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1,189 results on '"Lang, U"'

3. Assouad-Nagata dimension of wreath products of groups

Author

Brodskiy, N., Dydak, J., and Lang, U.

Subjects
Mathematics - Metric Geometry, Mathematics - Group Theory, Mathematics - Geometric Topology
Abstract

Consider the wreath product $H\wr G$, where $H\ne 1$ is finite and $G$ is finitely generated. We show that the Assouad-Nagata dimension $\dim_{AN}(H\wr G)$ of $H\wr G$ depends on the growth of $G$ as follows: If the growth of $G$ is not bounded by a linear function, then $\dim_{AN}(H\wr G)=\infty$, otherwise $\dim_{AN}(H\wr G)=\dim_{AN}(G)\leq 1$., Comment: 11 pages, new references added

Published
2006

4. Interlaboratory variability of Ki67 staining in breast cancer

Author

Anders, M., Bollmann, R., Eiting, Fr., Friedrich, K., Habeck, J.-O., Haroske, G., Hinrichs, B., Behrens, A., Krause, U., Lang, U., Lorenzen, J., Minew, N., Mlynek-Kersjes, M., Nenning, H., Packeisen, J., Poche-de Vos, F., Reyher–Klein, S., Rothacker, D., Schultz, M., Sturm, U., Tawfik, M., Berghäuser, K.-H., Böcker, W., Cserni, G., Habedank, S., Lax, S., Moinfar, F., Regitnig, P., Reiner-Concin, A., Rüschoff, J., Varga, Z., Woziwodski, J., Focke, Cornelia M., Bürger, Horst, van Diest, Paul J., Finsterbusch, Kai, Gläser, Doreen, Korsching, Eberhard, and Decker, Thomas

Published
2017
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9. Typ-1-Diabetes

Author

Danne, T., Freckmann, G., Kautzky-Willer, A., Lang, U., Lehmann, R., Pfohl, M., Schloot, N. C., Schumm-Draeger, P. M., Schatz, Helmut, editor, and Pfeiffer, Andreas F. H., editor

Published
2014
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16. Distributed and collaborative visualization of simulation results

Author

Lang, U., Hirschel, Ernst Heinrich, editor, Fujii, Kozo, editor, Haase, Werner, editor, van Leer, Bram, editor, Leschziner, Michael A., editor, Pandolfi, Maurizio, editor, Periaux, Jacques, editor, Rizzi, Arthur, editor, Roux, Bernard, editor, Shokin, Yurii I., editor, Krause, Egon, editor, Resch, Michael, editor, and Shokina, Nina, editor

Published
2005
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19. Knochendichtemessung

Author

Manolopoulos, K., Stracke, H., Lang, U., Tinneberg, H.-R., Tinneberg, Hans-Rudolf, Kirschbaum, Michael, and Oehmke, Frank

Published
2003
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37. Attentional salience and the neural substrates of response inhibition in borderline personality disorder.

Author

Wrege, J. S., Carcone, D., Lee, A. C. H., Cane, C., Lang, U. E., Borgwardt, S., Walter, M., and Ruocco, A. C.

Subjects
PREFRONTAL cortex, PARIETAL lobe, NEUROPHYSIOLOGY, BORDERLINE personality disorder, IMPULSIVE personality, SELF-evaluation, MAGNETIC resonance imaging, TASK performance, ATTENTION
Abstract

Background: Impulsivity is a central symptom of borderline personality disorder (BPD) and its neural basis may be instantiated in a frontoparietal network involved in response inhibition. However, research has yet to determine whether neural activation differences in BPD associated with response inhibition are attributed to attentional saliency, which is subserved by a partially overlapping network of brain regions. Methods: Patients with BPD (n = 45) and 29 healthy controls (HCs; n = 29) underwent functional magnetic resonance imaging while completing a novel go/no-go task with infrequent odd-ball trials to control for attentional saliency. Contrasts reflecting a combination of response inhibition and attentional saliency (no-go > go), saliency processing alone (oddball > go), and response inhibition controlling for attentional saliency (no-go > oddball) were compared between BPD and HC. Results: Compared to HC, BPD showed less activation in the combined no-go > go contrast in the right posterior inferior and middle-frontal gyri, and less activation for oddball > go in left-hemispheric inferior frontal junction, frontal pole, superior parietal lobe, and supramarginal gyri. Crucially, BPD and HC showed no activation differences for the no-go > oddball contrast. In BPD, higher vlPFC activation for no-go > go was correlated with greater self-rated BPD symptoms, whereas lower vlPFC activation for oddball > go was associated with greater self-rated attentional impulsivity. Conclusions: Patients with BPD show frontoparietal disruptions related to the combination of response inhibition and attentional saliency or saliency alone, but no specific response inhibition neural activation difference when attentional saliency is controlled. The findings suggest a neural dysfunction in BPD underlying attention to salient or infrequent stimuli, which is supported by a negative correlation with self-rated impulsiveness. [ABSTRACT FROM AUTHOR]

Published
2022
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