561 results on '"Kiriazis, Helen"'
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2. Lipoxin A4 improves cardiac remodeling and function in diabetes-associated cardiac dysfunction
3. Chemokine receptor CXCR7 antagonism ameliorates cardiac and renal fibrosis induced by mineralocorticoid excess
4. Sex-specific regulation of the cardiac transcriptome by the protein phosphatase 2A regulatory subunit B55α
5. An optimized plasmalogen modulating dietary supplement provides greater protection in a male than female mouse model of dilated cardiomyopathy
6. A gene therapy targeting medium-chain acyl-CoA dehydrogenase (MCAD) did not protect against diabetes-induced cardiac pathology
7. Estrogen receptor alpha deficiency in cardiomyocytes reprograms the heart-derived extracellular vesicle proteome and induces obesity in female mice
8. Mapping the cellular and molecular landscape of cardiac non-myocytes in murine diabetic cardiomyopathy
9. Lipoxin A4 improves cardiac remodeling and function in diabetes-associated cardiac dysfunction.
10. Single–cell transcriptional and epigenetic mapping reveals cellular and molecular mechanisms driving non-ischemic cardiac fibrosis
11. Loss of the long non-coding RNA OIP5-AS1 exacerbates heart failure in a sex-specific manner
12. Cardiomyopathy characterizing and heart failure risk predicting by echocardiography and pathoanatomy in aged male mice.
13. Distinct functional and molecular profiles between physiological and pathological atrial enlargement offer potential new therapeutic opportunities for atrial fibrillation.
14. The pro‐resolving mediator, annexin A1 regulates blood pressure, and age‐associated changes in cardiovascular function and remodeling
15. Diastolic dysfunction in a pre-clinical model of diabetes is associated with changes in the cardiac non-myocyte cellular composition
16. Novel formylpeptide receptor 1/2 agonist limits hypertension-induced cardiovascular damage.
17. Deletion of the muscle enriched lncRNA Oip5os1 induces atrial dysfunction in male mice with diabetes
18. A gene therapy targeting medium-chain acyl-CoA dehydrogenase (MCAD) did not protect against diabetes-induced cardiac pathology
19. Lipidomic Profiles of the Heart and Circulation in Response to Exercise versus Cardiac Pathology: A Resource of Potential Biomarkers and Drug Targets
20. Distinct lipidomic profiles in models of physiological and pathological cardiac remodeling, and potential therapeutic strategies
21. Deficiency of Prebiotic Fibre and Insufficient Signalling Through Gut Metabolite Sensing Receptors Leads to Cardiovascular Disease
22. Maternal Diet and Gut Microbiota Influence Predisposition to Cardiovascular Disease in Offspring.
23. Manipulation of the gut microbiota by the use of prebiotic fibre does not override a genetic predisposition to heart failure
24. Expressing an inhibitor of PLCβ1b sustains contractile function following pressure overload
25. Relaxin mitigates microvascular damage and inflammation following cardiac ischemia–reperfusion
26. A high-sucrose diet exacerbates the left ventricular phenotype in a high fat-fed streptozotocin rat model of diabetic cardiomyopathy
27. Therapeutic targeting of oxidative stress with coenzyme Q10 counteracts exaggerated diabetic cardiomyopathy in a mouse model of diabetes with diminished PI3K(p110α) signaling
28. The atypical ‘b’ splice variant of phospholipase Cβ1 promotes cardiac contractile dysfunction
29. Spontaneous retrotransposon insertion into TNF 3′UTR causes heart valve disease and chronic polyarthritis
30. Estrogen receptor α deficiency in cardiac myocytes reprograms heart-derived extracellular vesicle proteome and induces obesity in female mice
31. CD14 inhibition mitigates left ventricular damage and dysfunction following myocardial infarction
32. Phosphoproteomic profiling of isolated murine cardiomyocytes reveals a role for protein phosphatase 2A (PP2A) regulatory subunit B55α as a regulator of β-adrenergic receptor signaling in the heart
33. Differential roles of cardiac and leukocyte derived macrophage migration inhibitory factor in inflammatory responses and cardiac remodelling post myocardial infarction
34. High-density lipoprotein delivered after myocardial infarction increases cardiac glucose uptake and function in mice
35. Mechanisms responsible for increased circulating levels of galectin-3 in cardiomyopathy and heart failure
36. In Vivo Inhibition of miR-34a Modestly Limits Cardiac Enlargement and Fibrosis in a Mouse Model with Established Type 1 Diabetes-Induced Cardiomyopathy, but Does Not Improve Diastolic Function
37. Targeting the upregulation of reactive oxygen species subsequent to hyperglycemia prevents type 1 diabetic cardiomyopathy in mice
38. Compensated hypertrophy of cardiac ventricles in aged transgenic FVB/N mice overexpressing calsequestrin
39. Abstract 19389: Restoring Cardiac Function After Ischemia-Reperfusion Injury by Targeting Peripheral Blood Mononuclear Cells to Activated Platelets
40. Abstract 17150: Restoring Impaired L-Arginine Transport Can Attenuate Renal Fibrosis and Inflammation in a Mouse Model of Dilated Cardiomyopathy
41. Abstract 14223: N-acetylcysteine Reverses Established Renal Fibrosis and Restores Renal Function in a Mouse Model of Dilated Cardiomyopathy
42. Insulin replacement limits progression of diabetic cardiomyopathy in the low-dose streptozotocin-induced diabetic rat
43. Therapeutic inhibition of the miR-34 family attenuates pathological cardiac remodeling and improves heart function
44. Deletion of macrophage migration inhibitory factor protects the heart from severe ischemia–reperfusion injury: A predominant role of anti-inflammation
45. Relaxin remodels fibrotic healing following myocardial infarction
46. Maternal diet and gut microbiota influence predisposition to cardiovascular disease in the offspring
47. Tissue-specific expression of Cas9 has no impact on whole-body metabolism in four transgenic mouse lines
48. Infarct size and post-infarct inflammation determine the risk of cardiac rupture in mice
49. Inhibition of miR-154 Protects Against Cardiac Dysfunction and Fibrosis in a Mouse Model of Pressure Overload
50. Augmented endothelial l-arginine transport ameliorates pressure-overload-induced cardiac hypertrophy
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