31 results on '"Kozmin, Stanislav G."'
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2. Splitting the yeast centromere by recombination
3. Splitting the yeast centromere by recombination.
4. RNA viruses, M satellites, chromosomal killer genes, and killer/nonkiller phenotypes in the 100-genomes S. cerevisiae strains
5. A novel narnavirus is widespread in Saccharomyces cerevisiae and impacts multiple host phenotypes
6. Genetic characterization of moaB mutants of Escherichia coli
7. 2μ plasmid in Saccharomyces species and in Saccharomyces cerevisiae
8. A novel narnavirus is widespread in Saccharomyces cerevisiae and impacts multiple host phenotypes.
9. Dissecting Highly Mutagenic Processing of Complex Clustered DNA Damage in Yeast Saccharomyces cerevisiae
10. Role for CysJ flavin reductase in molybdenum cofactor-dependent resistance of Escherichia coli to 6-N-hydroxylaminopurine
11. Hypersensitivity of Escherichia coli Delta(uvrB-bio) mutants to 6-hydroxylaminopurine and other base analogs is due to a defect in molybdenum cofactor biosynthesis
12. Comment on “A commensal strain of Staphylococcus epidermidis protects against skin neoplasia” by Nakatsuji et al .
13. The formation of double-strand breaks at multiply damaged sites is driven by the kinetics of excision/incision at base damage in eukaryotic cells
14. YcbX and yiiM, two novel determinants for resistance of Escherichia coli to N-hydroxylated base analogues
15. Roles of Saccharomyces cerevisiae DNA polymerases Polη and Polζ in response to irradiation by simulated sunlight
16. Mitochondrial Genome Variation Affects Multiple Respiration and Nonrespiration Phenotypes in Saccharomyces cerevisiae
17. Genome-wide screening for genes whose deletions confer sensitivity to mutagenic purine base analogs in yeast
18. Mitochondrial Genome Variation Affects Multiple Respiration and Nonrespiration Phenotypes in Saccharomyces cerevisiae.
19. 2μ plasmid inSaccharomycesspecies and inSaccharomyces cerevisiae
20. The 100-genomes strains, an S. cerevisiae resource that illuminates its natural phenotypic and genotypic variation and emergence as an opportunistic pathogen
21. Structures of Naturally Evolved CUP1 Tandem Arrays in Yeast Indicate That These Arrays Are Generated by Unequal Nonhomologous Recombination
22. A Critical Role for the Putative NCS2 Nucleobase Permease YjcD in the Sensitivity of Escherichia coli to Cytotoxic and Mutagenic Purine Analogs
23. TusA (YhhP) and IscS are required for molybdenum cofactor-dependent base-analog detoxification
24. The Mechanism of Nucleotide Excision Repair-Mediated UV-Induced Mutagenesis in Nonproliferating Cells
25. Molybdenum cofactor-dependent resistance to N-hydroxylated base analogs in Escherichia coli is independent of MobA function
26. Structure of the orthorhombic form of human inosine triphosphate pyrophosphatase
27. Genome-wide screening for genes whose deletions confer sensitivity to mutagenic purine base analogs in yeast
28. Hypersensitivity of Escherichia coli Δ ( uvrB-bio ) Mutants to 6-Hydroxylaminopurine and Other Base Analogs Is Due to a Defect in Molybdenum Cofactor Biosynthesis
29. Multiple antimutagenesis mechanisms affect mutagenic activity and specificity of the base analog 6-N-hydroxylaminopurine in bacteria and yeast
30. The 100-genomes strains, an S. cerevisiaeresource that illuminates its natural phenotypic and genotypic variation and emergence as an opportunistic pathogen
31. A Critical Role for the Putative NCS2 Nucleobase Permease YjcD in the Sensitivity of Escherichia colito Cytotoxic and Mutagenic Purine Analogs
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