165 results on '"Meurs, Eliane F."'
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2. A potential robust antiviral defense state in the common vampire bat: Expression, induction and molecular characterization of the three interferon-stimulated genes -OAS1, ADAR1 and PKR
3. Hepatitis C virus triggers Golgi fragmentation and autophagy through the immunity-related GTPase M
4. HIV-1 translation and its regulation by cellular factors PKR and PACT
5. Genetic variations in toll‐like receptors 7 and 8 modulate natural hepatitis C outcomes and liver disease progression
6. Differential regulation of the Wnt/β-catenin pathway by hepatitis C virus recombinants expressing core from various genotypes
7. Increased levels of cerebrospinal fluid JNK3 associated with amyloid pathology: links to cognitive decline
8. Tumor Suppressor Function of the Interferon-Induced Double-Stranded RNA-Activated Protein Kinase
9. The Inflammasome Components NLRP3 and ASC Act in Concert with IRGM To Rearrange the Golgi Apparatus during Hepatitis C Virus Infection
10. Retinoic Acid Inducible Gene I and Protein Kinase R, but Not Stress Granules, Mediate the Proinflammatory Response to Yellow Fever Virus
11. Pivotal role of the IKK-related kinase IKK epsilon to modulate IL29 or type I IFN induction during hepatitis C virus infection: 57
12. The PKR Activator PACT Is Induced by Aβ: Involvement in Alzheimerʼs Disease
13. Hepatitis C Virus Reveals a Novel Early Control in Acute Immune Response: CS09-6
14. The inflammasome components NLRP3 and ASC act in concert with IRGM to rearrange the Golgi during viral infections
15. RIG-I like receptors: sensing and responding to RNA virus infection: CS2-1
16. Polo-like kinase 1 (PLK1) regulates IFN induction by MAVS
17. Regulation of innate immunity and inflammation by the mitotic kinase PLK1 through inhibition of IKKε activity
18. 367 Ubiquitin-regulated recruitment of IKKϵ to MAVS interferon signaling adapter
19. 291 Polo-like kinase 1 (PLK1) and protein kinase IKKϵ, two markers of cancer, down-regulate IFN induction by MAVS
20. The protein kinase IKKε can inhibit HCV expression independently of IFN and its own expression is downregulated in HCV-infected livers
21. Development of molecular and cellular tools to decipher the type I IFN pathway of the common vampire bat
22. Inhibition of the inflammatory response to stress by targeting interaction between PKR and its cellular activator PACT
23. Genetic variations in toll-like receptors 7 and 8 modulate natural hepatitis C outcomes and liver disease progression
24. Dual Kinase Inhibition Affords Extended in vitro Neuroprotection in Amyloid-β Toxicity
25. Genetic variations in toll‐like receptors 7 and 8 modulate natural hepatitis C outcomes and liver disease progression.
26. Ubiquitin-regulated recruitment of IkappaB kinase epsilon to the MAVS interferon signaling adapter
27. 57
28. NS3 of Bluetongue Virus Interferes with the Induction of Type I Interferon
29. The Double-Stranded RNA Bluetongue Virus Induces Type I Interferon in Plasmacytoid Dendritic Cells via a MYD88-Dependent TLR7/8-Independent Signaling Pathway
30. Hepatitis C Virus Reveals a Novel Early Control in Acute Immune Response
31. The PKR Activator PACT Is Induced by Aβ: Involvement in Alzheimer's Disease
32. A functional C-terminal TRAF3-binding site in MAVS participates in positive and negative regulation of the IFN antiviral response
33. CS2-1 RIG-I like receptors: sensing and responding to RNA virus infection
34. Hepatitis C Virus Controls Interferon Production through PKR Activation
35. ER targeting and retention of the HCV NS4B protein relies on the concerted action of multiple structural features including its transmembrane domains
36. ADAR1 Interacts with PKR during Human Immunodeficiency Virus Infection of Lymphocytes and Contributes to Viral Replication
37. Polo-like Kinase 1 (PLK1) Regulates Interferon (IFN) Induction by MAVS
38. Ubiquitin-Regulated Recruitment of IκB Kinase ε to the MAVS Interferon Signaling Adapter
39. TRBP Control of PACT-Induced Phosphorylation of Protein Kinase R Is Reversed by Stress
40. Regulation of Interferon Production by RIG-I and LGP2: A Lesson in Self-Control
41. The interferon inducing pathways and the hepatitis C virus
42. A hepatitis C virus (HCV) NS3/4A protease-dependent strategy for the identification and purification of HCV-infected cells
43. The N-terminus of PKR is responsible for the activation of the NF-κB signaling pathway by interacting with the IKK complex
44. Inhibition of RIG-I-Dependent Signaling to the Interferon Pathway during Hepatitis C Virus Expression and Restoration of Signaling by IKKε
45. The TAR RNA-binding Protein, TRBP, Stimulates the Expression of TAR-containing RNAs in Vitro and in VivoIndependently of Its Ability to Inhibit the dsRNA-dependent Kinase PKR
46. Two Dimerization Domains in the Trans-activation Response RNA-binding Protein (TRBP) Individually Reverse the Protein Kinase R Inhibition of HIV-1 Long Terminal Repeat Expression
47. PKR Stimulates NF-κB Irrespective of Its Kinase Function by Interacting with the IκB Kinase Complex
48. Activation of p38 Mitogen-Activated Protein Kinase and c-Jun NH 2 -Terminal Kinase by Double-Stranded RNA and Encephalomyocarditis Virus: Involvement of RNase L, Protein Kinase R, and Alternative Pathways
49. Full Peptide Synthesis, Purification, and Characterization of Six Tat Variants
50. Human PKR Transfected into Murine Cells Stimulates Expression of Genes under Control of the HIV1 or HTLV-1 LTR
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