37 results on '"Lin, Chien-liang Glenn"'
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2. Messenger RNA oxidation occurs early in disease pathogenesis and promotes motor neuron degeneration in ALS.
3. Enhancement of tripartite synapses as a potential therapeutic strategy for Alzheimer’s disease: a preclinical study in rTg4510 mice
4. Glutamate transporter EAAT2: regulation, function, and potential as a therapeutic target for neurological and psychiatric disease
5. Small-molecule activator of glutamate transporter EAAT2 translation provides neuroprotection
6. Emerging role of glutamate in the pathophysiology and therapeutics of Gulf War illness
7. Oxidative damage to RNA: mechanisms, consequences, and diseases
8. Increased glial glutamate transporter EAAT2 expression reduces visceral nociceptive response in mice
9. Translational Control of Glial Glutamate Transporter EAAT2 Expression
10. Restoring tripartite glutamatergic synapses: A potential therapy for mood and cognitive deficits in Gulf War illness
11. Quantification of oxidized RNAs in Alzheimer's disease
12. Glutamate transporter EAAT2: a new target for the treatment of neurodegenerative diseases
13. The Importance of Preclinical Trial Timing – a Potential Reason for the Disconnect between mouse Studies and Human Clinical Trials in ALS
14. Modulation of the neuronal glutamate transporter EAAC1 by the interacting protein GTRAP3-18
15. Effects of Axon Degeneration on Oligodendrocyte Lineage Cells: Dorsal Rhizotomy Evokes a Repair Response While Axon Degeneration Rostral to Spinal Contusion Induces Both Repair and Apoptosis
16. Increased expression of the glial glutamate transporter EAAT2 modulates excitotoxicity and delays the onset but not the outcome of ALS in mice
17. Methyl-β-cyclodextrin but not retinoic acid reduces EAAT3-mediated glutamate uptake and increases GTRAP3-18 expression
18. Pyridazine-derivatives Enhance Structural and Functional Plasticity of Tripartite Synapse Via Activation of Local Translation in Astrocytic Processes
19. Restored glial glutamate transporter EAAT2 function as a potential therapeutic approach for Alzheimer’s disease
20. Structure–activity relationship study of pyridazine derivatives as glutamate transporter EAAT2 activators
21. Increased glial glutamate transporter EAAT2 expression reduces epileptogenic processes following pilocarpine-induced status epilepticus
22. The presence of rRNA sequences in polyadenylated RNA and its potential functions
23. RNA oxidation: A contributing factor or an epiphenomenon in the process of neurodegeneration
24. Association of Excitatory Amino Acid Transporters, Especially EAAT2, with Cholesterol-rich Lipid Raft Microdomains
25. The Identification and Characterization of Oxidized RNAs in Alzheimer's Disease
26. Molecular cloning, gene structure, expression profile and functional characterization of the mouse glutamate transporter (EAAT3) interacting protein GTRAP3–18
27. Molecular cloning and expression of the rat EAAT4 glutamate transporter subtype
28. Selection of monoclonal antibodies for probing of functional intermediates in incision of UV-irradiated DNA by Uvr(A)BC endonuclease from Escherichia coli
29. Aberrant RNA Processing in a Neurodegenerative Disease: the Cause for Absent EAAT2, a Glutamate Transporter, in Amyotrophic Lateral Sclerosis
30. Oxidative damage to RNA: mechanisms, consequences, and diseases.
31. Messenger RNA Oxidation Occurs Early in Disease Pathogenesis and Promotes Motor Neuron Degeneration in ALS.
32. DNA damage-dependent recruitment of nucleotide excision repair and transcription proteins to Escherichia coli inner membranes
33. Human Glioma Cells and Undifferentiated Primary Astrocytes That Express Aberrant EAAT2 mRNA Inhibit Normal EAAT2 Protein Expression and Prevent Cell Death
34. Identification of translational activators of glial glutamate transporter EAAT2 through cell-based high-throughput screening: an approach to prevent excitotoxicity.
35. A novel noncoding RNA rescues mutant SOD1-mediated cell death.
36. Messenger RNA oxidation is an early event preceding cell death and causes reduced protein expression.
37. Methyl-beta-cyclodextrin but not retinoic acid reduces EAAT3-mediated glutamate uptake and increases GTRAP3-18 expression.
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