229 results on '"Manczak, Maria"'
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2. Aqua-soluble DDQ reduces the levels of Drp1 and Aβ and inhibits abnormal interactions between Aβ and Drp1 and protects Alzheimer’s disease neurons from Aβ- and Drp1-induced mitochondrial and synaptic toxicities
3. Pathogenicity of Myelin Basic Protein in Anterior Uveitis : Pathogenicity of MBP in uveitis
4. A partial reduction of Drp1 improves cognitive behavior and enhances mitophagy, autophagy and dendritic spines in a transgenic Tau mouse model of Alzheimer disease
5. A partial reduction of Drp1 enhances mitophagy, autophagy, mitochondrial biogenesis, dendritic spines and synaptic activity in a transgenic Tau mouse model of Alzheimer disease
6. Maternal Exercise Activates Genes Associated With Mitochondrial Biogenesis In Fetal Myocardium Of Mouse: 2497 Board #20 June 3, 9: 30 AM - 11: 00 AM
7. Mitochondrial division inhibitor 1 protects against mutant huntingtin-induced abnormal mitochondrial dynamics and neuronal damage in Huntingtonʼs disease
8. Mitochondrial Dysfunctioning Induced by Hyperglycemia in the Liver Tissues of Diabetic Mice, TALLYHO/JngJ Strain and Ameliorative Action of a Small Peptide, SS31
9. partial reduction of Drp1 improves cognitive behavior and enhances mitophagy, autophagy and dendritic spines in a transgenic Tau mouse model of Alzheimer disease.
10. Protective effects of a mitochondria-targeted small peptide SS31 against hyperglycemia-induced mitochondrial abnormalities in the liver tissues of diabetic mice, Tallyho/JngJ mice
11. Interaction of the nuclear matrix protein NAKAP with HypA and huntingtin: Implications for nuclear toxicity in Huntington’s disease pathogenesis
12. Differential expression of oxidative phosphorylation genes in patients with Alzheimer’s disease: Implications for early mitochondrial dysfunction and oxidative damage
13. Mitochondria-Targeted Small Peptide, SS31 Ameliorates Diabetes Induced Mitochondrial Dynamics in Male TallyHO/JngJ Mice
14. Abnormal interaction of VDAC1 with amyloid beta and phosphorylated tau causes mitochondrial dysfunction in Alzheimerʼs disease
15. Mitochondria-targeted catalase reduces abnormal APP processing, amyloid β production and BACE1 in a mouse model of Alzheimerʼs disease: implications for neuroprotection and lifespan extension
16. Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimerʼs disease neurons: implications for mitochondrial dysfunction and neuronal damage
17. Mutant huntingtinʼs interaction with mitochondrial protein Drp1 impairs mitochondrial biogenesis and causes defective axonal transport and synaptic degeneration in Huntingtonʼs disease
18. Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimerʼs disease
19. Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimerʼs disease: implications for neuronal damage
20. Abnormal mitochondrial dynamics, mitochondrial loss and mutant huntingtin oligomers in Huntingtonʼs disease: implications for selective neuronal damage
21. Protective effects of BACE1 inhibitory ligand molecules against amyloid beta-induced synaptic and mitochondrial toxicities in Alzheimer’s disease
22. Neutralization of granulocyte macrophage colony-stimulating factor decreases amyloid beta 1-42 and suppresses microglial activity in a transgenic mouse model of Alzheimerʼs disease
23. Mitochondria are a direct site of Aβ accumulation in Alzheimerʼs disease neurons: implications for free radical generation and oxidative damage in disease progression
24. Time-course of mitochondrial gene expressions in mice brains: implications for mitochondrial dysfunction, oxidative damage, and cytochrome c in aging
25. Gene expression profiles of transcripts in amyloid precursor protein transgenic mice: up-regulation of mitochondrial metabolism and apoptotic genes is an early cellular change in Alzheimerʼs disease
26. Distribution of LILRA3 ( ILT6/LIR4) deletion in psoriatic patients and healthy controls
27. Corrigendum: Mitochondrial division inhibitor 1 reduces dynamin-related protein 1 and mitochondrial fission activity
28. Mitochondrial division inhibitor 1 reduces dynamin-related protein 1 and mitochondrial fission activity
29. Epitope recognition and T cell receptors in recurrent autoimmune anterior uveitis in Lewis rats immunized with myelin basic protein
30. Protective effects of BACE1 inhibitory ligand molecules against amyloid beta-induced synaptic and mitochondrial toxicities in Alzheimer's disease.
31. Current Status of Healthy Aging and Dementia Research: A Symposium Summary.
32. Mutant APP and amyloid beta-induced defective autophagy, mitophagy, mitochondrial structural and functional changes and synaptic damage in hippocampal neurons from Alzheimer’s disease
33. Synergistic Protective Effects of Mitochondrial Division Inhibitor 1 and Mitochondria-Targeted Small Peptide SS31 in Alzheimer’s Disease
34. Hippocampal mutant APP and amyloid beta-induced cognitive decline, dendritic spine loss, defective autophagy, mitophagy and mitochondrial abnormalities in a mouse model of Alzheimer’s disease
35. Protective Effects of Indian Spice Curcumin Against Amyloid-β in Alzheimer’s Disease
36. Hippocampal phosphorylated tau induced cognitive decline, dendritic spine loss and mitochondrial abnormalities in a mouse model of Alzheimer’s disease
37. Mitochondria-Division Inhibitor 1 Protects Against Amyloid-β induced Mitochondrial Fragmentation and Synaptic Damage in Alzheimer’s Disease
38. Mitochondria-targeted small molecule SS31: a potential candidate for the treatment of Alzheimer’s disease
39. Maternal exercise upregulates mitochondrial gene expression and increases enzyme activity of fetal mouse hearts
40. Protective Effects of a Natural Product, Curcumin, against Amyloid β Induced Mitochondrial and Synaptic Toxicities in Alzheimer'S Disease
41. Protective effects of reduced dynamin-related protein 1 against amyloid beta-induced mitochondrial dysfunction and synaptic damage in Alzheimer’s disease
42. Reduced dynamin-related protein 1 protects against phosphorylated Tau-induced mitochondrial dysfunction and synaptic damage in Alzheimer’s disease
43. Mitochondrial division inhibitor 1 reduces dynamin-related protein 1 and mitochondrial fission activity.
44. P2-101: Protective Effects of Selective Serotonin Reuptake Inhibitor and Neurosteroids Against Alzheimer's Disease Neurons
45. Mitochondria-targeted molecules MitoQ and SS31 reduce mutant huntingtin-induced mitochondrial toxicity and synaptic damage in Huntington's disease
46. Garrison Institute on Aging: A New Hope for Elderly Individuals and Patients with Alzheimer’s Disease
47. Synergistic Protective Effects of Mitochondrial Division Inhibitor 1 and Mitochondria-Targeted Small Peptide SS31 in Alzheimer's Disease.
48. Hippocampal phosphorylated tau induced cognitive decline, dendritic spine loss and mitochondrial abnormalities in a mouse model of Alzheimer's disease.
49. MitoQ, a mitochondria-targeted antioxidant, delays disease progression and alleviates pathogenesis in an experimental autoimmune encephalomyelitis mouse model of multiple sclerosis
50. RNA silencing of genes involved in Alzheimer's disease enhances mitochondrial function and synaptic activity
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