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2. Major motor and gait deficits with sexual dimorphism in a Shank3 mutant mouse model

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3. Major Contribution of Somatostatin-Expressing Interneurons and Cannabinoid Receptors to Increased GABA Synaptic Activity in the Striatum of Huntington’s Disease Mice

6. Cerebellar and Striatal Implications in Autism Spectrum Disorders: From Clinical Observations to Animal Models

7. Striatal <scp>GABA</scp> ergic interneuron dysfunction in the Q175 mouse model of Huntington's disease

8. Major Contribution of Somatostatin-Expressing Interneurons and Cannabinoid Receptors to Increased GABA Synaptic Activity in the Striatum of Huntington's Disease Mice

9. CREB controls cortical circuit plasticity and functional recovery after stroke

10. CYP46A1, the rate-limiting enzyme for cholesterol degradation, is neuroprotective in Huntington’s disease

11. Mitochondria in Huntington's disease

12. Behavioral phenotyping of heterozygous acetylcholinesterase knockout (AChE+/−) mice showed no memory enhancement but hyposensitivity to amnesic drugs

13. Rhes, a protein with selective expression in the striatum, plays a major role in Huntington’s disease pathogenesis

14. Striatal long noncoding RNA Abhd11os is neuroprotective against an N-terminal fragment of mutant huntingtin in vivo

15. Possible involvement of self-defense mechanisms in the preferential vulnerability of the striatum in Huntington's disease

16. A role of mitochondrial complex II defects in genetic models of Huntington's disease expressing N-terminal fragments of mutant huntingtin

17. Multiple Sources of Striatal Inhibition Are Differentially Affected in Huntington’s Disease Mouse Models

18. Effects of the Pimelic Diphenylamide Histone Deacetylase Inhibitor HDACi 4b on the R6/2 and N171-82Q Mouse Models of Huntington's Disease

19. Functional Differences Between Direct and Indirect Striatal Output Pathways in Huntington's Disease

20. Capucin does not modify the toxicity of a mutant Huntingtin fragment in vivo

21. Hyposensitivity to the amnesic effects of scopolamine or amyloid β25–35 peptide in heterozygous acetylcholinesterase knockout (AChE+/−) mice

22. Enhanced GABAergic Inputs Contribute to Functional Alterations of Cholinergic Interneurons in the R6/2 Mouse Model of Huntington’s Disease

23. JAKMIP1, a Novel Regulator of Neuronal Translation, Modulates Synaptic Function and Autistic-like Behaviors in Mouse