47 results on '"Klinger, Bertram"'
Search Results
2. Up-regulated transcriptional regulators in mutant RAS gene signatures: a time-resolved multi-omics study in generic epithelial cell models
3. Spike-in enhanced phosphoproteomics uncovers synergistic signaling responses to MEK inhibition in colon cancer cells
4. Quantitative modelling of signaling in aggressive B cell lymphoma unveils conserved core network
5. Early senescence and production of senescence-associated cytokines are major determinants of radioresistance in head-and-neck squamous cell carcinoma
6. Modeling unveils sex differences of signaling networks in mouse embryonic stem cells
7. Neuronal activity regulates alternative exon usage
8. Reduced replication origin licensing selectively kills KRAS-mutant colorectal cancer cells via mitotic catastrophe
9. Cell type-dependent differential activation of ERK by oncogenic KRAS in colon cancer and intestinal epithelium
10. Neuroblastoma signalling models unveil combination therapies targeting feedback-mediated resistance
11. Mutation-specific effects of NRAS oncogenes in colorectal cancer cells
12. Additional file 15 of Neuronal activity regulates alternative exon usage
13. Additional file 2 of Neuronal activity regulates alternative exon usage
14. Additional file 7 of Neuronal activity regulates alternative exon usage
15. Additional file 3 of Neuronal activity regulates alternative exon usage
16. Additional file 4 of Neuronal activity regulates alternative exon usage
17. Additional file 8 of Neuronal activity regulates alternative exon usage
18. Additional file 9 of Neuronal activity regulates alternative exon usage
19. Additional file 5 of Neuronal activity regulates alternative exon usage
20. Additional file 6 of Neuronal activity regulates alternative exon usage
21. SFPQ Depletion Is Synthetically Lethal with BRAFV600E in Colorectal Cancer Cells
22. SPEED2: inferring upstream pathway activity from differential gene expression
23. Discovering causal signaling pathways through gene-expression patterns
24. The cancer cell proteome and transcriptome predicts sensitivity to targeted and cytotoxic drugs
25. Isoform-specific Ras signaling is growth factor dependent
26. Reverse engineering gene regulatory networks by modular response analysis – a benchmark
27. Comparative Network Reconstruction using mixed integer programming
28. Modelling signalling networks from perturbation data
29. Cell type-dependent differential activation of ERK by oncogenic KRAS in colon cancer and intestinal epithelium
30. Perturbation-response genes reveal signaling footprints in cancer gene expression
31. Abstract A20: Depletion of replication factor MCM7 is synthetically lethal to oncogenic KRAS expression
32. An immediate–late gene expression module decodes ERK signal duration
33. Drug Resistance Mechanisms in Colorectal Cancer Dissected with Cell Type–Specific Dynamic Logic Models
34. Dissecting cancer resistance to therapies with cell-type-specific dynamic logic models
35. Perturbation-response genes reveal signaling footprints in cancer gene expression
36. Annexin A1 sustains tumor metabolism and cellular proliferation upon stable loss of HIF1A
37. Hypoxia-induced gene expression results from selective mRNA partitioning to the endoplasmic reticulum
38. Computer-assisted curation of a human regulatory core network from the biological literature
39. Consequences of feedback in signal transduction for targeted therapies
40. Network quantification of EGFR signaling unveils potential for targeted combination therapy
41. Multilevel regulation of HIF-1 signaling by TTP
42. Reverse engineering a hierarchical regulatory network downstream of oncogenic KRAS
43. Computer-assisted curation of a human regulatory core network from the biological literature.
44. SFPQ Depletion Is Synthetically Lethal with BRAFV600Ein Colorectal Cancer Cells
45. Perturbation-response genes reveal signaling footprints in cancer gene expression
46. SFPQ Depletion Is Synthetically Lethal with BRAF V600E in Colorectal Cancer Cells.
47. Annexin A1 sustains tumor metabolism and cellular proliferation upon stable loss of HIF1A.
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