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1. Profiling of insulin-resistant kidney models and human biopsies reveals common and cell-type-specific mechanisms underpinning Diabetic Kidney Disease

2. An atlas of healthy and injured cell states and niches in the human kidney.

4. A reference tissue atlas for the human kidney

6. Precision Medicine in Nephrology: An Integrative Framework of Multidimensional Data in the Kidney Precision Medicine Project

8. An integrated organoid omics map extends modeling potential of kidney disease

9. Molecular Signatures of Glomerular Neovascularization in a Patient with Diabetic Kidney Disease

10. Participant Experience with Protocol Research Kidney Biopsies in the Kidney Precision Medicine Project

13. A Participant-Centered Approach to Understanding Risks and Benefits of Participation in Research Informed by the Kidney Precision Medicine Project

14. Endogenous adenine mediates kidney injury in diabetic models and predicts diabetic kidney disease in patients

16. Cadherin-11, Sparc-related modular calcium binding protein-2, and Pigment epithelium-derived factor are promising non-invasive biomarkers of kidney fibrosis

17. SGLT2 inhibitors mitigate kidney tubular metabolic and mTORC1 perturbations in youth-onset type 2 diabetes

18. SARS-CoV-2 receptor networks in diabetic and COVID-19–associated kidney disease

22. Protein N-Glycans in Healthy and Sclerotic Glomeruli in Diabetic Kidney Disease

23. Identification of Kidney Cell Types in scRNA-seq and snRNA-seq Data Using Machine Learning Algorithms

24. Pax protein depletion in proximal tubules triggers conserved mechanisms of resistance to acute ischemic kidney injury preventing transition to chronic kidney disease

25. An eQTL Landscape of Kidney Tissue in Human Nephrotic Syndrome

26. High-Throughput Screening Enhances Kidney Organoid Differentiation from Human Pluripotent Stem Cells and Enables Automated Multidimensional Phenotyping

27. Pax Protein Depletion in Proximal Tubules Triggers Conserved Mechanisms of Resistance to Acute Ischemic Kidney Injury and Prevents Transition to Chronic Kidney Disease

28. Regulation of heterotopic ossification by monocytes in a mouse model of aberrant wound healing

29. Genome-wide Association Study for Acute Kidney Injury

30. Defining the molecular correlate of arteriolar hyalinosis in kidney disease progression by integration of single cell transcriptomic analysis and pathology scoring

31. Role of endogenous adenine in kidney failure and mortality with diabetes

32. Pro-cachectic factors link experimental and human chronic kidney disease to skeletal muscle wasting programs

33. Endoplasmic reticulum-associated degradation is required for nephrin maturation and kidney glomerular filtration function

34. Supplemental Table S1. Wnt target genes from Activation of Wnt/β-Catenin in Ewing Sarcoma Cells Antagonizes EWS/ETS Function and Promotes Phenotypic Transition to More Metastatic Cell States

35. Supplemental Table S2. Primers from Activation of Wnt/β-Catenin in Ewing Sarcoma Cells Antagonizes EWS/ETS Function and Promotes Phenotypic Transition to More Metastatic Cell States

36. Supplementary Materials and Methods from Activation of Wnt/β-Catenin in Ewing Sarcoma Cells Antagonizes EWS/ETS Function and Promotes Phenotypic Transition to More Metastatic Cell States

37. Supplementary Figures from Activation of Wnt/β-Catenin in Ewing Sarcoma Cells Antagonizes EWS/ETS Function and Promotes Phenotypic Transition to More Metastatic Cell States

38. Data from Activation of Wnt/β-Catenin in Ewing Sarcoma Cells Antagonizes EWS/ETS Function and Promotes Phenotypic Transition to More Metastatic Cell States

39. Supplemental Figure Legends from Activation of Wnt/β-Catenin in Ewing Sarcoma Cells Antagonizes EWS/ETS Function and Promotes Phenotypic Transition to More Metastatic Cell States

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