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1. Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2–SP1–SAT1 axis

3. Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2–SP1–SAT1 axis

4. SIRT1–NOX4 signaling axis regulates cancer cachexia

9. The MUC1–HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling

10. Supplementary Fig 2 from Vitamin B6 Competition in the Tumor Microenvironment Hampers Antitumor Functions of NK Cells

11. Supplementary Fig 4 from Vitamin B6 Competition in the Tumor Microenvironment Hampers Antitumor Functions of NK Cells

12. Supplementary Fig 7 from Vitamin B6 Competition in the Tumor Microenvironment Hampers Antitumor Functions of NK Cells

13. Supplementary Fig 1 from Vitamin B6 Competition in the Tumor Microenvironment Hampers Antitumor Functions of NK Cells

14. Supplementary Fig 3 from Vitamin B6 Competition in the Tumor Microenvironment Hampers Antitumor Functions of NK Cells

15. Supplementary Fig 5 from Vitamin B6 Competition in the Tumor Microenvironment Hampers Antitumor Functions of NK Cells

16. Supplementary Fig 6 from Vitamin B6 Competition in the Tumor Microenvironment Hampers Antitumor Functions of NK Cells

18. Vitamin B6 Competition in the Tumor Microenvironment Hampers Antitumor Functions of NK Cells

23. DHODH inhibition enhances the efficacy of immune checkpoint blockade by increasing cancer cell antigen presentation

24. DHODH inhibition enhances the efficacy of immune checkpoint blockade by increasing cancer cell antigen presentation

25. Supplementary Fig S2 from IgE-Based Therapeutic Combination Enhances Antitumor Response in Preclinical Models of Pancreatic Cancer

32. IgE-Based Therapeutic Combination Enhances Antitumor Response in Preclinical Models of Pancreatic Cancer

34. Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression

45. A Polymeric Nanogel-Based Treatment Regimen for Enhanced Efficacy and Sequential Administration of Synergistic Drug Combination in Pancreatic Cancer

47. Combination of mAb-AR20.5, anti-PD-L1 and PolyICLC inhibits tumor progression and prolongs survival of MUC1.Tg mice challenged with pancreatic tumors

48. MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancreatic Cancer

50. Abstract 10: Silibinin exhibits anti-cachectic and anti-cancerous property by modulating metabolic properties of pancreatic cancer cells

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