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2. A comprehensive Drosophila resource to identify key functional interactions between SARS-CoV-2 factors and host proteins.

3. Homozygous missense variants in YKT6 result in loss of function and are associated with developmental delay, with or without severe infantile liver disease and risk for hepatocellular carcinoma

4. De novo variants in FRYL are associated with developmental delay, intellectual disability, and dysmorphic features

5. Loss of Neuron Navigator 2 Impairs Brain and Cerebellar Development

7. De novo variants in FRYL are associated with developmental delay, intellectual disability, and dysmorphic features

8. Allelic strengths of encephalopathy-associated UBA5 variants correlate between in vivo and in vitro assays

9. Author Response: Allelic strengths of encephalopathy-associated UBA5 variants correlate between in vivo and in vitro assays

11. The recurrent de novo c.2011C>T missense variant in MTSS2 causes syndromic intellectual disability

13. De novo variants in FRMD5 are associated with developmental delay, intellectual disability, ataxia, and abnormalities of eye movement

18. Loss-of-function variants in TIAM1 are associated with developmental delay, intellectual disability, and seizures

19. Novel dominant and recessive variants in human ROBO1 cause distinct neurodevelopmental defects through different mechanisms

20. Loss of Neuron Navigator 2 Impairs Brain and Cerebellar Development

22. TNPO2 variants associate with human developmental delays, neurologic deficits, and dysmorphic features and alter TNPO2 activity in Drosophila

23. TNPO2 variants associate with human developmental delays, neurologic deficits, and dysmorphic features and alter TNPO2 activity in Drosophila

24. Homozygous missense variants in YKT6result in loss of function and are associated with developmental delay, with or without severe infantile liver disease and risk for hepatocellular carcinoma

26. A comprehensive Drosophilaresource to identify key functional interactions between SARS-CoV-2 factors and host proteins

27. De novo variants in PLCG1 are associated with hearing impairment, ocular pathology, and cardiac defects.

28. Allelic strengths of encephalopathy-associated UBA5 variants correlate between in vivo and in vitro assays.

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