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1. Influences of amyloid-β and tau on white matter neurite alterations in dementia with Lewy bodies

8. Support vector machine learning and diffusion-derived structural networks predict amyloid quantity and cognition in adults with Down's syndrome

23. Texture‐based morphometry in relation to apolipoprotein ε4 genotype, ageing and sex in a midlife population.

25. Brain age gap, dementia risk factors and cognition in middle age

26. Investigating the impact of APOE4 genotype on the association between cerebral blood flow and haematological properties in mid‐life adults

27. Multi‐modal MR and synaptic PET changes in dementia with Lewy bodies

28. Apoe ε4 exacerbates age‐dependent decline of cortical microstructural changes in cognitively normal midlife individuals: the PREVENT‐Dementia and ALFA studies

29. Alzheimer’s disease biomarkers of amyloid and tau influence white matter neurite alterations in dementia with Lewy bodies: a NODDI study

30. DNA methylation alterations in Lewy Body Dementia

32. In vivo coupling of tau pathology and cortical thinning in Alzheimer's disease

33. Longitudinal associations between β-amyloid and cortical thickness in mild cognitive impairment

39. The PREVENT dementia programme: baseline demographic, lifestyle, imaging and cognitive data from a midlife cohort study investigating risk factors for dementia.

40. Microglial activation in the frontal cortex predicts cognitive decline in frontotemporal dementia

43. The PREVENT Dementia programme: Baseline demographic, lifestyle, imaging and cognitive data from a midlife cohort study investigating risk factors for dementia

49. APOE ɛ4 exacerbates age-dependent deficits in cortical microstructure.

50. Microglial activation in the frontal cortex predicts cognitive decline in frontotemporal dementia

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