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1. Metallothionein-3 is a multifunctional driver that modulates the development of sorafenib-resistant phenotype in hepatocellular carcinoma cells

2. p63 controls metabolic activation of hepatic stellate cells and fibrosis via an HER2-ACC1 pathway

3. Anti-miR-873-5p improves alcohol-related liver disease by enhancing hepatic deacetylation via SIRT1

4. Neddylation of phosphoenolpyruvate carboxykinase 1 controls glucose metabolism

5. Hepatocyte-specific O-GlcNAc transferase downregulation ameliorates nonalcoholic steatohepatitis by improving mitochondrial function

6. PI3K-regulated Glycine N-methyltransferase is required for the development of prostate cancer

7. Designing a broad-spectrum integrative approach for cancer prevention and treatment

8. miR-873-5p targets mitochondrial GNMT-Complex II interface contributing to non-alcoholic fatty liver disease

9. Involvement of G protein-coupled receptor kinase 2 (GRK2) in the development of non-alcoholic steatosis and steatohepatitis in mice and humans

13. Targeting NAE1-mediated protein hyper-NEDDylation halts cholangiocarcinogenesis and impacts on tumor-stroma crosstalk in experimental models

14. Inhibition of carnitine palmitoyltransferase 1A in hepatic stellate cells protects against fibrosis

15. Global multi-stakeholder endorsement of the MAFLD definition

16. Global multi-stakeholder endorsement of the MAFLD definition

17. Hepatoma Cells From Mice Deficient in Glycine N-Methyltransferase Have Increased RAS Signaling and Activation of Liver Kinase B1

18. Inhibition of ATG3 ameliorates liver steatosis by increasing mitochondrial function

21. Dual targeting of G9a and DNMT1 for the treatment of experimental cholangiocarcinoma

22. Inhibition of NAE‐dependent protein hyper‐NEDDylation in cystic cholangiocytes halts cystogenesis in experimental models of polycystic liver disease

23. MO38-2 Metallothionein-3: Potential therapeutic target for sorafenib resistance in hepatocellular carcinoma

24. Corrigendum: mTORC1-dependent AMD1 regulation sustains polyamine metabolism in prostate cancer

25. Dual Targeting of G9a and DNA Methyltransferase‐1 for the Treatment of Experimental Cholangiocarcinoma

26. Obese patients with NASH have increased hepatic expression of SARS-CoV-2 critical entry points

27. Targeting UBC9-mediated protein hyper-SUMOylation in cystic cholangiocytes halts polycystic liver disease in experimental models

28. mTORC1-dependent AMD1 regulation sustains polyamine metabolism in prostate cancer

29. Dual Pharmacological Targeting of HDACs and PDE5 Inhibits Liver Disease Progression in a Mouse Model of Biliary Inflammation and Fibrosis

31. The L‐α‐Lysophosphatidylinositol/G Protein–Coupled Receptor 55 System Induces the Development of Nonalcoholic Steatosis and Steatohepatitis

32. 178 - THERAPEUTIC POTENTIAL OF TARGETING PROTEIN HYPER- SUMOYLATION IN CHOLANGIOCARCINOMA

33. Solute Carrier Family 2 Member 1 Is Involved in the Development of Nonalcoholic Fatty Liver Disease

36. Genomic and Functional Regulation of TRIB1 Contributes to Prostate Cancer Pathogenesis

37. A Novel Serum Metabolomic Profile for the Differential Diagnosis of Distal Cholangiocarcinoma and Pancreatic Ductal Adenocarcinoma

38. Causes of hOCT1‐Dependent Cholangiocarcinoma Resistance to Sorafenib and Sensitization by Tumor‐Selective Gene Therapy

39. Epigenetic events involved in organic cation transporter 1-dependent impaired response of hepatocellular carcinoma to sorafenib

41. Erratum: Corrigendum: mTORC1-dependent AMD1 regulation sustains polyamine metabolism in prostate cancer

42. is involved in the development of nonalcoholic fatty liver disease

43. Sustained proliferation in cancer: Mechanisms and novel therapeutic targets

45. INHIBITION OF NK CELLS PROTECTS THE LIVER AGAINST ACUTE INJURY IN THE ABSENCE OF GNMT

46. The Promyelocytic Leukemia Protein Is Upregulated in Conditions of Obesity and Liver Steatosis

48. Solute carrier family 2 member 1is involved in the development of nonalcoholic fatty liver disease

50. S-adenosylmethionine and proliferation: new pathways, new targets

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