268 results on '"Mantel, Charlie"'
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2. Mouse hematopoietic cell–targeted STAT3 deletion: stem/progenitor cell defects, mitochondrial dysfunction, ROS overproduction, and a rapid aging–like phenotype
3. CD1d expression on and regulation of murine hematopoietic stem and progenitor cells
4. TIP110/p110nrb/SART3/p110 regulation of hematopoiesis through CMYC
5. Physical Characteristics and Polymerization During Iron Saturation of Lactoferrin, A Myelopoietic Regulatory Molecule with Suppressor Activity
6. Polymerization of Murine Macrophage Inflammatory Protein 1α Inactivates Its Myelosuppressive Effects In vitro: The Active Form is a Monomer
7. Dipeptidylpeptidase 4 negatively regulates colony-stimulating factor activity and stress hematopoiesis
8. AMD3100 and CD26 Modulate Mobilization, Engraftment, and Survival of Hematopoietic Stem and Progenitor Cells Mediated by the SDF-1/CXCL12-CXCR4 Axis
9. Survivin regulates hematopoietic progenitor cell proliferation through p21WAF1/Cip1-dependent and -independent pathways
10. Polymerization of murine macrophage inflammatory protein 1-alpha inactivates its myelosuppressive effects in vitro: the active form is a monomer
11. Chemokine Regulation of Hematopoiesis and the Involvement of Pertussis Toxin-Sensitive Gαi Proteins
12. Chemokines, chemokine receptors and hematopoiesis
13. Physical Characteristics and Polymerization During Iron Saturation of Lactoferrin, A Myelopoietic Regulatory Molecule With Suppressor Activity
14. Cyclosporine May Inhibit the Effect of Extra-Physiologic Oxygen Shock/Stress on Peripheral Blood Stem Cells
15. c-kit associated with the transmembrane 4 superfamily proteins constitutes a functionally distinct subunit in human hematopoietic progenitors
16. Enhancement of intracellular signaling associated with hematopoietic progenitor cell survival in response to SDF-1/CXCL12 in synergy with other cytokines
17. H-Ras Is Involved in the Inside-out Signaling Pathway of Interleukin-3–Induced Integrin Activation
18. p21cip-1/waf-1 Deficiency Causes Deformed Nuclear Architecture, Centriole Overduplication, Polyploidy, and Relaxed Microtubule Damage Checkpoints in Human Hematopoietic Cells
19. Metabolome Profiling of Partial and Fully Reprogrammed Induced Pluripotent Stem Cells
20. Dipeptidyl peptidase 4 (DPP4) truncated factors manifest distinct regulatory functions compared to their full length forms and DPP4 is altered by, and modulates stem cell response to, extra physiologic oxygen shock/stress (EPHOSS)
21. MiR-31/SDHA Axis Regulates Reprogramming Efficiency through Mitochondrial Metabolism
22. Effect of hydrazines on substrate utilization by a strain ofEnterobacter cloacae
23. Adaptation of a soil bacterium to hydrazine propellants
24. The importance of hypoxia and extra physiologic oxygen shock/stress for collection and processing of stem and progenitor cells to understand true physiology/pathology of these cells ex vivo
25. Enhancing Hematopoietic Stem Cell Transplantation Efficacy by Mitigating Oxygen Shock
26. Unique and Unexpected Functional Activities of Hematopoietically Active Cytokines Truncated By Dipeptidylpeptidase-4 (DPP4)
27. Mitigation of a Newly Discovered Phenomenon, Extra Physiologic Oxygen Shock/Stress (EPHOSS), Mediated By the Mitochondria Permeability Transition Pore, Greatly Improves Stem Cell Collection and Transplantation
28. Actions of H-Subunit Ferritin and Lactoferrin as Suppressor Molecules of Myelopoiesis in vitro and in vivo1
29. DPP4 truncation alters the signaling and functional activity of GM-CSF and LL-3 in normal and leukemic hematopoietic cells
30. DPP4 (CD26) Truncation Of GM-CSF and IL-3 Alters Their Signaling and Functional Activity In Normal and Leukemic Hematopoietic Stem and Progenitor Cells
31. Spontaneously DifferentiatedGATA6-Positive Human Embryonic Stem Cells Represent an Important Cellular Step in Human Embryonic Development; They Are Not Just an Artifact of In Vitro Culture
32. Epigenetic Regulation of Nanog by MiR-302 Cluster-MBD2 Completes Induced Pluripotent Stem Cell Reprogramming
33. CD26 Truncation of GM-CSF and IL-3 Alters Their Functional Interactions
34. A ROSy future for metabolic regulation of HSC division
35. Cyclin Dependent Kinase Inhibitors Differentially Modulate Synergistic Cytokine Responsiveness of Hematopoietic Progenitor Cells
36. Superoxide flashes, reactive oxygen species, and the mitochondrial permeability transition pore: potential implications for hematopoietic stem cell function
37. STAT3 Deletion In Hematopoietic Tissue Causes Mitochondrial Dysfunction with Increased ROS Leading to LT-Repopulating Stem Cell Depletion, Erythroid Dysplasia, Peripheral Neutrophilia, and a Rapid Aging-Like Phenotype.
38. Upregulation of nascent mitochondrial biogenesis in mouse hematopoietic stem cells parallels upregulation of CD34 and loss of pluripotency: A potential strategy for reducing oxidative risk in stem cells
39. The Earliest Stages of Loss of Stem Cell Self-Renewal in-Vivo Is Linked to Upregulated Biosynthesis of “Quiet” Mitochondria and Is Influenced by CXCR4 Activation and STAT3 Gene Deletion.
40. Embryonic Stem Cells Bypass Numerous Cell Cycle Checkpoints; Not Just G1.
41. Mouse Hematopoietic Stem Cells, Unlike Human and Mouse Embryonic Stem Cells, Exhibit Checkpoint–Apoptosis Coupling
42. Sirt1, Notch and stem cell "age asymmetry"
43. Sirtuin 1, stem cells, aging, and stem cell aging
44. SIRT1 Regulates Apoptosis and Nanog Expression in Mouse Embryonic Stem Cells by Controlling p53 Subcellular Localization
45. Cells enter a unique intermediate 4N stage, not 4N-G1, after aborted mitosis
46. Mouse Hematopoietic Stem Cells, Unlike Human and Mouse Embryonic Stem Cells, Exhibit Checkpoint-Apoptosis Coupling.
47. Molecular Mechanisms of Spindle Checkpoint-Apoptosis Linkage and Karyotypic Stability in Stem Cells.
48. Oct-4 Is Critical for Survival/Antiapoptosis of Murine Embryonic Stem Cells Subjected to Stress: Effects Associated with Stat3/Survivin
49. Regulation of myeloid progenitor cell proliferation/survival by IL-31 receptor and IL-31
50. Checkpoint-apoptosis uncoupling in human and mouse embryonic stem cells: a source of karyotpic instability
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