742 results on '"Lan, Hui‐Yao"'
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2. Disrupting Smad3 potentiates immunostimulatory function of NK cells against lung carcinoma by promoting GM-CSF production
3. Long Non-coding RNA NEAT1, NOD-Like Receptor Family Protein 3 Inflammasome, and Acute Kidney Injury
4. Activation of the YAP/KLF5 transcriptional cascade in renal tubular cells aggravates kidney injury
5. From inflammation to renal fibrosis: A one-way road in autoimmunity?
6. DNA methylation markers for kidney function and progression of diabetic kidney disease
7. Macrophages mediate psoriasis via Mincle-dependent mechanism in mice
8. SARS-CoV-2 N protein induces acute kidney injury in diabetic mice via the Smad3-Ripk3/MLKL necroptosis pathway
9. Smad3 is essential for polarization of tumor-associated neutrophils in non-small cell lung carcinoma
10. Therapeutic potential for renal fibrosis by targeting Smad3-dependent noncoding RNAs
11. Latent TGF-β1 protects against diabetic kidney disease via Arkadia/Smad7 signaling.
12. Asiatic acid alleviates cisplatin-induced renal fibrosis in tumor-bearing mice by improving the TFEB-mediated autophagy-lysosome pathway
13. Estrogen receptor β attenuates renal fibrosis by suppressing the transcriptional activity of Smad3
14. Kidney tubular transcription co-activator, Yes-associated protein 1 (YAP), controls the expression of collecting duct aquaporins and water homeostasis
15. Genetic and pharmacological inhibition of GRPR protects against acute kidney injury via attenuating renal inflammation and necroptosis
16. Angiotensin II mediates hypertensive cardiac fibrosis via an Erbb4-IR-dependent mechanism
17. Macrophage-derived macrophage migration inhibitory factor mediates renal injury in anti-glomerular basement membrane glomerulonephritis
18. #1771 ANXA13 protects against acute kidney injury by inhibiting TGF-β/Smad3 pathway
19. Self-carried nanodrug (SCND-SIS3): A targeted therapy for lung cancer with superior biocompatibility and immune boosting effects
20. High-density lipoprotein subclasses and cardiovascular disease and mortality in type 2 diabetes: analysis from the Hong Kong Diabetes Biobank
21. Neuropeptide Y attenuates cardiac remodeling and deterioration of function following myocardial infarction
22. Regulatory role and mechanisms of myeloid TLR4 in anti-GBM glomerulonephritis
23. Macrophages in Renal Fibrosis
24. Hematopoietic Transcription Factor RUNX1 is Essential for Promoting Macrophage–Myofibroblast Transition in Non‐Small‐Cell Lung Carcinoma (Adv. Sci. 1/2024)
25. Treatment for type 2 diabetes and diabetic nephropathy by targeting Smad3 signaling
26. Neural transcription factor Pou4f1 promotes renal fibrosis via macrophage–myofibroblast transition
27. GSDME-mediated pyroptosis promotes inflammation and fibrosis in obstructive nephropathy
28. Arid2-IR promotes NF-κB-mediated renal inflammation by targeting NLRC5 transcription
29. Deletion of Smad3 prevents renal fibrosis and inflammation in type 2 diabetic nephropathy
30. Commentary: Role of estrogen receptor β in kidney disease
31. Hematopoietic Transcription Factor RUNX1 is Essential for Promoting Macrophage–Myofibroblast Transition in Non‐Small‐Cell Lung Carcinoma
32. SARS-CoV-2 N protein induced acute kidney injury in diabetic db/db mice is associated with a Mincle-dependent M1 macrophage activation
33. From inflammation to renal fibrosis: A one-way road in autoimmunity?
34. SAP130 released by damaged tubule drives necroinflammation via miRNA-219c/Mincle signaling in acute kidney injury
35. Exosomal miRNA-19b-3p of tubular epithelial cells promotes M1 macrophage activation in kidney injury
36. Btg2 Promotes Focal Segmental Glomerulosclerosis via Smad3‐Dependent Podocyte‐Mesenchymal Transition
37. RGMb protects against acute kidney injury by inhibiting tubular cell necroptosis via an MLKL-dependent mechanism
38. Macrophages: versatile players in renal inflammation and fibrosis
39. Neuropeptide Y attenuates cardiac remodeling and deterioration of function following myocardial infarction
40. Identification of a Common Variant for Coronary Heart Disease at PDE1A Contributes to Individualized Treatment Goals and Risk Stratification of Cardiovascular Complications in Chinese Patients With Type 2 Diabetes
41. Data from The Mincle/Syk/NF-κB Signaling Circuit Is Essential for Maintaining the Protumoral Activities of Tumor-Associated Macrophages
42. Supplementary Table 1 from The Mincle/Syk/NF-κB Signaling Circuit Is Essential for Maintaining the Protumoral Activities of Tumor-Associated Macrophages
43. Supplementary Figures S1 to S10 from The Mincle/Syk/NF-κB Signaling Circuit Is Essential for Maintaining the Protumoral Activities of Tumor-Associated Macrophages
44. Data from Enhanced Cancer Immunotherapy with Smad3-Silenced NK-92 Cells
45. Tables S1 to S3 from Enhanced Cancer Immunotherapy with Smad3-Silenced NK-92 Cells
46. Figures S1 to S15 from Enhanced Cancer Immunotherapy with Smad3-Silenced NK-92 Cells
47. The incidence, risk factors, and long-term outcomes of acute kidney injury in hospitalized diabetic ketoacidosis patients
48. Supplementary Table 1 from Transforming Growth Factor β1 Promotes Chromosomal Instability in Human Papillomavirus 16 E6E7–Infected Cervical Epithelial Cells
49. supplementary Figure 5 from Transforming Growth Factor β1 Promotes Chromosomal Instability in Human Papillomavirus 16 E6E7–Infected Cervical Epithelial Cells
50. Supplementary Figure 3 from Transforming Growth Factor β1 Promotes Chromosomal Instability in Human Papillomavirus 16 E6E7–Infected Cervical Epithelial Cells
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