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1. Deep phenotyping of p.(V142I)‐associated variant ATTR amyloid cardiomyopathy: distinct from wild‐type ATTR amyloidosis?

Author

Razvi, Yousuf, primary, Ioannou, Adam, additional, Patel, Rishi K, additional, Chacko, Liza, additional, Karia, Nina, additional, Riefolo, Mattia, additional, Porcari, Aldostefano, additional, Rauf, Muhammad Umaid, additional, Starr, Neasa, additional, Ganesananthan, Sashiananthan, additional, Blakeney, Iona, additional, Kaza, Nandita, additional, Filisetti, Stefano, additional, Bolhuis, Roos Eline, additional, Rowczenio, Dorota, additional, Gilbertson, Janet, additional, Hutt, David, additional, Mahmood, Shameem, additional, Lachmann, Helen J, additional, Wechalekar, Ashutosh D, additional, Kotecha, Tushar, additional, Knight, Daniel S, additional, Coghlan, John G, additional, Petrie, Aviva, additional, Whelan, Carol J, additional, Venneri, Lucia, additional, Martinez‐Naharro, Ana, additional, Hawkins, Phillip, additional, Fontana, Marianna, additional, and Gillmore, Julian D, additional

Published
2023
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6. Myocardial Edema and Prognosis in Amyloidosis

Author

Kotecha, Tushar, Martinez-Naharro, Ana, Treibel, Thomas A., Francis, Rohin, Nordin, Sabrina, Abdel-Gadir, Amna, Knight, Daniel S., Zumbo, Giulia, Rosmini, Stefania, Maestrini, Viviana, Bulluck, Heerajnarain, Rakhit, Roby D., Wechalekar, Ashutosh D., Gilbertson, Janet, Sheppard, Mary N., Kellman, Peter, Gillmore, Julian D., Moon, James C., Hawkins, Philip N., and Fontana, Marianna

Published
2018
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7. Conventional heart failure therapy in cardiac ATTR amyloidosis

Author

Ioannou, Adam, primary, Massa, Paolo, additional, Patel, Rishi K, additional, Razvi, Yousuf, additional, Porcari, Aldostefano, additional, Rauf, Muhammad U, additional, Jiang, Anita, additional, Cabras, Giacomo, additional, Filisetti, Stefano, additional, Bolhuis, Roos E, additional, Bandera, Francesco, additional, Venneri, Lucia, additional, Martinez-Naharro, Ana, additional, Law, Steven, additional, Kotecha, Tushar, additional, Virsinskaite, Ruta, additional, Knight, Daniel S, additional, Emdin, Michele, additional, Petrie, Aviva, additional, Lachmann, Helen, additional, Wechelakar, Ashutosh, additional, Petrie, Mark, additional, Hughes, Alun, additional, Freemantle, Nick, additional, Hawkins, Philip N, additional, Whelan, Carol, additional, McMurray, John J V, additional, Gillmore, Julian D, additional, and Fontana, Marianna, additional

Published
2023
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8. Recurrent episodes of Takotsubo cardiomyopathy in systemic sclerosis

Author

Vreeburg, Yumiko L, Knight, Daniel S, Coghlan, John G, Ong, Voon H, and Denton, Christopher P

Abstract

Systemic sclerosis is an autoimmune disease characterized by fibrosis and small vessel vasculopathy, which affects various organ systems, such as the heart. Takotsubo cardiomyopathy is a transient cardiomyopathy in reaction to an emotional or physical trigger. There may be clinical and pathogenetic overlap between Takotsubo cardiomyopathy and primary systemic sclerosis heart disease, and some patients with systemic sclerosis have been diagnosed with recurrent Takotsubo cardiomyopathy. Our large systemic sclerosis clinical cohort was reviewed to identify cases diagnosed with Takotsubo cardiomyopathy. The clinical features, laboratory and imaging results were reviewed and evaluated to perform a comparison between cases. We identified five patients with systemic sclerosis, all female (age 68.6 ± 5.7 years), who were diagnosed with Takotsubo cardiomyopathy. Two of these patients had recurrent episodes: one case with a history of multiple episodes and the other with one recurrence. Typical features included repolarization abnormalities on the electrocardiogram and transient left ventricular dysfunction observed using echocardiography or cardiac magnetic resonance imaging. Our findings build upon previous reports and observations that systemic sclerosis may cause Takotsubo cardiomyopathy. To our knowledge, this is the largest case series of Takotsubo syndrome in patients with systemic sclerosis. This association may provide novel insights into the aetiopathogenesis of Takotsubo cardiomyopathy as part of primary systemic sclerosis heart involvement.

Published
2024
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10. COVID-19: Myocardial Injury in Survivors

Author

Knight, Daniel S., Kotecha, Tushar, Razvi, Yousuf, Chacko, Liza, Brown, James T., Jeetley, Paramjit S., Goldring, James, Jacobs, Michael, Lamb, Lucy E., Negus, Rupert, Wolff, Anthony, Moon, James C., Xue, Hui, Kellman, Peter, Patel, Niket, and Fontana, Marianna

Published
2020
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13. Cardiovascular magnetic resonance in light-chain amyloidosis to guide treatment

Author

Martinez-Naharro, Ana, primary, Patel, Rishi, additional, Kotecha, Tushar, additional, Karia, Nina, additional, Ioannou, Adam, additional, Petrie, Aviva, additional, Chacko, Liza A, additional, Razvi, Yousuf, additional, Ravichandran, Sriram, additional, Brown, James, additional, Law, Steven, additional, Quarta, Cristina, additional, Mahmood, Shameem, additional, Wisniowski, Brendan, additional, Pica, Silvia, additional, Sachchithanantham, Sajitha, additional, Lachmann, Helen J, additional, Moon, James C, additional, Knight, Daniel S, additional, Whelan, Carol, additional, Venneri, Lucia, additional, Xue, Hui, additional, Kellman, Peter, additional, Gillmore, Julian D, additional, Hawkins, Philip N, additional, Wechalekar, Ashutosh D, additional, and Fontana, Marianna, additional

Published
2022
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14. Distinct cardiovascular phenotypes are associated with prognosis in systemic sclerosis: a cardiovascular magnetic resonance study

Author

Knight, Daniel S, primary, Karia, Nina, additional, Cole, Alice R, additional, Maclean, Rory H, additional, Brown, James T, additional, Masi, Ambra, additional, Patel, Rishi K, additional, Razvi, Yousuf, additional, Chacko, Liza, additional, Venneri, Lucia, additional, Kotecha, Tushar, additional, Martinez-Naharro, Ana, additional, Kellman, Peter, additional, Scott-Russell, Ann M, additional, Schreiber, Benjamin E, additional, Ong, Voon H, additional, Denton, Christopher P, additional, Fontana, Marianna, additional, Coghlan, J Gerry, additional, and Muthurangu, Vivek, additional

Published
2022
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15. Distinct cardiovascular phenotypes are associated with prognosis in systemic sclerosis: a cardiovascular magnetic resonance study.

Author

Knight, Daniel S, Karia, Nina, Cole, Alice R, Maclean, Rory H, Brown, James T, Masi, Ambra, Patel, Rishi K, Razvi, Yousuf, Chacko, Liza, Venneri, Lucia, Kotecha, Tushar, Martinez-Naharro, Ana, Kellman, Peter, Scott-Russell, Ann M, Schreiber, Benjamin E, Ong, Voon H, Denton, Christopher P, Fontana, Marianna, Coghlan, J Gerry, and Muthurangu, Vivek

Subjects
MORTALITY risk factors, DECISION trees, PATIENT aftercare, HYPERTENSION, CAUSES of death, SCIENTIFIC observation, VENTRICULAR ejection fraction, SYSTEMIC scleroderma, MAGNETIC resonance imaging, RETROSPECTIVE studies, INDIVIDUALIZED medicine, RISK assessment, RIGHT ventricular dysfunction, DESCRIPTIVE statistics, CLUSTER analysis (Statistics), PREDICTION models, PHENOTYPES, CARDIOVASCULAR disease diagnosis, ALGORITHMS
Abstract

Aims Cardiovascular involvement in systemic sclerosis (SSc) is heterogeneous and ill-defined. This study aimed to: (i) discover cardiac phenotypes in SSc by cardiovascular magnetic resonance (CMR); (ii) provide a CMR-based algorithm for phenotypic classification; and (iii) examine for associations between phenotypes and mortality. Methods and results A retrospective, single-centre, observational study of 260 SSc patients who underwent clinically indicated CMR including native myocardial T1 and T2 mapping from 2016 to 2019 was performed. Agglomerative hierarchical clustering using only CMR variables revealed five clusters of SSc patients with shared CMR characteristics: dilated right hearts with right ventricular failure (RVF); biventricular failure dilatation and dysfunction (BVF); and normal function with average cavity (NF-AC), normal function with small cavity (NF-SC), and normal function with large cavity (NF-LC) sizes. Phenotypes did not co-segregate with clinical or antibody classifications. A CMR-based decision tree for phenotype classification was created. Sixty-three (24%) patients died during a median follow-up period of 3.4 years. After adjustment for age and presence of pulmonary hypertension (PH), independent CMR predictors of all-cause mortality were native T1 (P < 0.001) and right ventricular ejection fraction (RVEF) (P = 0.0032). NF-SC and NF-AC groups had more favourable prognoses (P ≤0.036) than the other three groups which had no differences in prognoses between them (P > 0.14). Hazard ratios (HR) were statistically significant for RVF (HR = 8.9, P < 0.001), BVF (HR = 5.2, P = 0.006), and NF-LC (HR = 4.9, P = 0.002) groups. The NF-LC group remained significantly predictive of mortality after adjusting for RVEF, native T1, and PH diagnosis (P = 0.0046). Conclusion We identified five CMR-defined cardiac SSc phenotypes that did not co-segregate with clinical data and had distinct outcomes, offering opportunities for a more precision-medicine based management approach. [ABSTRACT FROM AUTHOR]

Published
2023
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16. Ongoing Exercise Intolerance Following COVID‐19: A Magnetic Resonance–Augmented Cardiopulmonary Exercise Test Study

Author

Brown, James T., primary, Saigal, Anita, additional, Karia, Nina, additional, Patel, Rishi K., additional, Razvi, Yousuf, additional, Constantinou, Natalie, additional, Steeden, Jennifer A., additional, Mandal, Swapna, additional, Kotecha, Tushar, additional, Fontana, Marianna, additional, Goldring, James, additional, Muthurangu, Vivek, additional, and Knight, Daniel S., additional

Published
2022
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17. Non-invasive characterization of pleural and pericardial effusions using T1 mapping by magnetic resonance imaging

Author

Rosmini, Stefania, primary, Seraphim, Andreas, additional, Knott, Kristopher, additional, Brown, James T, additional, Knight, Daniel S, additional, Zaman, Sameer, additional, Cole, Graham, additional, Sado, Daniel, additional, Captur, Gabriella, additional, Gomes, Ana Caterina, additional, Zemrak, Filip, additional, Treibel, Thomas A, additional, Cash, Lizette, additional, Culotta, Veronica, additional, O’Mahony, Constantinos, additional, Kellman, Peter, additional, Moon, James C, additional, and Manisty, Charlotte, additional

Published
2021
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21. Non-invasive characterization of pleural and pericardial effusions using T1 mapping by magnetic resonance imaging.

Author

Rosmini, Stefania, Seraphim, Andreas, Knott, Kristopher, Brown, James T, Knight, Daniel S, Zaman, Sameer, Cole, Graham, Sado, Daniel, Captur, Gabriella, Gomes, Ana Caterina, Zemrak, Filip, Treibel, Thomas A, Cash, Lizette, Culotta, Veronica, O'Mahony, Constantinos, Kellman, Peter, Moon, James C, and Manisty, Charlotte

Subjects
ALBUMINS, BIOCHEMISTRY, PLEURAL effusions, CONFIDENCE intervals, PERICARDIAL effusion, MAGNETIC resonance imaging, DIFFERENTIAL diagnosis, RETROSPECTIVE studies, EXUDATES & transudates, STATISTICAL correlation, SENSITIVITY & specificity (Statistics), HEART failure
Abstract

Aims Differentiating exudative from transudative effusions is clinically important and is currently performed via biochemical analysis of invasively obtained samples using Light's criteria. Diagnostic performance is however limited. Biochemical composition can be measured with T1 mapping using cardiovascular magnetic resonance (CMR) and hence may offer diagnostic utility for assessment of effusions. Methods and results A phantom consisting of serially diluted human albumin solutions (25–200 g/L) was constructed and scanned at 1.5 T to derive the relationship between fluid T1 values and fluid albumin concentration. Native T1 values of pleural and pericardial effusions from 86 patients undergoing clinical CMR studies retrospectively analysed at four tertiary centres. Effusions were classified using Light's criteria where biochemical data was available (n  = 55) or clinically in decompensated heart failure patients with presumed transudative effusions (n = 31). Fluid T1 and protein values were inversely correlated both in the phantom (r = −0.992) and clinical samples (r = −0.663, P  < 0.0001). T1 values were lower in exudative compared to transudative pleural (3252 ± 207 ms vs. 3596 ± 213 ms, P  < 0.0001) and pericardial (2749 ± 373 ms vs. 3337 ± 245 ms, P  < 0.0001) effusions. The diagnostic accuracy of T1 mapping for detecting transudates was very good for pleural and excellent for pericardial effusions, respectively [area under the curve 0.88, (95% CI 0.764–0.996), P  = 0.001, 79% sensitivity, 89% specificity, and 0.93, (95% CI 0.855–1.000), P  < 0.0001, 95% sensitivity; 81% specificity]. Conclusion Native T1 values of effusions measured using CMR correlate well with protein concentrations and may be helpful for discriminating between transudates and exudates. This may help focus the requirement for invasive diagnostic sampling, avoiding unnecessary intervention in patients with unequivocal transudative effusions. [ABSTRACT FROM AUTHOR]

Published
2022
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24. Cardiac Magnetic Resonance–Derived Extracellular Volume Mapping for the Quantification of Hepatic and Splenic Amyloid

Author

Chacko, Liza, primary, Boldrini, Michele, additional, Martone, Raffaele, additional, Law, Steven, additional, Martinez-Naharrro, Ana, additional, Hutt, David F., additional, Kotecha, Tushar, additional, Patel, Rishi K., additional, Razvi, Yousuf, additional, Rezk, Tamer, additional, Cohen, Oliver C., additional, Brown, James T., additional, Srikantharajah, Mukunthan, additional, Ganesananthan, Sharmananthan, additional, Lane, Thirusha, additional, Lachmann, Helen J., additional, Wechalekar, Ashutosh D., additional, Sachchithanantham, Sajitha, additional, Mahmood, Shameem, additional, Whelan, Carol J., additional, Knight, Daniel S., additional, Moon, James C., additional, Kellman, Peter, additional, Gillmore, Julian D., additional, Hawkins, Philip N., additional, and Fontana, Marianna, additional

Published
2021
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27. Patterns of myocardial injury in recovered troponin-positive COVID-19 patients assessed by cardiovascular magnetic resonance

Author

Kotecha, Tushar, primary, Knight, Daniel S, additional, Razvi, Yousuf, additional, Kumar, Kartik, additional, Vimalesvaran, Kavitha, additional, Thornton, George, additional, Patel, Rishi, additional, Chacko, Liza, additional, Brown, James T, additional, Coyle, Clare, additional, Leith, Donald, additional, Shetye, Abhishek, additional, Ariff, Ben, additional, Bell, Robert, additional, Captur, Gabriella, additional, Coleman, Meg, additional, Goldring, James, additional, Gopalan, Deepa, additional, Heightman, Melissa, additional, Hillman, Toby, additional, Howard, Luke, additional, Jacobs, Michael, additional, Jeetley, Paramjit S, additional, Kanagaratnam, Prapa, additional, Kon, Onn Min, additional, Lamb, Lucy E, additional, Manisty, Charlotte H, additional, Mathurdas, Palmira, additional, Mayet, Jamil, additional, Negus, Rupert, additional, Patel, Niket, additional, Pierce, Iain, additional, Russell, Georgina, additional, Wolff, Anthony, additional, Xue, Hui, additional, Kellman, Peter, additional, Moon, James C, additional, Treibel, Thomas A, additional, Cole, Graham D, additional, and Fontana, Marianna, additional

Published
2021
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28. COVID-19

Author

Knight, Daniel S., primary, Kotecha, Tushar, additional, Razvi, Yousuf, additional, Chacko, Liza, additional, Brown, James T., additional, Jeetley, Paramjit S., additional, Goldring, James, additional, Jacobs, Michael, additional, Lamb, Lucy E., additional, Negus, Rupert, additional, Wolff, Anthony, additional, Moon, James C., additional, Xue, Hui, additional, Kellman, Peter, additional, Patel, Niket, additional, and Fontana, Marianna, additional

Published
2020
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29. The Authors Reply

Author

Baggiano, Andrea, primary, Boldrini, Michele, additional, Martinez-Naharro, Ana, additional, Kotecha, Tushar, additional, Petrie, Aviva, additional, Rezk, Tamer, additional, Gritti, Maurizio, additional, Quarta, Cristina, additional, Knight, Daniel S., additional, Wechalekar, Ashutosh D., additional, Lachmann, Helen J., additional, Perlini, Stefano, additional, Pontone, Gianluca, additional, Moon, James C., additional, Kellman, Peter, additional, Gillmore, Julian D., additional, Hawkins, Philip N., additional, and Fontana, Marianna, additional

Published
2020
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30. Noncontrast Magnetic Resonance for the Diagnosis of Cardiac Amyloidosis

Author

Baggiano, Andrea, primary, Boldrini, Michele, additional, Martinez-Naharro, Ana, additional, Kotecha, Tushar, additional, Petrie, Aviva, additional, Rezk, Tamer, additional, Gritti, Maurizio, additional, Quarta, Cristina, additional, Knight, Daniel S., additional, Wechalekar, Ashutosh D., additional, Lachmann, Helen J., additional, Perlini, Stefano, additional, Pontone, Gianluca, additional, Moon, James C., additional, Kellman, Peter, additional, Gillmore, Julian D., additional, Hawkins, Philip N., additional, and Fontana, Marianna, additional

Published
2020
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31. Anakinra treats fulminant myocarditis from Neisseria meningitidis septicaemia and haemophagocytic lymphohistiocytosis: a case report.

Author

Thomson, Ross J, Singh, Animesh, Knight, Daniel S, Buckley, Jim, Lamb, Lucy E, and Captur, Gabriella

Subjects
ANAKINRA, MYOCARDITIS, SEPSIS
Abstract

Background Fulminant myocarditis is a life-threatening condition characterized by acute cardiac dysfunction requiring pharmacological or mechanical circulatory support. Haemophagocytic lymphohistiocytosis (HLH) is an uncommon state of immune dysregulation and overactivation. Inflammation mediated by interleukin-1 (IL-1) is thought to play a role in the pathogenesis of myocarditis and HLH, and there is some evidence that the IL-1 receptor antagonist Anakinra may play a role in treating both these conditions. Case summary A 26-year-old previously healthy male presented to the Emergency Department with a 3-day history of malaise, headache, vomiting, diarrhoea, and fever. He was profoundly hypotensive on arrival, diagnosed with septic shock, and commenced on broad-spectrum antibiotics and vasopressors. Blood tests showed lymphopenia, thrombocytopenia, low fibrinogen and elevated high sensitivity troponin T, ferritin, and C-reactive protein. Echocardiography demonstrated severely impaired biventricular systolic function and a diagnosis of fulminant myocarditis was made. His condition deteriorated and he required intubation and additional inotropic support. A diagnosis of HLH was made and he was commenced on Anakinra and Methylprednisolone. His condition improved rapidly thereafter. Polymerase chain reaction testing subsequently confirmed infection with Neisseria meningitidis. Discussion In this case, fulminant myocarditis and HLH were life-threatening manifestations of meningococcal septicaemia, and the patient's condition improved rapidly following administration of the IL-1 receptor antagonist Anakinra. These complications should be borne in mind in septic patients with marked haemodynamic instability and multiorgan dysfunction, and treatment with Anakinra should be considered in those who fail to respond to conventional therapy. [ABSTRACT FROM AUTHOR]

Published
2021
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32. Native T1 and Extracellular Volume in Transthyretin Amyloidosis

Author

Martinez-Naharro, Ana, primary, Kotecha, Tushar, additional, Norrington, Karl, additional, Boldrini, Michele, additional, Rezk, Tamer, additional, Quarta, Candida, additional, Treibel, Thomas A., additional, Whelan, Carol J., additional, Knight, Daniel S., additional, Kellman, Peter, additional, Ruberg, Frederick L., additional, Gillmore, Julian D., additional, Moon, James C., additional, Hawkins, Philip N., additional, and Fontana, Marianna, additional

Published
2019
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33. Cardiac Structural and Functional Consequences of Amyloid Deposition by Cardiac Magnetic Resonance and Echocardiography and Their Prognostic Roles

Author

Knight, Daniel S., primary, Zumbo, Giulia, additional, Barcella, William, additional, Steeden, Jennifer A., additional, Muthurangu, Vivek, additional, Martinez-Naharro, Ana, additional, Treibel, Thomas A., additional, Abdel-Gadir, Amna, additional, Bulluck, Heerajnarain, additional, Kotecha, Tushar, additional, Francis, Rohin, additional, Rezk, Tamer, additional, Quarta, Candida C., additional, Whelan, Carol J., additional, Lachmann, Helen J., additional, Wechalekar, Ashutosh D., additional, Gillmore, Julian D., additional, Moon, James C., additional, Hawkins, Philip N., additional, and Fontana, Marianna, additional

Published
2019
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34. Redefining viability by cardiovascular magnetic resonance in acute ST-segment elevation myocardial infarction

Author

Bulluck, Heerajnarain, Rosmini, Stefania, Abdel-Gadir, Amna, Bhuva, Anish N., Treibel, Thomas A., Fontana, Marianna, Knight, Daniel S., Nordin, Sabrina, Sirker, Alex, Herrey, Anna S., Manisty, Charlotte, Moon, James C., and Hausenloy, Derek J.

Subjects
Male, lcsh:R, lcsh:Medicine, Middle Aged, Magnetic Resonance Imaging, Sensitivity and Specificity, Article, Percutaneous Coronary Intervention, Humans, ST Elevation Myocardial Infarction, lcsh:Q, Female, cardiovascular diseases, Coronary Artery Bypass, lcsh:Science
Abstract

In chronic myocardial infarction (MI), segments with a transmural extent of infarct (TEI) of ≤50% are defined as being viable. However, in the acute phase of an ST-segment elevation myocardial infarction (STEMI), late gadolinium enhancement (LGE) has been demonstrated to overestimate MI size and TEI. We aimed to identify the optimal cut-off of TEI by cardiovascular magnetic resonance (CMR) for defining viability during the acute phase of an MI, using ≤50% TEI at follow-up as the reference standard. 40 STEMI patients reperfused by primary percutaneous coronary intervention (PPCI) underwent a CMR at 4 ± 2 days and 5 ± 2 months. The large majority of segments with 1–25%TEI and 26–50%TEI that were viable acutely were also viable at follow-up (59/59, 100% and 75/82, 96% viable respectively). 56/84(67%) segments with 51–75%TEI but only 4/63(6%) segments with 76–100%TEI were reclassified as viable at follow-up. TEI on the acute CMR scan had an area-under-the-curve of 0.87 (95% confidence interval of 0.82 to 0.91) and ≤75%TEI had a sensitivity of 98% but a specificity of 66% to predict viability at follow-up. Therefore, the optimal cut-off by CMR during the acute phase of an MI to predict viability was ≤75% TEI and this would have important implications for patients undergoing viability testing prior to revascularization during the acute phase.

Published
2017

37. CMR-Verified Regression of Cardiac AL Amyloid After Chemotherapy

Author

Martinez-Naharro, Ana, primary, Abdel-Gadir, Amna, additional, Treibel, Thomas A., additional, Zumbo, Giulia, additional, Knight, Daniel S., additional, Rosmini, Stefania, additional, Lane, Thirusha, additional, Mahmood, Shameem, additional, Sachchithanantham, Sajitha, additional, Whelan, Carol J., additional, Lachmann, Helen J., additional, Wechalekar, Ashutosh D., additional, Kellman, Peter, additional, Gillmore, Julian D., additional, Moon, James C., additional, Hawkins, Philip N., additional, and Fontana, Marianna, additional

Published
2018
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38. Prospective comparison of novel dark blood late gadolinium enhancement with conventional bright blood imaging for the detection of scar

Author

Francis, Rohin, primary, Kellman, Peter, additional, Kotecha, Tushar, additional, Baggiano, Andrea, additional, Norrington, Karl, additional, Martinez-Naharro, Ana, additional, Nordin, Sabrina, additional, Knight, Daniel S., additional, Rakhit, Roby D., additional, Lockie, Tim, additional, Hawkins, Philip N., additional, Moon, James C., additional, Hausenloy, Derek J., additional, Xue, Hui, additional, Hansen, Michael S., additional, and Fontana, Marianna, additional

Published
2017
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40. Quantification of both the area-at-risk and acute myocardial infarct size in ST-segment elevation myocardial infarction using T1-mapping

Author

Bulluck, Heerajnarain, primary, Hammond-Haley, Matthew, additional, Fontana, Marianna, additional, Knight, Daniel S., additional, Sirker, Alex, additional, Herrey, Anna S., additional, Manisty, Charlotte, additional, Kellman, Peter, additional, Moon, James C., additional, and Hausenloy, Derek J., additional

Published
2017
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41. Full left ventricular coverage is essential for the accurate quantification of the area-at-risk by T1 and T2 mapping

Author

Bulluck, Heerajnarain, primary, Bryant, Jennifer A., additional, Lim, Mei Xing, additional, Tan, Xiao Wei, additional, Ramlall, Manish, additional, Francis, Rohin, additional, Kotecha, Tushar, additional, Cabrera-Fuentes, Hector A., additional, Knight, Daniel S., additional, Fontana, Marianna, additional, Moon, James C., additional, and Hausenloy, Derek J., additional

Published
2017
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42. Magnetic Resonance in Transthyretin Cardiac Amyloidosis

Author

Martinez-Naharro, Ana, primary, Treibel, Thomas A., additional, Abdel-Gadir, Amna, additional, Bulluck, Heerajnarain, additional, Zumbo, Giulia, additional, Knight, Daniel S., additional, Kotecha, Tushar, additional, Francis, Rohin, additional, Hutt, David F., additional, Rezk, Tamer, additional, Rosmini, Stefania, additional, Quarta, Candida C., additional, Whelan, Carol J., additional, Kellman, Peter, additional, Gillmore, Julian D., additional, Moon, James C., additional, Hawkins, Philip N., additional, and Fontana, Marianna, additional

Published
2017
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44. 024 Spectrum and significance of CMR findings in cardiac transthyretin amyloidosis

Author

Martinez-Naharro, Ana, primary, Treibel, Thomas A, additional, Abdel-Gadir, Amna, additional, Bulluck, Heerajnarain, additional, Zumbo, Giulia, additional, Knight, Daniel S, additional, Kotecha, Tushar, additional, Francis, Rohin, additional, Hutt, David, additional, Rezk, Tamer, additional, Rosmini, Stefania, additional, Quarta, Cristina, additional, Whelan, Carol J, additional, Kellman, Peter, additional, Gillmore, Julian D, additional, Moon, James C, additional, Hawkins, Philip N, additional, and Fontana, Marianna, additional

Published
2017
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45. 008 Demonstration of cardiac AL amyloidosis regression after succesful chemotherapy. a CMR study

Author

Martinez-Naharro, Ana, primary, Abdel-Gadir, Amna, additional, Treibel, Thomas A, additional, Zumbo, Giulia, additional, Knight, Daniel S, additional, Rosmini, Stefania, additional, Lane, RN Thirusha, additional, Mahmood, Shameem, additional, Sachchithanantham, Sajitha, additional, Whelan, Carol J, additional, Lachmann, Helen J, additional, Wechalekar, Ashutosh D, additional, Kellman, Peter, additional, Gillmore, Julian D, additional, Moon, James C, additional, Hawkins, Philip N, additional, and Fontana, Marianna, additional

Published
2017
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46. 001 Multiparametric mapping to understand pathophysiology in cardiac amyloidosis

Author

Kotecha, Tushar, primary, Martinez-Naharro, Ana, additional, Treibel, Thomas A, additional, Francis, Rohin, additional, Nordin, Sabrina, additional, Abdel-Gadir, Amna, additional, Knight, Daniel S, additional, Zumbo, Giulia, additional, Rosmini, Stefania, additional, Maestrini, Viviana, additional, Bulluck, Heerajnarain, additional, Rakhit, Roby, additional, Wechalekar, Ashutosh D, additional, Kellman, Peter, additional, Gillmore, Julian D, additional, Moon, James C, additional, Hawkins, Philip N, additional, and Fontana, Marianna, additional

Published
2017
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47. 028 Routine identification of hypoperfusion in cardiac amyloidosis by myocardial blood flow mapping

Author

Martinez-Naharro, Ana, primary, Knight, Daniel S, additional, Kotecha, Tushar, additional, Francis, Rohin, additional, Zumbo, Giulia, additional, Treibel, Thomas A, additional, Nickander, Jannike, additional, Themudo, Raquel, additional, Gillmore, Julian D, additional, Ugander, Martin, additional, Moon, James C, additional, Xue, Hui, additional, Kellman, Peter, additional, Hawkins, Philip N, additional, and Fontana, Marianna, additional

Published
2017
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