470 results on '"Lezoualc'H, Frank"'
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2. Signaling through cAMP-Epac1 induces metabolic reprogramming to protect podocytes in glomerulonephritis
3. Mechanisms of myocardial reverse remodelling and its clinical significance: A scientific statement of the ESC Working Group on Myocardial Function
4. Metformin protects against diclofenac-induced toxicity in primary rat hepatocytes by preserving mitochondrial integrity via a pathway involving EPAC
5. Elevated cAMP Protects against Diclofenac-Induced Toxicity in Primary Rat Hepatocytes: A Protective Effect Mediated by the Exchange Protein Directly Activated by cAMP/cAMP-Regulated Guanine Nucleotide Exchange Factors
6. Atrial Natriuretic Peptide Orchestrates a Coordinated Physiological Response to Fuel Non-shivering Thermogenesis
7. Mechanisms of myocardial reverse remodelling and its clinical significance: A scientific statement of the ESC Working Group on Myocardial Function
8. Modeling Epac1 interactions with the allosteric inhibitor AM-001 by co-solvent molecular dynamics
9. Mitochondrial 4-HNE derived from MAO-A promotes mitoCa2+ overload in chronic postischemic cardiac remodeling
10. Cyclic AMP-binding protein Epac1 acts as a metabolic sensor to promote cardiomyocyte lipotoxicity
11. Macromolecular Complex Including MLL3, Carabin and Calcineurin Regulates Cardiac Remodeling
12. Epac
13. Large Extracellular Vesicle-Associated Rap1 Accumulates in Atherosclerotic Plaques, Correlates with Vascular Risks and Is Involved in Atherosclerosis
14. Receptor-Specific Inside-Out cAMP Signaling Regulates Cardiomyocyte Fate
15. Sodium myo-inositol cotransporter-1, SMIT1, promotes cardiac hypertrophy and fibrosis in pressure overloaded mouse hearts
16. Acute heart failure: mechanisms and pre-clinical models—a Scientific Statement of the ESC Working Group on Myocardial Function
17. Ryanodine receptor dysfunction causes senescence and fibrosis in Duchenne dilated cardiomyopathy.
18. Acute heart failure: mechanisms and pre-clinical models-a Scientific Statement of the ESC Working Group on Myocardial Function
19. Macromolecular Complex Including MLL3, Carabin and Calcineurin Regulates Cardiac Remodeling.
20. PO-01-171 ROLE OF THE EXCHANGE PROTEIN ACTIVATED BY CAMP (EPAC) IN ATRIAL ELECTROPHYSIOLOGY AND ATRIAL FIBRILLATION MECHANISMS
21. Activation of Exchange Proteins directly Activated by cAMP (EPAC) lengthens action potential duration by inhibiting potassium currents in atrial cardiomyocytes
22. Inhibition of EPAC1 signaling pathway alters atrial electrophysiology and prevents atrial fibrillation
23. EPAC1 Pharmacological Inhibition with AM-001 Prevents SARS-CoV-2 and Influenza A Virus Replication in Cells
24. Inhalation of acidic nanoparticles prevents doxorubicin cardiotoxicity through improvement of lysosomal function
25. Epac1 promotes mitochondrial dysfunction and cardiomyocyte death during persistent lipid overload
26. Nanoparticles-mediated lysosomal acidification limits doxorubicin cardiotoxicity
27. Role of exchange protein directly activated by cAMP 1 in cardiac aging
28. Identification of Epac1 transcriptomic signature in ventricular myocytes
29. 5-HT3 and 5-HT4 Receptors as Targets for Drug Discovery for Dementia
30. Diabetic cardiomyopathy: the need for adjusting experimental models to meet clinical reality
31. Epac in cardiac calcium signaling
32. Specific interactions between Epac1, β-arrestin2 and PDE4D5 regulate β-adrenergic receptor subtype differential effects on cardiac hypertrophic signaling
33. Identification of a Tetrahydroquinoline Analog as a Pharmacological Inhibitor of the cAMP-binding Protein Epac
34. Sustained Epac activation induces calmodulin dependent positive inotropic effect in adult cardiomyocytes
35. Epac enhances excitation–transcription coupling in cardiac myocytes
36. Pharmacological activation of exchange protein directly activated by cAMP 1 (EPAC1) increases action potential duration in mouse atrial myocytes
37. Role of exchange protein directly activated by cAMP in cardiac aging
38. Exchange protein directly activated by cAMP 1, promotes iron-mediated cardiomyocyte death
39. Activation of Exchange Proteins directly Activated by cAMP (EPAC) promotes electrophysiological remodeling and arrhythmogenic activity in human atrial cardiomyocytes
40. Sodium myo-inositol cotransporter-1, SMIT1, promotes cardiac hypertrophy and fibrosis in pressure overloaded mouse hearts
41. The cAMP Binding Protein Epac Regulates Cardiac Myofilament Function
42. AMP-activated protein kinase and O-GlcNAcylation : a new paradigm to protect the failing heart
43. Rap-linked cAMP signaling Epac proteins: Compartmentation, functioning and disease implications
44. PKC-delta and PKC-epsilon: Foes of the same family or strangers?
45. Multifunctional Mitochondrial Epac1 Controls Myocardial Cell Death
46. Epac
47. Diabetic cardiomyopathy: the need for adjusting experimental models to meet clinical reality.
48. Epac activation induces histone deacetylase nuclear export via a Ras-dependent signalling pathway
49. Effectiveness and Safety of a Prolonged Hemodynamic Support by the IVAC2L System in Healthy and Cardiogenic Shock Pigs
50. Pharmacological Inhibition of Epac1 Averts Ferroptosis Cell Death by Preserving Mitochondrial Integrity
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