338 results on '"McCormick, John K."'
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2. Discovery of an antivirulence compound that targets the Staphylococcus aureus SaeRS two-component system to inhibit toxic shock syndrome toxin-1 production
3. Interplay between Staphylococcus aureus and the vaginal microbiota
4. Vaginal community state types (CSTs) alter environmental cues and production of the Staphylococcus aureus toxic shock syndrome toxin-1 (TSST-1)
5. Streptolysin S is required for Streptococcus pyogenes nasopharyngeal and skin infection in HLA-transgenic mice
6. Discovery of an anti-virulence compound that targets the Staphylococcus aureus SaeRS two-component system to inhibit toxic shock syndrome toxin 1 (TSST-1) production
7. The SrrAB two-component system regulates Staphylococcus aureus pathogenicity through redox sensitive cysteines
8. MAIT Cells Are Major Contributors to the Cytokine Response in Group A Streptococcal Toxic Shock Syndrome
9. Peptide Blockers of PD-1-PD-L1 Interaction Reinvigorate PD-1-Suppressed T Cells and Curb Tumor Growth in Mice.
10. Validation of an Algorithm to Identify Infective Endocarditis in People Who Inject Drugs
11. Toxins and Superantigens of Group A Streptococci
12. Nasopharyngeal infection by Streptococcus pyogenes requires superantigen-responsive Vβ-specific T cells
13. Invariant Natural Killer T Cells Are Pathogenic in the HLA-DR4—Transgenic Humanized Mouse Model of Toxic Shock Syndrome and Can Be Targeted to Reduce Morbidity
14. Genome Sequence of a Serotype M3 Strain of Group A Streptococcus: Phage-Encoded Toxins, the High-Virulence Phenotype, and Clone Emergence
15. Prophage exotoxins enhance colonization fitness in epidemic scarlet fever-causing Streptococcus pyogenes
16. HLA class I-associated expansion of TRBV11-2 T cells in multisystem inflammatory syndrome in children
17. Swift Intrahepatic Accumulation of Granulocytic Myeloid-Derived Suppressor Cells in a Humanized Mouse Model of Toxic Shock Syndrome
18. The fadXDEBA locus of Staphylococcus aureus is required for metabolism of exogenous palmitic acid and in vivo growth.
19. The Streptococcus pyogenes hyaluronic acid capsule promotes experimental nasal and skin infection by preventing neutrophil-mediated clearance
20. Staphylococcus lugdunensis Uses the Agr Regulatory System to Resist Killing by Host Innate Immune Effectors
21. Glucose Mediates Niche-Specific Repression of Staphylococcus aureus Toxic Shock Syndrome Toxin-1 through the Activity of CcpA in the Vaginal Environment
22. The Streptococcus pyogenes hyaluronic acid capsule promotes experimental nasal and skin infection by preventing neutrophil-mediated clearance
23. Superantigens promote Staphylococcus aureus bloodstream infection by eliciting pathogenic interferon-gamma production
24. Lactobacillus reuteri-produced cyclic dipeptides quench agr-mediated expression of toxic shock syndrome toxin-1 in staphylococci
25. Nasopharyngeal Infection of Mice with Streptococcus pyogenes and In Vivo Detection of Superantigen Activity
26. Neutralization of Multiple Staphylococcal Superantigens by a Single-Chain Protein Consisting of Affinity-Matured, Variable Domain Repeats
27. Streptococcal superantigens and the return of scarlet fever
28. Long-Range Cooperative Binding Effects in a T Cell Receptor Variable Domain
29. Effects of Total Body Irradiation and Cyclosporin A on the Lethality of Toxic Shock Syndrome Toxin-1 in a Rabbit Model of Toxic Shock Syndrome
30. Formation of Vegetations during Infective Endocarditis Excludes Binding of Bacterial-Specific Host Antibodies to Enterococcus faecalis
31. Superantigens promote Staphylococcus aureus bloodstream infection by eliciting pathogenic interferon-gamma (IFNγ) production that subverts macrophage function
32. Toxins and Superantigens of Group A Streptococci
33. Toll-like receptor 2 ligands on the staphylococcal cell wall downregulate superantigen-induced T cell activation and prevent toxic shock syndrome
34. Population Analysis of Staphylococcus aureus Reveals a Cryptic, Highly Prevalent Superantigen SElW That Contributes to the Pathogenesis of Bacteremia
35. The SrrAB two-component system regulates Staphylococcus aureus pathogenicity through redox sensitive cysteines
36. Inhibition of expression of a staphylococcal superantigen-like protein by a soluble factor from Lactobacillus reuteri
37. Pathogenic mechanisms of enterococcal endocarditis
38. Repression of the staphylococcus aureus accessory gene regulator in serum and in vivo
39. CD1d-independent activation of mouse and human iNKT cells by bacterial superantigens
40. Identification of a novel two-component regulatory system that acts in global regulation of virulence factors of Staphylococcus aureus
41. Toxic Shock Syndrome and Bacterial Superantigens: An Update
42. Population Analysis of Staphylococcus aureus Reveals a Cryptic, Highly Prevalent Superantigen SElW That Contributes to the Pathogenesis of Bacteremia
43. Discordant rearrangement of primary and anamnestic CD8+ T cell responses to influenza A viral epitopes upon exposure to bacterial superantigens: Implications for prophylactic vaccination, heterosubtypic immunity and superinfections
44. Prophage exotoxins enhance colonization fitness in epidemic scarlet fever-causing Streptococcus pyogenes
45. T Cell Signalling Induced by Bacterial Superantigens
46. Bacterial Superantigens Bypass Lck-Dependent T Cell Receptor Signaling by Activating a Gα11-Dependent, PLC-β-Mediated Pathway
47. Expression of the antimicrobial peptide carnobacteriocin B2 by a signal peptide-dependent general secretory pathway
48. An amino-terminal domain of Enterococcus faecalis aggregation substance is required for aggregation, bacterial internalization by epithelial cells and binding to lipoteichoic acid
49. Correction: A controlled-release oral opioid supports S. aureus survival in injection drug preparation equipment and may increase bacteremia and endocarditis risk
50. A controlled-release oral opioid supports S. aureus survival in injection drug preparation equipment and may increase bacteremia and endocarditis risk
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