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2. Contributors

4. Gut microbiota modulates weight gain in mice after discontinued smoke exposure

6. Diet Diurnally Regulates Small Intestinal Microbiome-Epithelial-Immune Homeostasis and Enteritis

7. Targeting purine synthesis in ASS1-expressing tumors enhances the response to immune checkpoint inhibitors

11. Potential roles of gut microbiome and metabolites in modulating ALS in mice

12. Publisher Correction: Gut microbiota modulates weight gain in mice after discontinued smoke exposure

20. Persistent microbiome alterations modulate the rate of post-dieting weight regain

21. Stress-related emotional and behavioural impact following the first COVID-19 outbreak peak

22. Chronic Activation of Corticotropin-Releasing Factor Type 2 Receptors Reveals a Key Role for 5-HT1A Receptor Responsiveness in Mediating Behavioral and Serotonergic Responses to Stressful Challenge

27. Artificial sweeteners induce glucose intolerance by altering the gut microbiota

28. Mechanistic dissection of dominant AIRE mutations in mouse models reveals AIRE autoregulation

34. ASL Metabolically Regulates Tyrosine Hydroxylase in the Nucleus Locus Coeruleus

37. ASL Metabolically Regulates Tyrosine Hydroxylase in the Nucleus Locus Coeruleus

43. Brown-adipose-tissue macrophages control tissue innervation and homeostatic energy expenditure

44. Loss of forebrain MTCH2 decreases mitochondria motility and calcium handling and impairs hippocampal-dependent cognitive functions

46. Loss of Muscle MTCH2 Increases Whole-Body Energy Utilization and Protects from Diet-Induced Obesity

47. Ucn3 and CRF-R2 in the medial amygdala regulate complex social dynamics

48. Increased anxiety in corticotropin-releasing factor type 2 receptor-null mice requires recent acute stress exposure and is associated with dysregulated serotonergic activity in limbic brain areas

49. Artificial Sweeteners Induce Glucose Intolerance by Altering the Gut Microbiota

50. Increased anxiety in corticotropin-releasing factor type 2 receptor-null mice requires recent acute stress exposure and is associated with dysregulated serotonergic activity in limbic brain areas

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