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1. SHP2 inhibitors maintain TGFβ signalling through SMURF2 inhibition

4. Mechanisms of sensitivity and resistance to CDK4/CDK6 inhibitors in hormone receptor-positive breast cancer treatment

8. CXCR4 expression is elevated in TNBC patient derived samples and Z-guggulsterone abrogates tumor progression by targeting CXCL12/CXCR4 signaling axis in preclinical breast cancer model

10. Contributors

15. Emerging role of exosomes in cancer progression and tumor microenvironment remodeling

20. Nanotechnological Approaches in Prostate Cancer Therapy: Integration of engineering and biology

26. Hyaluronic acid-based nanoplatforms for Doxorubicin: A review of stimuli-responsive carriers, co-delivery and resistance suppression

31. Epigenetic derepression converts PPARγ into a druggable target in triple-negative and endocrine-resistant breast cancers

35. Macrophage IL-1β contributes to tumorigenesis through paracrine AIM2 inflammasome activation in the tumor microenvironment

38. Supplementary Table S1 and Figures S1-6 from Ascochlorin Enhances the Sensitivity of Doxorubicin Leading to the Reversal of Epithelial-to-Mesenchymal Transition in Hepatocellular Carcinoma

39. Data from Ascochlorin Enhances the Sensitivity of Doxorubicin Leading to the Reversal of Epithelial-to-Mesenchymal Transition in Hepatocellular Carcinoma

40. Supplementary Methods from Repression of NHE1 Expression by PPARγ Activation Is a Potential New Approach for Specific Inhibition of the Growth of Tumor Cells In vitro and In vivo

41. Supplementary Figures 1-6 from Repression of NHE1 Expression by PPARγ Activation Is a Potential New Approach for Specific Inhibition of the Growth of Tumor Cells In vitro and In vivo

42. Supplementary Figure Legends 1-6 from Repression of NHE1 Expression by PPARγ Activation Is a Potential New Approach for Specific Inhibition of the Growth of Tumor Cells In vitro and In vivo

43. Supplementary Table 1 from Repression of NHE1 Expression by PPARγ Activation Is a Potential New Approach for Specific Inhibition of the Growth of Tumor Cells In vitro and In vivo

44. Data from Repression of NHE1 Expression by PPARγ Activation Is a Potential New Approach for Specific Inhibition of the Growth of Tumor Cells In vitro and In vivo

47. Rap1 regulates hematopoietic stem cell survival and affects oncogenesis and response to chemotherapy

48. Author Correction: c-Met activation leads to the establishment of a TGFβ-receptor regulatory network in bladder cancer progression

49. c-Met activation leads to the establishment of a TGFβ-receptor regulatory network in bladder cancer progression

50. Contributors

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