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1. Machine learning of dissection photographs and surface scanning for quantitative 3D neuropathology

2. Genome-wide association meta-analysis of neuropathologic features of Alzheimer's disease and related dementias.

3. Modeling intracerebral hemorrhage growth and response to anticoagulation.

4. Cyclic multiplex fluorescent immunohistochemistry and machine learning reveal distinct states of astrocytes and microglia in normal aging and Alzheimer’s disease

5. Melanocortin 1 receptor activation protects against alpha-synuclein pathologies in models of Parkinson’s disease

6. Exceptionally low likelihood of Alzheimer’s dementia in APOE2 homozygotes from a 5,000-person neuropathological study

7. Enhanced Virus Detection and Metagenomic Sequencing in Patients with Meningitis and Encephalitis

8. Impairment of visual cortical plasticity by amyloid-beta species

9. Distinct cytokine profiles in human brains resilient to Alzheimer's pathology

11. Isoform-selective decrease of glycogen synthase kinase-3-beta (GSK-3β) reduces synaptic tau phosphorylation, transcellular spreading, and aggregation

13. T2 Hypointense Schwannoma Masquerading As A Vascular Lesion: A Case Report

14. Repetitive head impacts and chronic traumatic encephalopathy are associated with TDP-43 inclusions and hippocampal sclerosis

15. An in vitro paradigm to assess potential anti-Aβ antibodies for Alzheimer’s disease

16. Molecular and clinicopathologic characteristics of gliomas with EP300::BCOR fusions

18. Differential Vulnerability of Hippocampal Subfields in Primary Age-Related Tauopathy and Chronic Traumatic Encephalopathy

19. Creating the Pick’s disease International Consortium: Association study of MAPT H2 haplotype with risk of Pick’s disease

20. A peptide-centric quantitative proteomics dataset for the phenotypic assessment of Alzheimer’s disease

21. Stereology neuron counts correlate with deep learning estimates in the human hippocampal subregions

23. Somatic genomic changes in single Alzheimer’s disease neurons

24. New insights into the genetic etiology of Alzheimer's disease and related dementias

26. Perivascular space dilation is associated with vascular amyloid-β accumulation in the overlying cortex

27. Supplementary Figure 1 from Glioblastoma Recurrence after Cediranib Therapy in Patients: Lack of 'Rebound' Revascularization as Mode of Escape

28. Supplementary Figure 2 from Glioblastoma Recurrence after Cediranib Therapy in Patients: Lack of 'Rebound' Revascularization as Mode of Escape

29. Supplementary Table 1 from Glioblastoma Recurrence after Cediranib Therapy in Patients: Lack of 'Rebound' Revascularization as Mode of Escape

30. Plasma biomarkers for diagnosis of Alzheimers disease and prediction of cognitive decline in individuals with mild cognitive impairment

32. Pentad: A reproducible cytoarchitectonic protocol and its application to parcellation of the human hippocampus

33. Histopathology of Cerebral Microinfarcts and Microbleeds in Spontaneous Intracerebral Hemorrhage

35. Genome-wide association study and functional validation implicates JADE1 in tauopathy

36. Altered localization of nucleoporin 98 in primary tauopathies

37. Elucidation of Amyloid-Beta’s Gambit in Oligomerization: Truncated Aβ fragments of residues Aβ1-23, Aβ1-24 and Aβ1-25 rapidly seed to form SDS-stable, LMW Aβ oligomers that impair synaptic plasticity

38. Correction to: Clinical trial-ready patient cohorts for multiple system atrophy: coupling biospecimen and iPSC banking to longitudinal deep-phenotyping

39. Single‐Neuron Genome Sequencing in Alzheimer’s Disease Identifies Disease‐Specific Somatic Alterations and Insights into Pathogenesis

40. Clinical Trial-Ready Patient Cohorts for Multiple System Atrophy: Coupling Biospecimen and iPSC Banking to Longitudinal Deep-Phenotyping

42. Impact of Sterilization Methods on the Seeding Ability of Human Tau Proteopathic Seeds

43. Continuous Monitoring of Tau-Induced Neurotoxicity in Patient-Derived iPSC-Neurons

45. Hippocampal Pyramidal Neuron Size Differs Among Subfields: Implications for Subfield Parcellation

46. Changes in glial cell phenotypes precede overt neurofibrillary tangle formation, correlate with markers of cortical cell damage, and predict cognitive status of individuals at Braak III-IV stages

47. Entorhinal Subfield Vulnerability to Neurofibrillary Tangles in Aging and the Preclinical Stage of Alzheimer's Disease

48. Predicting the location of entorhinal cortex from MRI.

49. A Simplified Brain Blocking Protocol Optimized for the Diagnosis of Neurodegenerative Disease Saves Time and Money While Preserving Anatomic Relationships

50. Risk of Transmissibility From Neurodegenerative Disease-Associated Proteins: Experimental Knowns and Unknowns

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