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4. Prolonged dipyridamole administration reduces myocardial perfusion defects in experimental chronic Chagas cardiomyopathy

Author

Tanaka, Denise Mayumi, de Oliveira, Luciano Fonseca Lemos, Marin-Neto, José Antônio, Romano, Minna Moreira Dias, de Carvalho, Eduardo Elias Vieira, de Barros Filho, Antonio Carlos Leite, Ribeiro, Fernando Fonseca França, Cabeza, Jorge Mejia, Lopes, Carla Duque, Fabricio, Camila Godoy, Kesper, Norival, Moreira, Henrique Turin, Wichert-Ana, Lauro, Schmidt, André, Higuchi, Maria de Lourdes, Cunha-Neto, Edécio, and Simões, Marcus Vinícius

Published
2019
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8. Chagas Disease

Author

Rassi, Anis, Jr, Rassi, Anis, Marin-Neto, Jose Antonio, Hotez, Peter, Series editor, Franco-Paredes, Carlos, editor, and Santos-Preciado, José Ignacio, editor

Published
2015
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9. Chagas Disease: A Neglected Disease

Author

Marin-Neto, José Antonio, Rassi, Anis, Jr., de Sousa, Andréa Silvestre, Dias, João Carlos Pinto, Rassi, Anis, Andrade, Jadelson, editor, Pinto, Fausto, editor, and Arnett, Donna, editor

Published
2015
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12. Pilot study testing the effect of physical training over the myocardial perfusion and quality of life in patients with primary microvascular angina

Author

de Carvalho, Eduardo Elias Vieira, Santi, Giovani Luiz, Crescêncio, Júlio César, de Oliveira, Luciano Fonseca Lemos, dos Reis, Daniela Caetano Costa, Figueiredo, Alexandre Baldini, Pintya, Antonio Osvaldo, Lima-Filho, Moyses Oliveira, Gallo-Júnior, Lourenço, Marin-Neto, José Antonio, and Simões, Marcus Vinícius

Published
2015
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13. Recommendations for Multimodality Cardiac Imaging in Patients with Chagas Disease: A Report from the American Society of Echocardiography in Collaboration With the InterAmerican Association of Echocardiography (ECOSIAC) and the Cardiovascular Imaging Department of the Brazilian Society of Cardiology (DIC-SBC)

Author

Acquatella, Harry, Asch, Federico M., Barbosa, Marcia M., Barros, Marcio, Bern, Caryn, Cavalcante, Joao L., Correa, Luis Eduardo Echeverria, Lima, Joao, Marcus, Rachel, Marin-Neto, Jose Antonio, Migliore, Ricardo, Milei, Jose, Morillo, Carlos A., Nunes, Maria Carmo Pereira, Vieira, Marcelo Luiz Campos, and Viotti, Rodolfo

Published
2018
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14. The Occluded Artery Trial (OAT) Viability Ancillary Study (OAT-NUC): Influence of infarct zone viability on left ventricular remodeling after percutaneous coronary intervention versus optimal medical therapy alone

Author

Udelson, James E., Pearte, Camille A., Kimmelstiel, Carey D., Kruk, Mariusz, Kufera, Joseph A., Forman, Sandra A., Teresinska, Anna, Bychowiec, Bartosz, Marin-Neto, Jose Antonio, Höchtl, Thomas, Cohen, Eric A., Caramori, Paulo, Busz-Papiez, Benita, Adlbrecht, Christopher, Sadowski, Zygmunt P., Ruzyllo, Witold, Kinan, Debra J., Lamas, Gervasio A., and Hochman, Judith S.

Published
2011
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15. Randomized Trial of Benznidazole for Chronic Chagasʼ Cardiomyopathy

Author

Morillo, Carlos A., Marin-Neto, Jose Antonio, Avezum, Alvaro, Sosa-Estani, Sergio, Rassi, Anis, Jr., Rosas, Fernando, Villena, Erick, Quiroz, Roberto, Bonilla, Rina, Britto, Constança, Guhl, Felipe, Velazquez, Elsa, Bonilla, Laura, Meeks, Brandi, Rao-Melacini, Purnima, Pogue, Janice, Mattos, Antonio, Lazdins, Janis, Rassi, Anis, Connolly, Stuart J., and Yusuf, Salim

Published
2015
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17. Evaluation and treatment of Chagas disease in the United States

Author

Bern Caryn, Montgomery, Susan P., Herwaldt, Barbara L., Rassi Anis., Jr., Marin-Neto, Jose Antonio, Reyes, Pedro A., Salvatella Roberto, Moore, Anne C., Morillo, Carlos, Kirchhoff, Louis V., Gilman, Robert H., Magure, James H., Dantas, Roberto O., and Acquatella, Harry

Subjects
Chagas' disease -- Care and treatment, Chagas' disease -- Diagnosis
Abstract

The article reviews the evaluation and treatment of Chagas disease in the U.S. that relies on serologic methods and proper screening has to offer for patients with such exposure.

Published
2007

23. Prediction of left ventricular wall motion recovery after acute myocardial infarction by Tl-201 gated SPECT: Incremental value of integrated contractile reserve assessment

Author

Simões, Marcus Vinicius, de Almeida-Filho, Oswaldo César, Pintya, Antonio Osvaldo, de Figueiredo, Alexandre Baldini, Antloga, Cleide Marques, Salis, Fernando Vilela, Batista, Nadia de Paula, Lima-Filho, Moysés de Oliveira, Maciel, Benedito Carlos, and Marin-Neto, José Antonio

Published
2002
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25. Recommendations for Multimodality Cardiac Imaging in Patients with Chagas Disease: A Report from the American Society of Echocardiography in collaboration with the InterAmerican Association of Echocardiography (ECOSIAC) and the Cardiovascular Imaging Department of the Brazilian Society of Cardiology (DIC-SBC)

Author

Acquatella, Harry, Asch, Federico M., Barbosa, Marcia M., Barros, Marcio, Bern, Caryn, Cavalcante, Joao L., Echeverria Correa, Luis Eduardo, Lima, Joao, Marcus, Rachel, Marin-Neto, Jose Antonio, Migliore, Ricardo, Milei, Jose, Morillo, Carlos A., Nunes, Maria, Campos Vieira, Marcelo Luiz, and Viotti, Rodolfo

Published
2024
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26. Cardiovascular and other outcomes postintervention with insulin glargine and omega-3 fatty acids (ORIGINALE)

Author

Punthakee, Zubin, Gerstein, Hertzel C., Bosch Pagans, Jordi, Tyrwhitt, Jessica, Jung, Hyejung, Lee, Shun Fu, Lonn, Eva, Marsden, Tamara, McKelvie, Robert, McQueen, Matthew J., Morillo, Carlos, Yusuf, Shazzid, Dagenais, Gilles, Diaz, Rafael, Maggioni, Aldo P., Probstfield, Jeffrey, Ramachandran, Ambady, Riddle, Matthew C., Rydén, Lars, Badings, Erik, Birkeland, Kare, Cardona Munoz, Ernesto G., Commerford, Patrick, Davies, Melanie, Fodor, George J., Gomis, Ramon, Hanefeld, Markolf, Hildebrandt, Per, Kacerovsky Bielesz, Gertrud, Keltai, Matyas, Lanas, Fernando, Lewis, Basil S., Lopez-Jaramillo, Patricio, Marin Neto, Jose Antonio, Marre, Michel, Mendoza, Ivan, Pan, Chun yue, Pirags, Valdis, Rosenstock, Julio, Spinas, Giatgen A., Sreenan, Seamus, Syvänne, Mikko, Yale, Jean Francois, and ORIGIN Trial Investigators

Subjects
Omega-3, Fatty Acids, Insulin Glargine, Postintervention, Cardiovascular
Abstract

8 p., OBJECTIVE The Outcome Reduction With Initial Glargine Intervention (ORIGIN) trial reported neutral effects of insulin glargine on cardiovascular outcomes and cancers and reduced incident diabetes in high–cardiovascular risk adults with dysglycemia after 6.2 years of active treatment. Omega-3 fatty acids had neutral effects on cardiovascular outcomes. The ORIGIN and Legacy Effects (ORIGINALE) study measured posttrial effects of these interventions during an additional 2.7 years. RESEARCH DESIGN AND METHODS Surviving ORIGIN participants attended up to two additional visits. The hazard of clinical outcomes during the entire follow-up period from randomization was calculated. RESULTS Of 12,537 participants randomized, posttrial data were analyzed for 4,718 originally allocated to insulin glargine (2,351) versus standard care (2,367), and 4,771 originally allocated to omega-3 fatty acid supplements (2,368) versus placebo (2,403). Posttrial, small differences in median HbA1c persisted (glargine 6.6% [49 mmol/mol], standard care 6.7% [50 mmol/mol], P = 0.025). From randomization to the end of posttrial follow-up, no differences were found between the glargine and standard care groups in myocardial infarction, stroke, or cardiovascular death (1,185 vs. 1,165 events; hazard ratio 1.01 [95% CI 0.94–1.10]; P = 0.72); myocardial infarction, stroke, cardiovascular death, revascularization, or hospitalization for heart failure (1,958 vs. 1,910 events; 1.03 [0.97–1.10]; P = 0.38); or any cancer (524 vs. 529 events; 0.99 [0.88–1.12]; P = 0.91) or between omega-3 and placebo groups in cardiovascular death (688 vs. 700; 0.98 [0.88–1.09]; P = 0.68) or other outcomes. CONCLUSIONS During >6 years of treatment followed by >2.5 years of observation, insulin glargine had neutral effects on health outcomes and salutary effects on metabolic control, whereas omega-3 fatty acid supplementation had no effect.

Published
2016

28. Polymorphism in the Alpha Cardiac Muscle Actin 1 Gene Is Associated to Susceptibility to Chronic Inflammatory Cardiomyopathy

Author

Frade, Amanda Farage, Teixeira, Priscila Camilo, Ianni, Barbara Maria, Pissetti, Cristina Wide, Saba, Bruno, Tzu Wang, Lin Hui, Kuramoto, Andreia, Nogueira, Luciana Gabriel, Buck, Paula, Dias, Fabrício, Giniaux, Helene, Llored, Agnes, Alves, Sthefanny, Schmidt, Andre, Donadi, Eduardo, Marin-Neto, Jose Antonio, Hirata, Mario, Sampaio, Marcelo, Fragata, Abílio, Bocchi, Edimar Alcides, Stolf, Antonio Noedir, Fiorelli, Alfredo Inacio, Barros Santos, Ronaldo Honorato, Rodrigues, Virmondes, Pereira, Alexandre Costa, Kalil, Jorge, Cunha-Neto, Edecio, Chevillard, Christophe, Heart Institute (SAO PAULO - Heart Institute), University of São Paulo Medical School, Universidade Federal do Triângulo Mineiro Uberaba, Instituto de Cardiologia Dante Pazzanese (IDPC), Heart Institute (InCor), Universidade de São Paulo (USP)-Faculdade de Medicina FMUSP, School of Medicine of Ribeirão Preto (FMRP), University of São Paulo (USP), Aix Marseille Université (AMU), Department of Allergy and Clinical Immunology [São Paulo, Brazil], Hospital Sírio-Libanês [São Paulo, Brazil], Laboratory of Immunology, Universidade Federal do Triângulo Mineiro (UFTM), Institute for Investigation in Immunology (III), National Institute of Science and Technology (INCT), Division of Clinical Immunology and Allergy, University of São Paulo School of Medicine, Génétique et immunologie des maladies parasitaires (GIMP), Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)-Aix Marseille Université (AMU), Universidade de São Paulo = University of São Paulo (USP)-Faculdade de Medicina FMUSP, Universidade de São Paulo = University of São Paulo (USP), and Aix Marseille Université (AMU)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)

Subjects
Chagas Cardiomyopathy, Male, Chagas disease, Myocardium, lcsh:R, lcsh:Medicine, Heart, Apoptosis, Inflammatory diseases, Polymorphism, Single Nucleotide, Actins, [SDV.MP]Life Sciences [q-bio]/Microbiology and Parasitology, Gene Expression Regulation, Haplotypes, Humans, Female, Genetic Predisposition to Disease, lcsh:Q, RNA, Messenger, Variant genotypes, lcsh:Science, Cardiomyopathies, Research Article
Abstract

International audience; AimsChagas disease, caused by the protozoan Trypanosoma cruzi is endemic in Latin America, and may lead to a life-threatening inflammatory dilated, chronic Chagas cardiomyopathy (CCC). One third of T. cruzi-infected individuals progress to CCC while the others remain asymptomatic (ASY). A possible genetic component to disease progression was suggested by familial aggregation of cases and the association of markers of innate and adaptive immunity genes with CCC development. Since mutations in multiple sarcomeric genes, including alpha-cardiac actin (ACTC1) have been involved in hereditary dilated cardiomyopathy, we investigated the involvement of the ACTC1 gene in CCC pathogenesis.Methods and ResultsWe conducted a proteomic and genetic study on a Brazilian study population. The genetic study was done on a main cohort including 118 seropositive asymptomatic subjects and 315 cases and the replication was done on 36 asymptomatic and 102 CCC cases. ACTC1 protein and mRNA levels were lower in myocardial tissue from patients with end-stage CCC than those found in hearts from organ donors. Genotyping a case-control cohort of CCC and ASY subjects for all informative single nucleotide polymorphism (SNP) in the ACTC1 gene identified rs640249 SNP, located at the 5’ region, as associated to CCC. Associations are borderline after correction for multiple testing. Correlation and haplotype analysis led to the identification of a susceptibility haplotype. Functional assays have shown that the rs640249A/C polymorphism affects the binding of transcriptional factors in the promoter regions of the ACTC1 gene. Confirmation of the detected association on a larger independent replication cohort will be useful.ConclusionsGenetic variations at the ACTC1 gene may contribute to progression to chronic Chagas Cardiomyopathy among T. cruzi-infected patients, possibly by modulating transcription factor binding to ACTC1 promoter regions.

Published
2013

32. Towards the establishment of a consensus real-time qPCR to monitor Trypanosoma cruzi parasitemia in patients with chronic Chagas disease cardiomyopathy: A substudy from the BENEFIT trial

Author

Moreira, Otacilio C., primary, Ramírez, Juan David, additional, Velázquez, Elsa, additional, Melo, Myllena F. A. Dias, additional, Lima-Ferreira, Carolina, additional, Guhl, Felipe, additional, Sosa-Estani, Sergio, additional, Marin-Neto, Jose Antonio, additional, Morillo, Carlos A., additional, and Britto, Constança, additional

Published
2013
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33. I Diretriz Latino-Americana para o diagnóstico e tratamento da cardiopatia chagásica: resumo executivo

Author

Andrade, Jadelson Pinheiro de, primary, Marin Neto, Jose Antonio, additional, Paola, Angelo Amato Vincenzo de, additional, Vilas-Boas, Fábio, additional, Oliveira, Glaucia Maria Moraes, additional, Bacal, Fernando, additional, Bocchi, Edimar Alcides, additional, Almeida, Dirceu Rodrigues, additional, Fragata Filho, Abilio Augusto, additional, Moreira, Maria da Consolação Vieira, additional, Xavier, Sergio Salles, additional, Oliveira Junior, Wilson Alves de, additional, and Dias, João Carlos Pinto, additional

Published
2011
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34. Abstract 2810: The Occluded Artery Trial (OAT) Viability Ancillary Study (OAT-NUC): Influence of Infarct Zone Viability on Left Ventricular Remodeling After PCI vs. Medical Therapy Alone

Author

Udelson, James E, primary, Pearte, Camille A, additional, Kimmelstiel, Carey D, additional, Kruk, Mariusz, additional, Teresinska, Anna, additional, Bychowiec, Bartosz, additional, Marin-Neto, Jose Antonio, additional, Höchtl, Thomas, additional, Cohen, Eric A, additional, Caramori, Paulo, additional, Busz-Papiez, Benita, additional, Adlbrecht, Christopher, additional, Sadowski, Zygmunt P, additional, Ruzyllo, Witold, additional, Forman, Sandy A, additional, Kinan, Deborah J, additional, Lamas, Gervasio A, additional, and Hochman, Judith S, additional

Published
2007
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39. Acute myocardial infarction complicating the clinical course of dilated cardiomyopathy in childhood

Author

Simoes, Marcus Vinicius, Felix, Paulo Roberto, and Marin-Neto, Jose Antonio

Subjects
Cardiomyopathy, Dilated -- Complications and side effects, Heart attack -- Causes of -- Complications and side effects, Pediatric cardiology -- Causes of, Cardiac arrest in children -- Causes of -- Complications and side effects, Cardiac arrest -- Causes of -- Complications and side effects, Health, Complications and side effects, Causes of
Abstract

An acute myocardial infraction occurred in a 6-year-old child with dilated cardiomyopathy. This caused severe hemodynamic deterioration that led to a fatal outcome. Autopsy revealed diffuse myocardial atrophy without cell infiltrate, normal epicardial coronary arteries, and a massive healed anteroapical infraction. Coronary embolism or spasm could be ruled out as the cause of the infraction., Acute myocardial infarction is extremely rare in children.[1,2] Even though acute myocardial infarction may be detected in association with dilated cardiomyopathy in adults, this association has never been previously proved [...]

Published
1992

43. I Latin American Guidelines for the diagnosis and treatment of Chagas' heart disease: executive summary.

Author

Andrade JP, Marin Neto JA, Paola AA, Vilas-Boas F, Oliveira GM, Bacal F, Bocchi EA, Almeida DR, Fragata Filho AA, Moreira Mda C, Xavier SS, Oliveira Junior WA, and Dias JC

Subjects
Humans, Chagas Cardiomyopathy diagnosis, Chagas Cardiomyopathy therapy
Abstract

Much has been achieved in one century after Carlos Chagas' discovery. However, there is surely much to be done in the next decades. At present, we are witnessing many remarkable efforts to monitor the epidemiology of the disease, to better understand the biology of the T. cruzi and its interaction with human beings as well as the pathogenesis and pathophysiology of the complications in the chronic phase, and deal more appropriately and effectively with late cardiac and digestive manifestations. Although the vector and transfusion-derived transmission of the disease has been controlled in many countries, there remains a pressing need for sustained surveillance of the measures that led to this achievement. It is also necessary to adopt initiatives that enable appropriate management of social and medical conditions resulting from the migration of infected individuals to countries where the disease formerly did not exist. It's also necessary to standardize the most reliable methods of detection of infection with T. cruzi, not only for diagnosis purposes, but more crucially, as a cure criterion. The etiological treatment of millions of patients in the chronic stage of the disease is also to be unraveled. A renewed interest in this area is observed, including prospects of studies focusing on the association of drugs with benznidazole. We also wait for full evidence of the actual effectiveness of the etiological treatment to impact favorably on the natural history of the disease in its chronic phase. Eventually, cardiologists are primarily responsible for improving the clinical management of their patients with Chagas' disease, judiciously prescribing drugs and interventions that respect, as much as possible, the peculiar pathophysiology of the disease, wasting no plausible therapeutic opportunities.

Published
2011
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