240 results on '"LaJeunesse D"'
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2. Biochemical Factors in Joint Articular Tissue Degradation in Osteoarthritis
3. Subchondral bone sclerosis in osteoarthritis: not just an innocent bystander
4. IGF/IGFBP axis in cartilage and bone in osteoarthritis pathogenesis
5. Subchondral and trabecular bone metabolism regulation in canine experimental knee osteoarthritis
6. Activation of Maxi-K Channels by Parathyroid Hormone and Prostaglandin E2 in Human Osteoblast Bone Cells
7. Treatment with licofelone prevents abnormal subchondral bone cell metabolism in experimental dog osteoarthritis
8. The hypocalciuric effect of thiazides: subcellular localization of the action
9. Therapeutic role of dual inhibitors of 5-LOX and COX, selective and non-selective non-steroidal anti-inflammatory drugs
10. Can altered production of interleukin-1β, interleukin-6, transforming growth factor-β and prostaglandin E2 by isolated human subchondral osteoblasts identify two subgroups of osteoarthritic patients
11. Carboxylated and uncarboxylated osteocalcin levels are not linked with insulin resistance in osteoarthritis patients
12. The protective effect of licofelone on experimental osteoarthritis is correlated with the downregulation of gene expression and protein synthesis of several major cartilage catabolic factors: MMP-13, cathepsin K and aggrecanases
13. Resveratrol Regulates the Wnt/ß-catenin pathway in human osteoarthritis osteoblasts
14. Alterations of the non-canonical Wnt/PKC pathway in human osteoarthritis osteoblasts
15. FRI0048 Hypoxia in human osteoarthritic subchondral bone: a risk factor for leptin production by osteoblasts
16. Osteoarthritis: rationale for a new paradigm shift involving transforming growth factor-β1
17. Elevated hepatocyte growth factor levels in osteoarthritis osteoblasts contribute to their altered response to bone morphogenetic protein-2 and reduced mineralization
18. 241 ELEVATED ENDOGENOUS SCLEROSTIN LEVELS IN OSTEOARTHRITIS SUBCHONDRAL OSTEOBLASTS CONTRIBUTE TO REDUCED WNT/B-CATENIN SIGNALING IN THESE CELLS
19. Elevated endogenous sclerostin levels in osteoarthritis subchondral osteoblasts contribute to reduced Wnt/B-catenin signaling in these cells
20. 058 R-SPONDINS ARE NEW PLAYERS IN OSTEOARTHRITIS THAT REGULATE WNT SIGNALING IN OSTEOBLASTS
21. 153 MODULATION OF THE PHENOTYPE OF HUMAN OSTEOARTHRITIC SUBCHONDRAL OSTEOBLASTS BY CONJUGATED LIPIDS
22. Validity of pain assessment methods in an experimental dog model
23. Thiazide Diuretics Affect Osteocalcin Production in Human Osteoblasts at the Transcription Level Without Affecting Vitamin D3 Receptors
24. Modulation of OPG, RANK and RANKL by human chondrocytes and their implication during osteoarthritis
25. 039 VALIDITY OF PAIN ASSESSMENT METHODS IN THE EXPERIMENTAL DOG POND-NUKI MODEL
26. 004 TILUDRONATE IN AN EXPERIMENTAL DOG MODEL OF OSTEOARTHRITIS: BONE ANTI-RESORPTIVE ACTIVITY AND RELATED FUNCTIONAL IMPROVEMENT
27. 238 MODULATION OF OSTEOPROTEGERIN, RANK, RANKL BY HUMAN CHONDROCYTES AND THEIR IMPLICATION IN OSTEOARTHRITIS
28. I-12 ROLE OF BONE IN THE DEVELOPMENT AND PROGRESSION OF OSTEOARTHRITIS
29. 056 ALTERED WNT/b-CATENIN SIGNALING IN HUMAN OSTEOARTHRITIC SUBCHONDRAL OSTEOBLASTS IS DUE TO DICKKOPF-2 (DKK2) AND PROSTAGLANDIN E2 (PGE2) LEVELS
30. DOES SUBCHONDRAL BONE TISSUE REMODELING OR CELL FUNCTION PLAY A ROLE IN ARTICULAR CARTILAGE LOSS IN OSTEOARTHRITIS?
31. 13 THE REMODELLING PROCESS OF THE SUBCHONDRAL BONE DURING HUMAN OSTEOARTHRITIS IS CLOSELY DEPENDENT ON RANKL MODULATION
32. Up-Regulation of Liver Glucose-6-Phosphatase in X-Linked Hypophosphatemic Mice
33. Abnormal collagen type I production in osteoarthritic subchondral bone is associated with a reduced capacity of osteoblasts to mineralize in vitro
34. Hepatocyte growth factor in osteoarthritis: when bone and cartilage decide to have a chat
35. Altered phosphorylation of Syp may be responsible for abnormal insulin-like growth factor-1 signaling in human osteoarthritic osteoblasts
36. Influence of Aluminum on the Regulation of PTH- and 1,25(OH)2D3-Dependent Pathways in the Rat Osteosarcoma Cell Line ROS 17/2.8
37. Pharmacological and Biochemical Evidence for the Regulation of Osteocalcin Secretion by Potassium Channels in Human Osteoblast-like MG–63 Cells
38. Demonstration of an osteoblast defect in two cases of human malignant osteopetrosis. Correction of the phenotype after bone marrow transplant.
39. E(z): a polycomb group gene or a trithorax group gene?
40. Activation of Maxi-K Channels by Parathyroid Hormone and Prostaglandin E 2 in Human Osteoblast Bone Cells
41. Calcitonin receptor in human placental syncytiotrophoblast brush border and basal plasma membranes
42. Parathyroid hormone and hydrochlorothiazide increase calcium transport by the luminal membrane of rabbit distal nephron segments through different pathways.
43. Strontium ranelate reduces the progression of experimental dog osteoarthritis by inhibiting the expression of key proteases in cartilage and of IL-1[beta] in the synovium.
44. Effect of vitamin D depletion on calcium transport by the luminal and basolateral membranes of the proximal and distal nephrons.
45. The high affinity system of calcium (Ca2+) transport by the luminal membrane of distal tubule (DT) is a Ca2+ channel sensitive to parathyroid hormone and vitamin D
46. The Mechanism of Parathyroid Hormone Action on Calcium Reabsorption by the Distal Tubule*
47. Effect of insulin on d-glucose transport by human placental brush border membranes
48. Thiazide Diuretics Affect Osteocalcin Production in Human Osteoblasts at the Transcription Level Without Affecting Vitamin D3 Receptors.
49. Influence of Aluminum on the Regulation of PTH- and 1,25(OH)2D3-Dependent Pathways in the Rat Osteosarcoma Cell Line ROS 17/2.8.
50. Can altered production of interleukin-1β, interleukin-6, transforming growth factor-β and prostaglandin E2by isolated human subchondral osteoblasts identify two subgroups of osteoarthritic patients
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