38 results on '"Malaney, Prerna"'
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2. Assessing the role of intrinsic disorder in RNA-binding protein function: hnRNP K as a case study
3. Insight into the mechanism of AML del(9q) progression: hnRNP K targets the myeloid master regulators CEBPA (C/EBPα) and SPI1 (PU.1)
4. S179: HNRNPK OVEREXPRESSION DRIVES NUCLEOLAR ABERRANCIES CAUSING RIBOSOMPATHIES
5. The nucleolar aberrancies that drive ribosome impairment induced by RNA binding proteins are hallmarks of aging
6. Creating accessibility in academic negotiations
7. Identification of intrinsically disordered regions in PTEN and delineation of its function via a network approach
8. PTEN proteoforms in biology and disease
9. Adar1 deletion causes degeneration of the exocrine pancreas via Mavs-dependent interferon signaling
10. One mouse, one patient paradigm: New avatars of personalized cancer therapy
11. Assay Development and Small Molecule Screens for Inhibitors of an RNA Binding Protein Implicated in Hematologic Malignancies
12. Heterogeneous nuclear ribonucleoprotein K is overexpressed in acute myeloid leukemia and causes myeloproliferation in mice via altered Runx1 splicing
13. Loss of Phosphatase and Tensin Homolog (PTEN) Induces Leptin-mediated Leptin Gene Expression: FEED-FORWARD LOOP OPERATING IN THE LUNG
14. Targeting the Oncogenic Functions of an RNA-Binding Protein Involved in Hematologic Malignancies
15. Increased hnRNP K Expression Impacts Myelopoiesis By Altering RUNX1 Splicing
16. AXL/MERTK inhibitor ONO-7475 potently synergizes with venetoclax and overcomes venetoclax resistance to kill FLT3-ITD acute myeloid leukemia
17. Adar1 deletion causes degeneration of exocrine pancreas via Mavs-dependent interferon signaling
18. Hnrnpk Overexpression Drives Nucleolar Aberrancies Causing Ribosomopathies
19. A Potential Link between hnRNP K and Cebpα: Implications for Ribosomal Dysfunction in Acute Myeloid Leukemia (AML)
20. The Eµ-hnRNP K Murine Model of Lymphoma: Novel Insights into the Role of hnRNP K in B-Cell Malignancies
21. Heterogeneous nuclear ribonucleoprotein K is overexpressed in acute myeloid leukemia and causes myeloproliferative disease in mice via altered Runx1 splicing
22. Abstract 4826: Enhancement of PTEN activity via peptidomimetics
23. Uncovering the Role of RNA-Binding Protein hnRNP K in B-Cell Lymphomas
24. hnRNP K Overexpression Drives Myeloid Malignancy Via Interaction with RUNX1
25. Combination Therapy with BTK Inhibitor Plus Anti-PD-1 Antibody Results in a Hyperprogressor Phenotype in a Mouse Model of CLL
26. hnRNP K: A Regulator of Global Transcription and Translation That Drives Lymphomagenesis
27. PTEN Physically Interacts with and Regulates E2F1-mediated Transcription in Lung Cancer
28. hnRNPK as a Driver of Lymphomagenesis
29. Uncovering the Role of RNA-Binding Protein hnRNP K in B-Cell Lymphomas.
30. Abstract 3682: Phosphorylation mediated conformational changes defines nuclear role of phosphatase and tensin homology (PTEN) in tumor suppression
31. hnRNP K: A Driver of Myeloid Malignancy
32. A unified nomenclature and amino acid numbering for human PTEN.
33. Abstract B09: Loss of PTEN cooperates with mutant KRAS initiating EMT and increased stemness in a mouse model of lung cancer
34. A Unified Nomenclature and Amino Acid Numbering for Human PTEN
35. Pathological Unfoldomics of Uncontrolled Chaos: Intrinsically Disordered Proteins and Human Diseases
36. Intrinsic Disorder in PTEN and its Interactome Confers Structural Plasticity and Functional Versatility
37. The PTEN Long N-tail is intrinsically disordered: increased viability for PTEN therapy
38. AXL/MERTK inhibitor ONO-7475 potently synergizes with venetoclax and overcomes venetoclax resistance to kill FLT3 -ITD acute myeloid leukemia.
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