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Your search keyword '"Merliss, A. D."' showing total 10 results
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10 results on '"Merliss, A. D."'

2. Extraction of chronically implanted coronary sinus leads active fixation vs passive fixation leads

Author

Crossley, George H., primary, Sorrentino, Robert A., additional, Exner, Derek V., additional, Merliss, Andrew D., additional, Tobias, Serge M., additional, Martin, David O., additional, Augostini, Ralph, additional, Piccini, Jonathan P., additional, Schaerf, Raymond, additional, Li, Shelby, additional, Miller, Clayton T., additional, and Adler, Stuart W., additional

Published
2016
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6. Identification of a Novel Genetic Locus for Familial Cardiac Myxomas and Carney Complex

Author

Casey, Mairead, primary, Mah, Caroline, additional, Merliss, Andrew D., additional, Kirschner, Lawrence S., additional, Taymans, Susan E., additional, Denio, Alfred E., additional, Korf, Bruce, additional, Irvine, Alan D., additional, Hughes, Anne, additional, Carney, J. Aidan, additional, Stratakis, Constantine A., additional, and Basson, Craig T., additional

Published
1998
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8. Ventricular Arrhythmia in the X-linked Cardiomyopathy Barth Syndrome.

Author

Spencer, C. T., Byrne, B. J., Gewitz, M. H., Wechsler, S. B., Kao, A. C., Gerstenfeld, E. P., Merliss, A. D., Carboni, M. P., and Bryant, R. M.

Subjects
X chromosome abnormalities, SEX chromosome abnormalities, CARDIOMYOPATHIES, ARRHYTHMIA, HEART diseases, HEART beat, PEDIATRIC cardiology, CARDIOLOGY
Abstract

Barth syndrome is an X-linked disorder characterized by dilated cardiomyopathy, cyclic neutropenia, skeletal myopathy, abnormal mitochondria, and growth deficiency. The primary defect is a mutation in the TAZ gene on the X chromosome at Xq28, resulting in abnormal phospholipid biosynthesis and cardiolipin deficiency. To date, there has been no systematic evaluation of the cardiac phenotype. We report five cases of cardiac arrest and/or placement of an internal cardiac defibrillator with documented ventricular arrhythmia. We suggest that ventricular arrhythmia is part of the primary phenotype of the disorder and that patients should be screened accordingly. [ABSTRACT FROM AUTHOR]

Published
2005
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10. Pleuropericardial Reaction to Treatment With Dantrolene

Author

Petusevsky, Mitchell L., Faling, L. Jack, Rocklin, Ross E., Snider, Gordon L., Merliss, Andrew D., Moses, John M., and Dorman, Sanford A.

Abstract

Chronic pleural effusion occurred in three patients, one of whom also developed acute pericarditis. A fourth patient developed both pleural and pericardial effusions. All patients had been receiving dantrolene sodium for at least two months. The pleural fluid was a sterile exudate with pleural and peripheral blood eosinophilia in all patients. No pulmonary parenchymal involvement was apparent. Pleural biopsy specimens showed nonspecific inflammation. Resolution of the pleural process was prolonged after dantrolene therapy was discontinued. Although a causal relationship between dantrolene and serosal inflammation remains unproved, this association in four patients warrants careful observation of others receiving long-term dantrolene therapy.(JAMA 242:2772-2774, 1979)

Published
1979
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