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3. Clonal Megakaryocyte Dysplasia with Isolated Thrombocytosis Is a Distinct Myeloproliferative Neoplasm Phenotype

Author

Barosi, Giovanni, primary, Campanelli, Rita, additional, Massa, Margherita, additional, Catarsi, Paolo, additional, Carolei, Adriana, additional, Abbà, Carlotta, additional, Villani, Laura, additional, Magrini, Umberto, additional, Gregato, Giuliana, additional, Bertolini, Francesco, additional, de Silvestri, Annalisa, additional, Gale, Robert Peter, additional, and Rosti, Vittorio, additional

Published
2022
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4. Spleen endothelial cells from patients with myelofibrosis harbor the JAK2V617F mutation

Author

Rosti, Vittorio, Villani, Laura, Riboni, Roberta, Poletto, Valentina, Bonetti, Elisa, Tozzi, Lorenzo, Bergamaschi, Gaetano, Catarsi, Paolo, Dallera, Elena, Novara, Francesca, Massa, Margherita, Campanelli, Rita, Fois, Gabriela, Peruzzi, Benedetta, Lucioni, Marco, Guglielmelli, Paola, Pancrazzi, Alessandro, Fiandrino, Giacomo, Zuffardi, Orsetta, Magrini, Umberto, Paulli, Marco, Vannucchi, Alessandro M., and Barosi, Giovanni

Published
2013
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5. Clonal Megakaryocyte Dysplasia with Isolated Thrombocytosis Is a Distinct Myeloproliferative Neoplasm Phenotype.

Author

Barosi, Giovanni, Campanelli, Rita, Massa, Margherita, Catarsi, Paolo, Carolei, Adriana, Abbà, Carlotta, Villani, Laura, Magrini, Umberto, Gregato, Giuliana, Bertolini, Francesco, de Silvestri, Annalisa, Gale, Robert Peter, and Rosti, Vittorio

Subjects
MYELOPROLIFERATIVE neoplasms, MYELOFIBROSIS, THROMBOCYTOSIS, DYSPLASIA, PHENOTYPES, BONE marrow
Abstract

Introduction: About 15% of people with a myeloproliferative neoplasm (MPN) are identified as MPN, unclassifiable using the 2016 WHO classification. Methods: We tested whether persons with platelet concentration ≥450 × 10E+9/L, bone marrow megakaryocyte morphology typical of prefibrotic/early myelofibrosis (pre-MF), and no minor criteria of pre-MF should be classified as a distinct MPN subtype, clonal megakaryocyte dysplasia with isolated thrombocytosis (CMD-IT). Results: 139 subjects meet these criteria who we compared with primary myelofibrosis (PMF) including 402 with pre-MF and 521 with overt myelofibrosis. CMD-IT subjects were more likely female and younger. They had lower frequencies of JAK2V617F compared with persons with PMF (55% vs. 70%; p < 0.001) and higher frequencies of CALR mutations (37% vs. 17%; p < 0.001). They also had lower frequency of variations associated with JAK2V617F susceptibility, JAK2 46/1 (35% vs. 47%; p = 0.021), and VEGFA rs3025039 (12% vs. 17%; p = 0.030). Subjects with CMD-IT had lower incidences of thrombotic events compared with those with pre-MF (9.7% vs. 26%; p < 0.001) and longer survival (median, not reached vs. 23 years; HR = 0.34 (0.10, 0.30); p < 0.001). Conclusion: Our data indicate CMD-IT is a distinct MPN subtype and should be included in the classification of myeloid neoplasms. [ABSTRACT FROM AUTHOR]

Published
2023
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6. Clonal Megakaryocyte Dysplasia with Normal Blood Values Is a Distinct Myeloproliferative Neoplasm

Author

Barosi, Giovanni, primary, Rosti, Vittorio, additional, Massa, Margherita, additional, Campanelli, Rita, additional, Villani, Laura, additional, Catarsi, Paolo, additional, Carolei, Adriana, additional, Abbà, Carlotta, additional, Lodigiani, Corrado, additional, Primignani, Massimo, additional, Gregato, Giuliana, additional, Bertolini, Francesco, additional, Magrini, Umberto, additional, and Gale, Robert Peter, additional

Published
2021
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7. Clonal Megakaryocyte Dysplasia with Normal Blood Values Is a Distinct Myeloproliferative Neoplasm.

Author

Barosi, Giovanni, Rosti, Vittorio, Massa, Margherita, Campanelli, Rita, Villani, Laura, Catarsi, Paolo, Carolei, Adriana, Abbà, Carlotta, Lodigiani, Corrado, Primignani, Massimo, Gregato, Giuliana, Bertolini, Francesco, Magrini, Umberto, and Gale, Robert Peter

Subjects
MYELOPROLIFERATIVE neoplasms, DYSPLASIA, MYELOFIBROSIS, DISEASE progression, BONE marrow, CONFIDENCE intervals
Abstract

Introduction: In 1991, we reported 18 persons with a clinical-pathologic entity and termed atypical myeloproliferative disorder because they did not meet the contemporary diagnostic criteria for a myeloproliferative neoplasm. We sought to gain further knowledge on this disease entity. Methods: This retrospective cohort study included consecutive subjects registered in the database of the Center for the Study of Myelofibrosis in Pavia, Italy, from 1998 to 2020 (June), and diagnosed with atypical myeloproliferative disorder according to our adjudicated criteria. We studied clinical, histological, cytogenetic, and molecular covariates and risks of thrombosis, disease progression, and death. Data were compared with those of concurrent subjects with prefibrotic myelofibrosis. Results: Fifteen new subjects with atypical myeloproliferative disorder were identified. Seven were male. Median age was 50 years (IQR, 41–54 years). Thirteen were diagnosed with a synchronous symptomatic or incidentally detected thrombotic event. The bone marrow showed megakaryocyte hyperplasia with dysplasia. JAK2V617F was present in 10 subjects and CALR mutation in one. No other somatic mutations were identified in next generation sequencing. After a median follow-up of 101 months (IQR, 40–160 months), no subject had disease progression or blast transformation. Incidence of post-diagnosis or recurrent thrombosis was 3.9 events (95% confidence interval, 3.5–4.0) and 5.0 events (4.6–5.6) per 100 person-years. Features of subjects with atypical myeloproliferative disorder differed markedly from those of 546 subjects with prefibrotic myelofibrosis. Conclusion: Our data indicate that these 15 persons have a distinct myeloproliferative neoplasm. We propose naming this new disorder clonal megakaryocyte dysplasia with normal blood values. [ABSTRACT FROM AUTHOR]

Published
2022
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16. Splenic and Nodal marginal zone lymphomas are indolent disorders at high hipatitis C virus seroprevalence with distinct presenting features but similar Morphologic and Phenotypic profiles

Author

Arcaini, Luca, Paulli, Marco, Boveri, Emanuela, Vallisa, Daniele, Bernuzzi, Patrizia, Orlandi, Ester, Incardona, Paolo, Brusamolino, Ercole, Passamonti, Francesco, Burcheri, Sara, Schena, Claudia, Pascutto, Cristiana, Cavanna, Luigi, Magrini, Umberto, and Lazzarino, Mario

Subjects
Non-Hodgkin's lymphomas -- Care and treatment, Non-Hodgkin's lymphomas -- Research, Lymphatic diseases -- Research, Lymphomas -- Research, Health
Published
2004

17. MYH9-related disease: May-Hegglin anomaly, Sebastian syndrome, Fechtner syndrome, and Epstein syndrome are not distinct entities but represent a variable expression of a single illness

Author

Seri, Marco, Pecci, Alessandro, Di Bari, Filomena, Cusano, Roberto, Savino, Maria, Panza, Emanuele, Nigro, Alessandra, Noris, Patrizia, Gangarossa, Simone, Rocca, Bianca, Gresele, Paolo, Bizzaro, Nicola, Malatesta, Paola, Koivisto, Pasi A., Longo, Ilaria, Musso, Roberto, Pecoraro, Carmine, Iolascon, Achille, Magrini, Umberto, Soriano, Juan Rodriguez, Renieri, Alessandra, Ghiggeri, Gian Marco, Ravazzolo, Roberto, Balduini, Carlo L., and Savoia, Anna

Subjects
Thrombocytopenia -- Development and progression, Thrombocytopenia -- Genetic aspects, Thrombocytopenia -- Identification and classification
Published
2003

20. Thrombocytopenia, giant platelets, and leukocyte inclusion bodies (May-Hegglin anomaly): clinical and laboratory findings

Author

Noris, Patrizia, Spedini, Pierangelo, Belletti, Simona, Magrini, Umberto, and Balduini, Carlo L.

Subjects
Thrombocytopenia -- Diagnosis, Genetic disorders -- Diagnosis, Health, Health care industry
Abstract

PURPOSE: May-Hegglin anomaly is a rare hereditary condition characterized by the triad of thrombocytopenia, giant Platelets, and inclusion bodies in leukocytes. Clinical features and the pathogenesis of bleeding in this disease are poorly defined. PATIENTS AND METHODS: From 1988 to 1996 we studied 15 new May-Hegglin anomaly patients from 7 unrelated Italian families. In addition to clinical examination and routine laboratory testing, we measured bleeding time, platelet aggregation and release reaction, and platelet staining for tubulin, and performed ultrastructural study of polymorphonuclear leukocytes. RESULTS: Although the mean age of our patients was 33 years, May-Hegglin anomaly had not been previously recognized in any of them. Bleeding diatheses ranged from severe to absent, and platelet count from 26 to 178 X [10.sup.9]/L. No correlation was found between bleeding tendency and platelet count. Previous therapy with corticosteroids, high-dose immunoglobulins, and splenectomy had no effect on platelet count or bleeding diathesis. Desmopressin infusion greatly shortened the bleeding time in the most severely affected patient. The in vitro function of platelets was normal except for the absence of shape change in all subjects and defective response to epinephrine in 8 of 15 patients. Platelet tubulin was distributed unevenly instead of being organized in a circumferential band at the cell periphery. CONCLUSION: The diagnosis of May-Hegglin is easily missed, and its frequency is probably underestimated. A qualitative defect of platelets may be responsible for mild bleeding diathesis even in the absence of thrombocytopenia, while severe bleeding results from both qualitative and quantitative platelet defects. May-Hegglin anomaly should be suspected whenever a patient has a low platelet count or a bleeding diathesis of unknown origin. Am J Med. 1998;104:355-360. [C] 1998 by Excerpta Medica, Inc.

Published
1998

22. Primary gastric CD30 (Ki-1)-positive large cell non-Hodgkin's lymphomas: a clinicopathologic analysis of six cases

Author

Paulli, Marco, Rosso, Renato, Kindl, Sandra, Boveri, Emanuela, Bonoldi, Emanuela, Stracca, Vincenzo, Motta, Teresio, Arrigoni, Gianguido, Lazzarino, Mario, Menestrina, Fabio, and Magrini, Umberto

Subjects
Non-Hodgkin's lymphomas -- Prognosis, Tumor antigens, Health
Abstract

Background. The CD30/Ki-1 antigen characterizes a series of non-Hodgkin's lymphomas (NHL) predominantly showing anaplastic large cell (ALCL) morphology and frequently involving the skin and other extranodal sites. In cutaneous large cell lymphomas, the CD30 expression was indicated as a favorable prognostic marker independently from cytology, anaplastic versus nonanaplastic. The stomach is the most common site of extranodal lymphomas in the adult population, but primary gastric CD30+ lymphomas have been reported rarely. Methods. The clinical, morphologic, and immunohistochemical features of six cases with primary CD30/Ki-1+ gastric large cell lymphomas were analyzed. Results. The mean age of patients was 64 years with a prevalence of women (M:F ration = 1:2). Patients were assigned to Stage IE or IIE. Three of them died of disease, whereas the others are still alive (mean follow-up, 18 months). Three of six cases had ALCL morphology, whereas other cases had centroblastic, immunoblastic, and high-grade mucosa-associated lymphoid tissue lymphoma. Immunohistochemistry revealed a B-cell phenotype in three of six cases; a T-cell phenotype in one of six cases; and a null, non-B, non-T phenotype in two of six cases. Conclusions. Within CD30+ primary gastric large cell lymphomas, cytology, anaplastic versus nonanaplastic, did not affect clinical presentation and/or prognosis. The survival rate of the patients studied is in keeping with literature reports regarding prognosis of primary high-grade gastric NHL. The findings suggest that clinical behavior of this extranodal lymphoma is more closely related to clinical symptoms and initial stage of disease than to CD30 expression.

Published
1994

23. An atypical myeloproliferative disorder with high thrombotic risk and slow disease progression

Author

Barosi, Giovanni, Buratti, Alberto, Costa, Attilia, Liberato, Lucio N., Balduini, Carlo, Cazzola, Mario, Rosti, Vittorio, Magrini, Umberto, and Ascari, Edoardo

Subjects
Splenomegaly -- Causes of, Thrombosis -- Causes of, Myeloproliferative disorders -- Case studies, Health
Abstract

The chronic myeloproliferative disorders (CMD) represent a group of diseases which have in common the abnormal proliferation of some cell contributing to the blood. All are thought to arise when a single cell in the bone marrow goes awry and multiplies without adherence to its original 'plan'. If the cell is a precursor of red blood cells, then polycythemia vera is the result. If the precursor cell is a megakaryocyte, which forms platelets (thrombocytes), then idiopathic thrombocythemia is the result. Other myeloproliferative syndromes include myelofibrosis with myeloid metaplasia and chronic myelogenous leukemia. While these classes represent many of the cases observed in the clinic, there are intermediate forms which share features of two typical classes. Furthermore, there seem to be variant forms which have features of chronic myeloproliferative disorders but are not immediately recognizable as such. The authors describe the important features of 18 cases that failed to satisfy the diagnostic criteria for the typical forms of CMD. All 18 cases occurred in patients younger than 46 years of age. In 11 cases, the patients had signs such as enlarged spleen, excessive bone marrow growth, and a mild increase of platelets or white blood cells in the blood. Rare immature bone marrow cells appeared in the blood of some of the 11 patients. In the other seven patients, a diagnosis of CMD was made only after an enlarged spleen was removed and blood clotting complications began. In two cases, these complications were fatal. Fifteen patients are alive and appear stable; in 12 chemotherapeutic treatment to kill the renegade cells has not proved necessary. These cases illustrate the features of a slowly progressing chronic myeloproliferative disorder affecting younger patients. They also illustrate the dangers of removing an enlarged spleen when the cause of the splenomegaly has not been determined. (Consumer Summary produced by Reliance Medical Information, Inc.)

Published
1991

24. Deregulated Genes in Hematopoietic Stem Cells Isolated from Spleen of Patients with Myelofibrosis

Author

Catarsi, Paolo, primary, Cordero, Francesca, additional, Ferrero, Giulio, additional, Beccuti, Marco, additional, Poletto, Valentina, additional, Bonetti, Elisa, additional, Villani, Laura, additional, Massa, Margherita, additional, Fois, Gabriela, additional, Campanelli, Rita, additional, Magrini, Umberto, additional, Rosti, Vittorio, additional, and Barosi, Giovanni, additional

Published
2016
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25. Spleen neoangiogenesis in patients with myelofibrosis with myeloid metaplasia

Author

Ramaioli Isabella, Marchetti Monia, Viarengo G. Luca, Massa Margherita, Bazzan Mario, Giovanni Barosi, Necchi Vittorio, Campanelli Rita, Rosti Vittorio, Magrini Umberto, and Pecci Alessandro

Subjects
Pathology, medicine.medical_specialty, education.field_of_study, Myeloid, business.industry, medicine.medical_treatment, Splenectomy, Population, CD34, Spleen, Hematology, medicine.disease, Haematopoiesis, medicine.anatomical_structure, medicine, Bone marrow, education, Myelofibrosis, business
Abstract

Summary Neoangiogenesis is an integral component of bone marrow myeloproliferation in patients with myelofibrosis with myeloid metaplasia (MMM). As extramedullary haematopoiesis is a constitutive feature of MMM, we studied spleen neoangiogenesis by a computerized image analysis in MMM patients. Compared with five normal subjects, spleen CD34-staining capillary vascular density (CVD) was 2·1–3·03 times higher than the upper range of normal in six of the 15 (40%) MMM patients. CD8-staining sinusoidal vascular density (SVD) was constantly normal or lesser than normal and was inversely correlated with CVD (R = −0·53; P = 0·04). In MMM patients who did not receive cytoreductive or radiation therapy in the month before splenectomy (n = 9), the CVD was a significant determinant of spleen size (R = 0·88; P = 0·04). In MMM patients, the number of spleen CD34+ haematopoietic stem cells was increased from 1·2 to 98 times the upper limit of normal, and predicted the expansion of CVD (R = 0·57; P = 0·03). A population of cells expressing the CD34+/CD133+/VEGFR-2+ angiopoietic phenotype was present in the blood and spleen of five of seven patients. These results document that neoangiogenesis is an integral component of spleen re-localization of haematopoietic stem cells and suggest a cellular mechanism for spleen neoangiogenesis.

Published
2004

26. Increased plasma nicotinamide phosphoribosyltransferase is associated with a hyperproliferative phenotype and restrains disease progression in MPN-associated myelofibrosis

Author

Rosti, Vittorio, primary, Campanelli, Rita, additional, Massa, Margherita, additional, Viarengo, Gianluca, additional, Villani, Laura, additional, Poletto, Valentina, additional, Bonetti, Elisa, additional, Catarsi, Paolo, additional, Magrini, Umberto, additional, Grolla, Ambra A., additional, Travelli, Cristina, additional, Genazzani, Armando A., additional, and Barosi, Giovanni, additional

Published
2016
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27. V617FJAK2-Positive Endothelial Cells Are Present in Bone Marrow Neovessels of Patients with Myelofibrosis and Could Derive from the Transdifferentiation of Mutated Hematopoietic Cells

Author

Villani, Laura, primary, Campanelli, Rita, additional, Gaudioso, Gabriella, additional, Poletto, Valentina, additional, Bonetti, Elisa, additional, Catarsi, Paolo, additional, Fois, Gabriela, additional, Erba, Benedetta G, additional, Massa, Margherita, additional, Magrini, Umberto, additional, Barosi, Giovanni, additional, Gianelli, Umberto, additional, and Rosti, Vittorio, additional

Published
2015
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28. Il danno epatico è una nuova caratteristica della Malattia MYH9-correlata

Author

Zaninetti, Carlo, Pecci, Alessandro, Biino, Ginevra, Bozzi, Valeria, Fierro, Tiziana, Noris, Patrizia, Magrini, Umberto, Gresele, Paolo, and Balduini, Carlo Luigi

Abstract

La Malattia MYH9-correlata (MYH9-RD) è un raro disordine sindromico, a trasmissione autosomico dominante, caratterizzato da piastrinopenia congenita associata al rischio di sviluppare nefropatia, sordità neurosensoriale e cataratta presenile. Nel corso dello studio del database di pazienti arruolati al Registro Italiano della MYH9-RD, abbiamo osservato che diversi soggetti presentavano una elevazione non spiegata dei markers di danno epatocellulare. Scopo del presente studio è stato quello di verificare l’ipotesi che il danno epatico sia una manifestazione non riconosciuta della MYH9-RD e di valutarne il significato clinico e prognostico. Le concentrazioni di AST, ALT e GGT osservate nei pazienti consecutivamente arruolati al Registro sono state confrontate con quelle determinate in tre casistiche di controllo: una composta dai soggetti partecipanti ad un ampio studio di popolazione condotto in un isolato geografico italiano e due casistiche di pazienti con piastrinopenia cronica derivante da cause diverse dalle mutazioni di MYH9. Trentotto dei 75 pazienti con MYH9-RD valutabili (50.7%) mostravano elevazione di ALT e/o AST, mentre la GGT era incrementata nel 27% dei casi (17/63). L’incremento medio era da 1.9±0.7 a 2.7±1.6 volte il limite superiore dell’intervallo di normalità. La prevalenza dell’alterazione di tali markers nei pazienti con MYH9-RD era significativamente superiore rispetto a quella osservata in ciascuna delle tre casistiche di controllo, con valori di odds ratio compresi tra 8.2 (95% CIs 2.2-44.8) e 24.7 (14.8-40.8). Il follow-up clinico e ulteriori studi della funzione epatica non hanno mostrato alcun significativo danno strutturale d’organo né la tendenza all’evoluzione verso l’insufficienza epatica. Il danno epatico è quindi una caratteristica frequente, e sinora non riconosciuta, del quadro sindromico della MYH9-RD; allo stato attuale, tale alterazione non sembra possedere un significato prognostico sfavorevole., Bollettino della Società Medico Chirurgica di Pavia, Vol 125, N° 4 (2012)

Published
2013
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29. Somatic and Germ-Line Molecular Characteristics Of Prefibrotic Myelofibrosis

Author

Barosi, Giovanni, primary, Bonetti, Elisa, additional, Catarsi, Paolo, additional, Massa, Margherita, additional, Campanelli, Rita, additional, Poletto, Valentina, additional, Viarengo, Gianluca, additional, Villani, Laura, additional, Fois, Gabriela, additional, Biamonte, Flavia, additional, Rotunno, Giada, additional, Guglielmelli, Paola, additional, Magrini, Umberto, additional, Vannucchi, Alessandro M, additional, and Rosti, Vittorio, additional

Published
2013
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30. Evidence that Prefibrotic Myelofibrosis Is Aligned along a Clinical and Biological Continuum Featuring Primary Myelofibrosis

Author

Barosi, Giovanni, primary, Rosti, Vittorio, additional, Bonetti, Elisa, additional, Campanelli, Rita, additional, Carolei, Adriana, additional, Catarsi, Paolo, additional, Isgrò, Antonina M., additional, Lupo, Letizia, additional, Massa, Margherita, additional, Poletto, Valentina, additional, Viarengo, Gianluca, additional, Villani, Laura, additional, and Magrini, Umberto, additional

Published
2012
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31. Prefibrotic Myelofibrosis (PreMF) Belongs to a Continuum of Epidemiological, Clinical and Histological Characteristics Featuring Primary Myelofibrosis (PMF)

Author

Barosi, Giovanni, primary, Rosti, Vittorio, additional, Campanelli, Rita, additional, Catarsi, Paolo, additional, Isgrò, Antonina M., additional, Lupo, Letizia, additional, Massa, Margherita, additional, Poletto, Valentina, additional, Viarengo, Gianluca, additional, Villani, Laura, additional, and Magrini, Umberto, additional

Published
2011
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33. High Frequency of Circulating Endothelial Colony Forming Cells (ECFCs) in Myeloproliferative Neoplasms (MPNs) Is Associated with Diagnosis of Prefibrotic Myelofibrosis, History of Splanchnic Vein Thrombosis, and Vascular Splenomegaly.

Author

Rosti, Vittorio, primary, Bonetti, Elisa, additional, Bergamaschi, Gaetano, additional, Campanelli, Rita, additional, Gattoni, Elisabetta, additional, Po, Sonia Lo, additional, Magrini, Umberto, additional, Massa, Margherita, additional, Vannucchi, Alessandro, additional, Viarengo, Gianluca, additional, Guglielmelli, Paola, additional, Primignani, Massimo, additional, and Barosi, Giovanni, additional

Published
2009
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35. Bone marrow and blood involvement by non-Hodgkin's lymphoma: A study of clinicopathologic correlations and prognostic significance in relationship to the Working Formulation

Author

Morra, Enrica, primary, Lazzarino, Mario, additional, Castello, Alessandro, additional, Inverardi, Daniela, additional, Coci, Anna, additional, Pagnucco, Guido, additional, Orlandi, Ester, additional, Merante, Serena, additional, Magrini, Umberto, additional, Zei, Gianna, additional, and Bernasconi, Carlo, additional

Published
2009
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36. Clinical Relevance of Bone Marrow Fibrosis and CD34-Positive Cell Clusters in Primary Myelodysplastic Syndromes

Author

Della Porta, Matteo Giovanni, primary, Malcovati, Luca, additional, Boveri, Emanuela, additional, Travaglino, Erica, additional, Pietra, Daniela, additional, Pascutto, Cristiana, additional, Passamonti, Francesco, additional, Invernizzi, Rosangela, additional, Castello, Alessandro, additional, Magrini, Umberto, additional, Lazzarino, Mario, additional, and Cazzola, Mario, additional

Published
2009
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37. Clinical, radiological, and biochemical features of a bilateral buttock amyloidoma emerging after 27 years of hemodialysis

Author

Montagna, Giovanni, primary, Raimondi, Sara, additional, Moro, Guido, additional, Uggetti, Carla, additional, Relini, Annalisa, additional, Magrini, Umberto, additional, Esposito, Gennaro, additional, Giorgetti, Sofia, additional, Congi, Luigi, additional, Mosconi, Mario, additional, Galli, Giovanni, additional, Villa, Giuseppe, additional, Segagni, Siro, additional, Donadei, Simona, additional, Obici, Laura, additional, Merlini, Giampaolo, additional, Stoppini, Monica, additional, and Bellotti, Vittorio, additional

Published
2009
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38. Bone marrow microvessel density in chronic myeloproliferative disorders: a study of 115 patients with clinicopathological and molecular correlations

Author

Boveri, Emanuela, primary, Passamonti, Francesco, additional, Rumi, Elisa, additional, Pietra, Daniela, additional, Elena, Chiara, additional, Arcaini, Luca, additional, Pascutto, Cristiana, additional, Castello, Alessandro, additional, Cazzola, Mario, additional, Magrini, Umberto, additional, and Lazzarino, Mario, additional

Published
2007
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40. B-cell Posttransplant Lymphoproliferative Disorders in Heart and/or Lungs Recipients: Clinical and Molecular-Histogenetic Study of 17 Cases from a Single Institution

Author

Lucioni, Marco, primary, Capello, Daniela, additional, Riboni, Roberta, additional, Ippoliti, Giovanbattista, additional, Campana, Carlo, additional, Bandiera, Laura, additional, Arcaini, Luca, additional, Rossi, Davide, additional, Cerri, Michaela, additional, Dionigi, Paolo, additional, Lazzarino, Mario, additional, Magrini, Umberto, additional, Viganò, Mario, additional, Gaidano, Gianluca, additional, and Paulli, Marco, additional

Published
2006
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42. Relationship between JAK2 V617F Mutation Status and Constitutive Mobilization of CD34-Positive Cells into Peripheral Blood in Patients with Chronic Myeloproliferative Disorder.

Author

Passamonti, Francesco, primary, Rumi, Elisa, primary, Boveri, Emanuela, primary, Pietra, Daniela, primary, Vanelli, Laura, primary, Arcaini, Luca, primary, Porta, Matteo G. Della, primary, Pascutto, Cristiana, primary, Magrini, Umberto, primary, Lazzarino, Mario, primary, and Cazzola, Mario, primary

Published
2005
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45. MYH9-Related Disease

Author

Seri, Marco, primary, Pecci, Alessandro, additional, Di Bari, Filomena, additional, Cusano, Roberto, additional, Savino, Maria, additional, Panza, Emanuele, additional, Nigro, Alessandra, additional, Noris, Patrizia, additional, Gangarossa, Simone, additional, Rocca, Bianca, additional, Gresele, Paolo, additional, Bizzaro, Nicola, additional, Malatesta, Paola, additional, Koivisto, Pasi A., additional, Longo, Ilaria, additional, Musso, Roberto, additional, Pecoraro, Carmine, additional, Iolascon, Achille, additional, Magrini, Umberto, additional, Soriano, Juan Rodriguez, additional, Renieri, Alessandra, additional, Ghiggeri, Gian Marco, additional, Ravazzolo, Roberto, additional, Balduini, Carlo L., additional, and Savoia, Anna, additional

Published
2003
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46. Splenic and nodal marginal zone lymphomas are indolent disorders at high hepatitis C virus seroprevalence with distinct presenting features but similar morphologic and phenotypic profiles

Author

Arcaini, Luca, primary, Paulli, Marco, additional, Boveri, Emanuela, additional, Vallisa, Daniele, additional, Bernuzzi, Patrizia, additional, Orlandi, Ester, additional, Incardona, Paolo, additional, Brusamolino, Ercole, additional, Passamonti, Francesco, additional, Burcheri, Sara, additional, Schena, Claudio, additional, Pascutto, Cristiana, additional, Cavanna, Luigi, additional, Magrini, Umberto, additional, and Lazzarino, Mario, additional

Published
2003
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47. Primary cutaneous large B-cell lymphoma of the leg: Histogenetic analysis of a controversial clinicopathologic entity

Author

Paulli, Marco, primary, Viglio, Alessandra, additional, Vivenza, Daniela, additional, Capello, Daniela, additional, Rossi, Davide, additional, Riboni, Roberta, additional, Lucioni, Marco, additional, Incardona, Paolo, additional, Boveri, Emanuela, additional, Bellosta, Mario, additional, Orlandi, Ester, additional, Borroni, Giovanni, additional, Lazzarino, Mario, additional, Berti, Emilio, additional, Alessi, Elvio, additional, Magrini, Umberto, additional, and Gaidano, Gianluca, additional

Published
2002
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48. Mediastinal B-cell lymphoma: A study of its histomorphologic spectrum based on 109 cases

Author

Paulli, Marco, primary, Sträter, Jörn, additional, Gianelli, Umberto, additional, Rousset, Marie-Thérèse, additional, Gambacorta, Marcello, additional, Orlandi, Ester, additional, Klersy, Catherine, additional, Lavabre-Bertrand, Thierry, additional, Morra, Enrica, additional, Manegold, Christian, additional, Lazzarino, Mario, additional, Magrini, Umberto, additional, and Möller, Peter, additional

Published
1999
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49. Cutaneous CD30+ lymphoproliferative disorders: Expression of bcl-2 and proteins of the tumor necrosis factor receptor superfamily

Author

Paulli, Marco, primary, Berti, Emilio, additional, Boveri, Emanuela, additional, Kindl, Sandra, additional, Bonoldi, Emanuela, additional, Gambini, Claudio, additional, Rosso, Renato, additional, Borroni, Giovanni, additional, Straccapansa, Vincenzo, additional, Magrini, Umberto, additional, DeCoteau, John E, additional, Krammer, Peter H, additional, Moller, Peter, additional, and Kadin, Marshall E, additional

Published
1998
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50. ALK Expression Defines a Distinct Group of T/Null Lymphomas (“ALK Lymphomas”) with a Wide Morphological Spectrum

Author

Falini, Brunangelo, primary, Bigerna, Barbara, additional, Fizzotti, Marco, additional, Pulford, Karen, additional, Pileri, Stefano A., additional, Delsol, Georges, additional, Carbone, Antonino, additional, Paulli, Marco, additional, Magrini, Umberto, additional, Menestrina, Fabio, additional, Giardini, Roberto, additional, Pilotti, Silvana, additional, Mezzelani, Alessandra, additional, Ugolini, Barbara, additional, Billi, Monia, additional, Pucciarini, Alessandra, additional, Pacini, Roberta, additional, Pelicci, Pier-Giuseppe, additional, and Flenghi, Leonardo, additional

Published
1998
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