157 results on '"Lam, Hilaire C."'
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2. Hypersensitivity to ferroptosis in chromophobe RCC is mediated by a glutathione metabolic dependency and cystine import via solute carrier family 7 member 11
3. Interleukin-6 mediates PSAT1 expression and serine metabolism in TSC2-deficient cells
4. TSC2 regulates lysosome biogenesis via a non-canonical RAGC and TFEB-dependent mechanism
5. Rapamycin-upregulated miR-29b promotes mTORC1-hyperactive cell growth in TSC2-deficient cells by downregulating tumor suppressor retinoic acid receptor β (RARβ)
6. Striated preferentially expressed gene deficiency leads to mitochondrial dysfunction in developing cardiomyocytes
7. mTORC1 Couples Nucleotide Synthesis to Nucleotide Demand Resulting in a Targetable Metabolic Vulnerability
8. Renal disease in tuberous sclerosis complex: pathogenesis and therapy
9. Inactivation of Tsc2 in Mesoderm-Derived Cells Causes Polycystic Kidney Lesions and Impairs Lung Alveolarization
10. Mammalian Target of Rapamycin
11. Data from TSC2 Interacts with HDLBP/Vigilin and Regulates Stress Granule Formation
12. Figure S2 from TSC2 Interacts with HDLBP/Vigilin and Regulates Stress Granule Formation
13. Supplementary Table 1 from TSC2 Interacts with HDLBP/Vigilin and Regulates Stress Granule Formation
14. Supplementary Table S1 from p62/SQSTM1 Cooperates with Hyperactive mTORC1 to Regulate Glutathione Production, Maintain Mitochondrial Integrity, and Promote Tumorigenesis
15. Raw data for Supplementary Table 1 and 2 from p62/SQSTM1 Cooperates with Hyperactive mTORC1 to Regulate Glutathione Production, Maintain Mitochondrial Integrity, and Promote Tumorigenesis
16. Figure S4 from p62/SQSTM1 Cooperates with Hyperactive mTORC1 to Regulate Glutathione Production, Maintain Mitochondrial Integrity, and Promote Tumorigenesis
17. Supplementary Materials and Methods from p62/SQSTM1 Cooperates with Hyperactive mTORC1 to Regulate Glutathione Production, Maintain Mitochondrial Integrity, and Promote Tumorigenesis
18. Data from p62/SQSTM1 Cooperates with Hyperactive mTORC1 to Regulate Glutathione Production, Maintain Mitochondrial Integrity, and Promote Tumorigenesis
19. Figure S2 from Therapeutic Targeting of DGKA-Mediated Macropinocytosis Leads to Phospholipid Reprogramming in Tuberous Sclerosis Complex
20. Spectrum of germline and somatic mitochondrial DNA variants in Tuberous Sclerosis Complex
21. Vps34-mediated macropinocytosis in Tuberous Sclerosis Complex 2-deficient cells supports tumorigenesis
22. Drug Inhibition of Redox Factor-1 Restores Hypoxia-Driven Changes in Tuberous Sclerosis Complex 2 Deficient Cells
23. Autophagy protein microtubule-associated protein 1 light chain-3B (LC3B) activates extrinsic apoptosis during cigarette smoke-induced emphysema
24. Protease activity sensors enable real-time treatment response monitoring in lymphangioleiomyomatosis
25. New developments in the genetics and pathogenesis of tumours in tuberous sclerosis complex
26. Histone deacetylase 6-mediated selective autophagy regulates COPD-associated cilia dysfunction
27. Mammalian Target of Rapamycin
28. Abstract 14210: Impaired Mitophagy and Reduced Parkin Expression in Human Pulmonary Arterial Smooth Muscle Cells (PASMCs) in Pulmonary Arterial Hypertension (PAH)
29. Protease activity sensors enable real-time treatment response monitoring in lymphangioleiomyomatosis
30. mTORC1 is a mechanosensor that regulates surfactant function and lung compliance during ventilator-induced lung injury
31. Interleukin-6 mediates PSAT1 expression and serine metabolism in TSC2-deficient cells
32. TSC2 Interacts with HDLBP/Vigilin and Regulates Stress Granule Formation
33. Hypersensitivity to ferroptosis in chromophobe RCC is mediated by a glutathione metabolic dependency and cystine import via solute carrier family 7 member 11.
34. Therapeutic Targeting of DGKA-Mediated Macropinocytosis Leads to Phospholipid Reprogramming in Tuberous Sclerosis Complex
35. Correction: p62/SQSTM1 Cooperates with Hyperactive mTORC1 to Regulate Glutathione Production, Maintain Mitochondrial Integrity, and Promote Tumorigenesis
36. Histone deacetylase 6–mediated selective autophagy regulates COPD-associated cilia dysfunction
37. Abstract A28: Rapamycin-upregulated miR-29b promotes mTORC1-hyperactivative cell growth by downregulating retinoic acid receptor β (RARβ)
38. Tumors with TSC mutations are sensitive to CDK7 inhibition through NRF2 and glutathione depletion
39. Interleukin-6 mediates PSAT1 expression and serine metabolism in TSC2-deficient cells.
40. TSC2-deficient tumors have evidence of T cell exhaustion and respond to anti–PD-1/anti–CTLA-4 immunotherapy
41. TSC2 regulates microRNA biogenesis via mTORC1 and GSK3β
42. Rapamycin-induced miR-21 promotes mitochondrial homeostasis and adaptation in mTORC1 activated cells
43. p62/SQSTM1 Cooperates with Hyperactive mTORC1 to Regulate Glutathione Production, Maintain Mitochondrial Integrity, and Promote Tumorigenesis
44. New developments in the genetics and pathogenesis of tumours in tuberous sclerosis complex
45. Abstract A35: Regulation of the tumor microenvironment by oncogenic p62 in mTORC1-hyperactive diseases: Tuberous sclerosis complex (TSC) and lymphangioleiomyomatosis (LAM)
46. Cecal Ligation and Puncture-induced Sepsis as a Model To Study Autophagy in Mice
47. Abstract B12: Rapamycin-induced miR-21 promotes the survival of mTORC1 hyperactivated cells in tuberous sclerosis complex models
48. Cecal Ligation and Puncture-induced Sepsis as a Model To Study Autophagy in Mice
49. “Ciliophagy”
50. Abstract A35: Identification of pro-survival rapamycin-dependent microRNA (Rapa-miRs) in tuberous sclerosis complex
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