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5 results on '"Koenigs B"'

1. A $\sim$15 kpc outflow cone piercing through the halo of the blue compact metal-poor galaxy SBS0335-052

Author

Herenz, E. C., Inoue, J., Salas, H., Koenigs, B., Moya-Sierralta, C., Cannon, J. M., Hayes, M., Papaderos, P., Östlin, G., Bik, A., Reste, A. Le, Kusakabe, H., Monreal-Ibero, A., and Puschnig, J.

Subjects
Astrophysics - Astrophysics of Galaxies
Abstract

Context: Outflows from low-mass star-forming galaxies are a fundamental ingredient for models of galaxy evolution and cosmology. Aims: The onset of kpc-scale ionised filaments in the halo of the metal-poor compact dwarf SBS 0335-052E was previously not linked to an outflow. We here we investigate whether these filaments provide evidence for an outflow. Methods: We obtained new VLT/MUSE WFM and deep NRAO/VLA B-configuration 21cm data of the galaxy. The MUSE data provide morphology, kinematics, and emission line ratios H$\beta$/H$\alpha$ and [\ion{O}{iii}]$\lambda5007$/H$\alpha$ of the low surface-brightness filaments, while the VLA data deliver morphology and kinematics of the neutral gas in and around the system. Both datasets are used in concert for comparisons between the ionised and the neutral phase. Results: We report the prolongation of a lacy filamentary ionised structure up to a projected distance of 16 kpc at $\mathrm{SB}_\mathrm{H\alpha} = 1.5\times10^{-18}$erg s$^{-1}$ cm$^{-2}$arcsec$^{-2}$. The filaments exhibit unusual low H$\alpha$/H$\beta \approx 2.4$ and low [\ion{O}{iii}]/H$\alpha \sim 0.4 - 0.6$ typical of diffuse ionised gas. They are spectrally narrow ($\sim 20$ km s$^{-1}$) and exhibit no velocity sub-structure. The filaments extend outwards of the elongated \ion{H}{I} halo. On small scales the $N_\mathrm{HI}$ peak is offset from the main star-forming sites. Morphology and kinematics of \ion{H}{I} and \ion{H}{II} reveal how star-formation driven feedback interacts differently with the ionised and the neutral phase. Conclusions: We reason that the filaments are a large scale manifestation of star-formation driven feedback, namely limb-brightened edges of a giant outflow cone that protrudes through the halo of this gas-rich system. A simple toy model of such a conical-structure is found to be commensurable with the observations., Comment: Accepted version in A&A after language editing. 22 pages, 24 figures

Published
2022
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2. A ∼15 kpc outflow cone piercing through the halo of the blue compact metal-poor galaxy SBS 0335–052E

Author

Herenz, E. C., primary, Inoue, J., additional, Salas, H., additional, Koenigs, B., additional, Moya-Sierralta, C., additional, Cannon, J. M., additional, Hayes, M., additional, Papaderos, P., additional, Östlin, G., additional, Bik, A., additional, Le Reste, A., additional, Kusakabe, H., additional, Monreal-Ibero, A., additional, and Puschnig, J., additional

Published
2023
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5. Reduced myocardial sarcoplasmic reticulum Ca(2+)-ATPase protein expression in compensated primary and secondary human cardiac hypertrophy.

Author

Schotten U, Koenigs B, Rueppel M, Schoendube F, Boknik P, Schmitz W, and Hanrath P

Subjects
Adult, Calcium Channels metabolism, Cardiomegaly pathology, Cardiomegaly physiopathology, Female, Humans, Ion Transport, Male, Middle Aged, Ryanodine metabolism, Sarcoplasmic Reticulum enzymology, Sarcoplasmic Reticulum pathology, Calcium metabolism, Calcium-Transporting ATPases biosynthesis, Cardiomegaly enzymology
Abstract

Pathological intracellular calcium handling has been proposed to underlie the alterations of contractile behavior in hypertrophied myocardium. However, the myocardial protein expression of intracellular calcium transport proteins in compensated human left ventricular hypertrophy has not yet been studied. We investigated septal myocardial specimens of patients suffering from hypertrophic obstructive cardiomyopathy (n=14) or from acquired aortic valve stenosis (n=11) undergoing myectomy or aortic valve replacement, respectively. For comparison, we studied non-hypertrophied myocardium of six non-failing hearts which could not be transplanted for technical reasons. The myocardial density of the calcium release channel of the sarcoplasmic reticulum (SR) was determined by(3)H-ryanodine binding. Myocardial contents of SR Ca(2+)-ATPase, phospholamban, calsequestrin and Na(+)/Ca(2+)-exchanger were analysed by Western blot analysis. The myocardial SR calcium release channel density was not significantly different in hypertrophied and non-failing human myocardium. In both hypertrophic obstructive cardiomyopathy and in aortic valve stenosis, SR Ca(2+)-ATPase expression was reduced by about 30% compared to non-failing myocardium (P<0.05), whereas the expression of phospholamban, calsequestrin, and the Na(+)/Ca(2+)-exchanger was unchanged. The decrease of SR Ca(2+)-ATPase expression was still observable when related to its regulatory protein phospholamban or to the myosin content of the homogenates (P<0.05). Furthermore, the SR Ca(2+)-ATPase expression was inversely correlated to the septum thickness assessed by echocardiography, but not to age, cardiac index or outflow tract gradient. In primary as well as in secondary hypertrophied human myocardium, the expression of SR Ca(2+)-ATPase is reduced and inversely related to the degree of the hypertrophy. The diminished SR Ca(2+)-ATPase expression might result in reduced Ca(2+)reuptake into the SR and might contribute to altered contractile behavior in hypertrophied human myocardium., (Copyright 1999 Academic Press.)

Published
1999
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