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1. Author Correction: The evolution of lung cancer and impact of subclonal selection in TRACERx

2. Antibodies against endogenous retroviruses promote lung cancer immunotherapy

3. The evolution of non-small cell lung cancer metastases in TRACERx

4. The evolution of lung cancer and impact of subclonal selection in TRACERx

5. Tracking early lung cancer metastatic dissemination in TRACERx using ctDNA

6. Genomic–transcriptomic evolution in lung cancer and metastasis

7. Lung adenocarcinoma promotion by air pollutants

8. Evolutionary characterization of lung adenocarcinoma morphology in TRACERx

9. Pervasive chromosomal instability and karyotype order in tumour evolution

10. Using DNA sequencing data to quantify T cell fraction and therapy response

11. Tumour-suppressor microRNAs regulate ovarian cancer cell physical properties and invasive behaviour

13. A clonal expression biomarker associates with lung cancer mortality

14. Data from Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution

15. Supplementary Figure 4 from Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution

16. TRACERx Consortium Members from Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution

17. Supplementary Figure 2 from Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution

18. Supplementary Tables S1-S4 from Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution

19. Supplementary Figure 3 from Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution

20. Supplementary Figure 1 from Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution

22. Escape from nonsense-mediated decay associates with anti-tumor immunogenicity

23. Body composition and lung cancer-associated cachexia in TRACERx

24. The evolution of lung cancer and impact of subclonal selection in TRACERx

25. The molecular landscape of pediatric acute myeloid leukemia reveals recurrent structural alterations and age-specific mutational interactions

26. Antibodies against endogenous retroviruses promote lung cancer immunotherapy

28. Abstract 1394: Pervasive allele specific transcriptional repression of the class I and II HLA genes in TRACERx non-small cell lung cancer

29. Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution

30. Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution

31. Publisher Correction: A clonal expression biomarker associates with lung cancer mortality

32. Publisher Correction: The molecular landscape of pediatric acute myeloid leukemia reveals recurrent structural alterations and age-specific mutational interactions

33. Abstract 2678: A clonal expression biomarker improves prognostic accuracy: TRACERx lung

34. Erratum: The molecular landscape of pediatric acute myeloid leukemia reveals recurrent structural alterations and age-specific mutational interactions

35. The molecular landscape of pediatric acute myeloid leukemia reveals recurrent structural alterations and age-specific mutational interactions

36. MicroRNA Expression-Based Model Indicates Event-Free Survival in Pediatric Acute Myeloid Leukemia

37. Comprehensive whole genome sequence analyses yields novel genetic and structural insights for Intellectual Disability

38. Marked Differences in the Genomic Landscape of Pediatric Compared to Adult Acute Myeloid Leukemia: A Report from the Children's Oncology Group and NCI/COG Therapeutically Applicable Research to Generate Effective Treatments (TARGET) Initiative

39. Alteration of Chromatin Modifiers and Misregulation of Transcription Factors Define the Genomic Profile of Infant AML

40. A microRNA Expression-Based Model Predicts Event Free Survival in Pediatric Acute Myeloid Leukemia

41. Divergent Epigenomes in Pediatric and Adult Acute Myeloid Leukemia Implicate Cell of Origin and Transcriptional Silencing of Immune Responses As Sources of Clinically Relevant Heterogeneity: A Report from the Children's Oncology Group and NCI/COG Therapeutically Applicable Research to Generate Effective Treatments (TARGET) Initiative

43. Interplay between whole-genome doubling and the accumulation of deleterious alterations in cancer evolution

44. Genome-Wide Profiles of Extra-cranial Malignant Rhabdoid Tumors Reveal Heterogeneity and Dysregulated Developmental Pathways

45. Comprehensive Sequence Analysis of Relapse and Refractory Pediatric Acute Myeloid Leukemia Identifies miRNA and mRNA Transcripts Associated with Treatment Resistance - a Report from the COG/NCI-Target AML Initiative

46. Comprehensive miRNA sequence analysis reveals survival differences in diffuse large B-cell lymphoma patients

47. Mutational and structural analysis of diffuse large B-cell lymphoma using whole-genome sequencing

48. MicroRNA dysregulation in B-cell non-Hodgkin lymphoma.

49. Electronic Supplementary Material from Tumour-suppressor microRNAs regulate ovarian cancer cell physical properties and invasive behaviour

50. Comprehensive whole genome sequence analyses yields novel genetic and structural insights for Intellectual Disability

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