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6. Robust SARS-CoV-2-specific T cell immunity is maintained at 6 months following primary infection

7. Bioengineered small extracellular vesicles deliver multiple SARS‐CoV‐2 antigenic fragments and drive a broad immunological response

9. Immunology of THymectomy And childhood CArdiac transplant (ITHACA): protocol for a UK-wide prospective observational cohort study to identify immunological risk factors of post-transplant lymphoproliferative disease (PTLD) in thymectomised children

10. Author Correction: Robust SARS-CoV-2-specific T cell immunity is maintained at 6 months following primary infection

11. Tspan6 stimulates the chemoattractive potential of breast cancer cells for B cells in an EV- and LXR-dependent manner

17. Guidelines for the use of flow cytometry and cell sorting in immunological studies (third edition)

19. Guidelines for the use of flow cytometry and cell sorting in immunological studies (third edition)

20. Mutations in SARS-CoV-2 spike protein impair epitope-specific CD4+T cell recognition

22. The CD151‐midkine pathway regulates the immune microenvironment in inflammatory breast cancer

28. Asymptomatic Primary Infection with Epstein-Barr Virus: Observations on Young Adult Cases

29. Early virological and immunological events in asymptomatic Epstein-Barr virus infection in African children

30. Early virological and immunological events in asymptomatic Epstein-Barr virus infection in African children

31. CXCR5+ follicular cytotoxic T cells control viral infection in B cell follicles

34. Cellular immune controls over Epstein-Barr virus infection: new lessons from the clinic and the laboratory

35. MHC II tetramers visualize human CD4+ T cell responses to Epstein–Barr virus infection and demonstrate atypical kinetics of the nuclear antigen EBNA1 response

36. Suppression of Antigen-Specific T Cell Responses by the Kaposi's Sarcoma-Associated Herpesvirus Viral OX2 Protein and Its Cellular Orthologue, CD200

37. Structural definition of HLA class II-presented SARS-CoV-2 epitopes reveals a mechanism to escape pre-existing CD4+T cell immunity

41. An Epstein-Barr Virus Anti-Apoptotic Protein Constitutively Expressed in Transformed Cells and Implicated in Burkitt Lymphomagenesis: The Wp/BHRF1 Link

45. CXCR5+follicular cytotoxic T cells control viral infection in B cell follicles

46. Epstein-Barr Virus Evades CD4+ T Cell Responses in Lytic Cycle through BZLF1-mediated Downregulation of CD74 and the Cooperation of vBcl-2.

47. Cytotoxic CD4+ T Cell Responses to EBV Contrast with CD8 Responses in Breadth of Lytic Cycle Antigen Choice and in Lytic Cycle Recognition.

48. CD4+T-cell clones recognizing human lymphoma-associated antigens: generation by in vitro stimulation with autologous Epstein-Barr virus–transformed B cells

49. Regression of Epstein-Barr Virus-Induced B-Cell Transformation In Vitro Involves Virus-Specific CD8+ T Cells as the Principal Effectors and a Novel CD4+ T-CeIl Reactivity.

50. Primary EBV Infection Induces an Acute Wave of Activated Antigen-Specific Cytotoxic CD4+ T Cells.

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