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1. Evidence-Based Assessment of Genes in Dilated Cardiomyopathy

2. Rare variants in KDR, encoding VEGF Receptor 2, are associated with tetralogy of Fallot

3. Genetic analyses of the electrocardiographic QT interval and its components identify additional loci and pathways

4. Enhancing rare variant interpretation in inherited arrhythmias through quantitative analysis of consortium disease cohorts and population controls

5. Shared genetic pathways contribute to risk of hypertrophic and dilated cardiomyopathies with opposite directions of effect

6. Biallelic variants in POPDC2 cause a novel autosomal recessive syndrome presenting with cardiac conduction defects and variable hypertrophic cardiomyopathy

7. Common Genetic Variants Contribute to Risk of Transposition of the Great Arteries

8. The yield of postmortem genetic testing in sudden death cases with structural findings at autopsy

10. Transethnic Genome-Wide Association Study Provides Insights in the Genetic Architecture and Heritability of Long QT Syndrome

11. Prognostic significance of fever-induced Brugada syndrome

13. Homozygous frameshift mutations in FAT1 cause a syndrome characterized by colobomatous-microphthalmia, ptosis, nephropathy and syndactyly

14. Genetic analyses of the electrocardiographic QT interval and its components identify additional loci and pathways

15. Genetic analyses of the electrocardiographic QT interval and its components identify additional loci and pathways

16. PO-01-077 IDENTIFYING NOVEL DISEASE MODIFIERS IN PATIENTS WITH CONGENITAL LONG QT SYNDROME

18. Correction to: Rare variants in KDR, encoding VEGF Receptor 2, are associated with tetralogy of Fallot (Genetics in Medicine, (2021), 23, 10, (1952-1960), 10.1038/s41436-021-01212-y)

19. Genetic analyses of the QT interval and its components in over 250K individuals identifies new loci and pathways affecting ventricular depolarization and repolarization

21. Genetic analyses of the QT interval and its components in over 250K individuals identifies new loci and pathways affecting ventricular depolarization and repolarization

24. Common Genetic Variants Contribute to Risk of Transposition of the Great Arteries

25. Shared genetic pathways contribute to risk of hypertrophic and dilated cardiomyopathies with opposite directions of effect

26. Evidence-Based Assessment of Genes in Dilated Cardiomyopathy

27. Biallelic loss-of-function variants in PLD1 cause congenital right-sided cardiac valve defects and neonatal cardiomyopathy

28. An Evidence-based Assessment of Genes in Dilated Cardiomyopathy

30. Predicting cardiac electrical response to sodium-channel blockade and Brugada syndrome using polygenic risk scores

31. Genome-wide association studies of cardiac electrical phenotypes

32. Discovery of rare variants associated with blood pressure regulation through meta-analysis of 1.3 million individuals.

33. Transethnic Genome-Wide Association Study Provides Insights in the Genetic Architecture and Heritability of Long QT Syndrome

35. The yield of postmortem genetic testing in sudden death cases with structural findings at autopsy

36. GATA6 mutations: Characterization of two novel patients and a comprehensive overview of the GATA6 genotypic and phenotypic spectrum

37. Calmodulin mutations and life-threatening cardiac arrhythmias: insights from the International Calmodulinopathy Registry

38. GNB5 Mutations Cause an Autosomal-Recessive Multisystem Syndrome with Sinus Bradycardia and Cognitive Disability

39. Yield and pitfalls of ajmaline testing in the evaluation of unexplained cardiac arrest and sudden unexplained death: Single centre experience of 482 families

40. Erratum: GNB5 Mutations Cause an Autosomal-Recessive Multisystem Syndrome with Sinus Bradycardia and Cognitive Disability (American Journal of Human Genetics (2016) 99(3) (704–710)(S0002929716302294)(10.1016/j.ajhg.2016.06.025))

41. Correction: GNB5 Mutations Cause an Autosomal-Recessive Multisystem Syndrome with Sinus Bradycardia and Cognitive Disability

43. Utility of Post-Mortem Genetic Testing in Cases of Sudden Arrhythmic Death Syndrome

44. Utility of Post-Mortem Genetic Testing in Cases of Sudden Arrhythmic Death Syndrome

45. Exome sequencing identifies primary carnitine deficiency in a family with cardiomyopathy and sudden death

46. GNB5 Mutations Cause an Autosomal-Recessive Multisystem Syndrome with Sinus Bradycardia and Cognitive Disability

47. GNB5 Mutations Cause an Autosomal-Recessive Multisystem Syndrome with Sinus Bradycardia and Cognitive Disability

48. Correction: GNB5 Mutations Cause an Autosomal-Recessive Multisystem Syndrome with Sinus Bradycardia and Cognitive Disability

50. Inherited dilated cardiomyopathy in a large Moroccan family caused by LMNA mutation.

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