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1. SETD2 and histone H3 lysine 36 methylation deficiency in advanced systemic mastocytosis

6. Conut score as a tool for early detection of poor nutritional status in advances forms of SM

9. Achieving a Major Molecular Response at the Time of a Complete Cytogenetic Response (CCgR) Predicts a Better Duration of CCgR in Imatinib-Treated Chronic Myeloid Leukemia Patients

10. Ultra-deep sequencing (uds) allows more sensitive detection of the D816V and other kit gene mutations in systemic mastocytosis

12. Age at disease onset has a major impact on clinical characteristics and course of mastocytosis

13. Impact of allergic disorders and anaphylaxis in mastocytosis: Preliminary data from a study of the European Competence Network on Mastocytosis (ECNM) registry

14. The body mass index is an indicator of advanced systemic mastocytosis: preliminary results from a registry project of the European Competence Network on Mastocytosis (ECNM) registry

15. Long term outcome of ph+ cml patients achieving complete cytogenetic remission with interferon based therapy moving from interferon to imatinib era

16. SETD2 and histone H3 lysine 36 methylation deficiency in advanced systemic mastocytosis

17. THE ITALIAN MASTOCYTOSIS REGISTRY: THE FIRST FIVE YEARS

18. Bcr-Abl kinase domain mutations and resistance in Ph+ acute lymphoblastic leukemia from the imatinib to the 2nd-generation TKI era

19. FOLLOW UP A LUNGO TERMINE DEI PAZIENTI CON LMC-PH+ IN RISPOSTA CITOGENETICA COMPLETA CON TERAPIA INCLUDENTE INTERFERONE NELL’ERA IMATINIB - PROTOCOLLO GIMEMA CML0509

20. BCR-ABL KINASE DOMAIN MUTATIONS IN IMATINIB AND IN SECOND- GENERATION TYROSINE KINASE INHIBITOR ERAS: A REVIEW OF SEVEN YEARS OF MUTATION ANALYSIS BY THE GIMEMA CML WORKING PARTY

24. Prediction response to imatinib by prospective quantitation of BCR-ABL transcript in late chronic phase chronic myeloid leukemia patients. Study by the GIMEMA working party on chronic myeloid leukemia

30. Idiopathic hypereosinphilic synndrome (HES) with FIP1L1-PDGFR-alpha rearrangement can be effectively treated with imatinib

32. European multicenter experience on idiopathic hypereosynophilic syndrome with FIP1L1-PDGFR-alpha rearrangement treated with imatinib

33. Italian Cooperative study on idiopathic hypereosinophilic syndrome (HES): FIP1-L1/PDGFRA rearrangement can be effectively treated with Imatinib

40. Evolving approaches with alpha interferon in chronic myelogenous leukemia

41. Risk assessment in myelodysplastic syndromes: Value of clinical, hematologic and bone marrow histologic findings at presentation

42. The long-term durability of cytogenetic responses in patients with accelerated phase chronic myeloid leukemia treated with imatinib 600 mg: the GIMEMA CML Working Party experience after a 7-year follow-up

43. Response to imatinib mesylate in patients with the FIP1L1-PDGFR-alpha positive hypereosinophilic syndrome: Update of a multicenter prospective study and guidelines for eosinophilia diagnosis

44. The efficacy of imatinib mesylate in patients with FIP1L1-PDGFR -positive hypereosinophilic syndrome. Results of a multicenter prospective study

49. Treatment of terminal‐phase chronic myelogenous leukemia with intermediate‐dose cytarabine and hydroxyurea

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