36 results on '"Marrapodi, Ramona"'
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2. Breakthrough infections after COVID-19 vaccinations do not elicit platelet hyperactivation and are associated with high platelet–lymphocyte and low platelet–neutrophil aggregates
3. Distinct platelet crosstalk with adaptive and innate immune cells after adenoviral and mRNA vaccination against SARS-CoV-2
4. Platelet and immune signature associated with a rapid response to the BNT162b2 mRNA COVID‐19 vaccine
5. CD21low B cells in systemic sclerosis: A possible marker of vascular complications
6. The Keratinocyte in the Picture Cutaneous Melanoma Microenvironment.
7. Dual stimulation by autoantigen and CpG fosters the proliferation of exhausted rheumatoid factor-specific CD21low B cells in hepatitis C virus-cured mixed cryoglobulinemia.
8. Platelets from patients with decompensated cirrhosis display pro-inflammatory features
9. Correction to: Reduced Lysosomal Acid Lipase Activity in Blood and Platelets Is Associated With Nonalcoholic Fatty Liver Disease
10. Reduced Lysosomal Acid Lipase Activity in Blood and Platelets Is Associated With Nonalcoholic Fatty Liver Disease
11. Opposite Effects of mRNA-Based and Adenovirus-Vectored SARS-CoV-2 Vaccines on Regulatory T Cells: A Pilot Study
12. Dual stimulation by autoantigen and CpG fosters the proliferation of exhausted rheumatoid factor-specific CD21low B cells in hepatitis C virus-cured mixed cryoglobulinemia
13. Increased von Willebrand Factor Platelet-Binding Capacity Is Related to Poor Prognosis in COVID-19 Patients
14. Dysregulated extracellular signal-regulated kinase signaling associated with impaired B-cell receptor endocytosis in patients with common variable immunodeficiency
15. Increased von Willebrand Factor Platelet-Binding Capacity Is Related to Poor Prognosis in COVID-19 Patients.
16. Intravenous immunoglobulin replacement therapy in common variable immunodeficiency induces B cell depletion through differentiation into apoptosis-prone CD21low B cells
17. CD21low B cells are predictive markers of new digital ulcers in systemic sclerosis
18. THU-350 - Platelets from patients with decompensated cirrhosis display pro-inflammatory features
19. Clinico‐immunological outcomes of HCV‐cured cryoglobulinemia: Lower relapse rate with interferon‐based than interferon‐free therapy
20. Biomarkers of minimal residual disease in rituximab‐treated patients with mixed cryoglobulinemia
21. CD21low B cells are predictive markers of new digital ulcers in systemic sclerosis.
22. Biomarkers of minimal residual disease in rituximab-treated patients with mixed cryoglobulinemia.
23. Flow cytometry in formamide treated cells
24. Reversion of anergy signatures in clonal CD21low B cells of mixed cryoglobulinemia after clearance of HCV viremia
25. Clinico‐immunological outcomes of HCV‐cured cryoglobulinemia: Lower relapse rate with interferon‐based than interferon‐free therapy.
26. DEC1/STRA13 is a key negative regulator of activation-induced proliferation of human B cells highly expressed in anergic cells
27. Immunomodulatory effects of intravenous immunoglobulin - assembling a jigsaw puzzle
28. Intravenous Immunoglobulin and Immunomodulation of B-Cell – in vitro and in vivo Effects
29. Clonal expansion and functional exhaustion of monoclonal marginal zone B cells in mixed cryoglobulinemia: The yin and yang of HCV-driven lymphoproliferation and autoimmunity
30. Reversion of anergy signatures in clonal CD21lowB cells of mixed cryoglobulinemia after clearance of HCV viremia
31. Intravenous immunoglobulin and immunomodulation of B-cell - in vitro and in vivo effects.
32. Reversion of anergy signatures in clonal CD21low B cells of mixed cryoglobulinemia after clearance of HCV viremia.
33. Dual stimulation by autoantigen and CpG fosters the proliferation of exhausted rheumatoid factor-specific CD21 low B cells in hepatitis C virus-cured mixed cryoglobulinemia.
34. CD21 low B cells are predictive markers of new digital ulcers in systemic sclerosis.
35. Rheumatoid factor-producing CD21low anergic clonal B-cells in essential mixed cryoglobulinaemia: a model for autoantigen-driven pathogenesis of infectious and non-infectious cryoglobulinaemias.
36. Hepatitis B virus causes mixed cryoglobulinaemia by driving clonal expansion of innate B-cells producing a VH1-69-encoded antibody.
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