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153 results on '"Mitropoulou, V."'

2. Constraints on the dynamics and spatio-temporal evolution of the 2011 Oichalia seismic swarm (SW Peloponnesus, Greece)

Author

Kassaras, I. Kapetanidis, V. Karakonstantis, A. Kouskouna, V. Ganas, A. Chouliaras, G. Drakatos, G. Moshou, A. Mitropoulou, V. Argyrakis, P. Lekkas, E. Makropoulos, K.

Abstract

In this paper, we present a detailed study of a shallow seismic swarm which took place in the area of Oichalia (SW Peloponnesus), between August and December 2011. The seismic crisis started on 14/8/2011 with an Mw=4.8 earthquake and was followed by more than 1600 events, several of which having magnitude over 4.0. The activity was recorded by local temporary and regional permanent seismic stations. Thousands of records were collected and routinely analyzed. P- and S-wave arrival times were manually picked and incorporated in the HYPOINVERSE algorithm together with a new optimum local velocity model. Hypocentral solutions were improved by applying a double-difference method. Focal mechanisms show that the activated fault zone is dominated by dip-slip normal faulting, trending NNW-SSE, with the average T-axes orientation being N70°E, consistent with regional tectonics.We have investigated towards stress triggering and fluid diffusion, by employing Coulomb stress transfer, spatio-temporal and Frequency-Magnitude Distribution (FMD) analyses. The negligible Coulomb stress transfer and seismicity rate changes that were calculated imply for a stress deficit in the broader study area, hence an external triggering mechanism is required to justify the observed pattern. The b-values increase towards the SSE, compatible with the similarly directed migration of seismicity, showed that the Oichalia swarm could possibly be adapted to an Epidemic Type Aftershock Sequence model (ETAS). Fluid diffusion is reflected in the spatio-temporal hypocenter migration. Clustering analysis, combined with the temporal distribution of b-values, has shown that the swarm evolved in three major phases, the first two being initiated by major events, which were probably triggered externally due to fluid injection that brought the seismogenic volume into a critical state, likely followed by afterslip. The last phase signified a relaxation period, with dispersed seismicity throughout the area and the b-values gently diminishing towards unity. © 2013 Elsevier B.V.

Published
2014

3. Affective instability and impulsivity in borderline personality and bipolar II disorders : similarities and differences

Author

Henry, C., Mitropoulou, V., New, A.S., Koenigsberg, H.W., Silverman, J., Siever, L.J., Unité mixte de recherche neurobiologie intégrative, and Université Bordeaux Segalen - Bordeaux 2-Institut National de la Recherche Agronomique (INRA)

Subjects
TROUBLE BIPOLAIRE, [SDV]Life Sciences [q-bio], ComputingMilieux_MISCELLANEOUS
Abstract

International audience

Published
2001

33. MONOAMINES IN PERSONALITY DISORDER

Author

SIEVER, L. J., primary, TRESTMAN, R. L., additional, COCCARO, E., additional, AMIN, F., additional, LAWRENCE, T., additional, GABRIEL, S., additional, and MITROPOULOU, V., additional

Published
1992
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34. Relationship between depression and borderline personality disorder.

Author

Koenigsberg, Harold W., Anwunah, Iseoma, New, Antonia S., Mitropoulou, Vivian, Schopick, Frances, Siever, Larry J., Koenigsberg, H W, Anwunah, I, New, A S, Mitropoulou, V, Schopick, F, and Siever, L J

Subjects
BORDERLINE personality disorder, PERSONALITY disorders, MENTAL depression, PSYCHOSOCIAL factors, ANXIETY
Abstract

The frequent occurrence of depressive symptoms in patients with borderline personality disorder has generated considerable interest in the nature of the relationship between borderline personality disorder and the depressive disorders. Data from the perspectives of phenomenology, biology, family history, course of illness, comorbidity patterns, and treatment response have been brought to bear on the question. Reviews based on research available by 1985 and 1991, respectively, arrived at differing conclusions: (1) that both disorders shared common but non-specific sources, and (2) that the two disorders were unrelated but co-occurred because of the high prevalence of each. Since the time of these reviews, additional evidence has become available from a wider range of biological investigations, better controlled comorbidity studies, studies of the relationship of psychosocial stressors to the course of each disorder and neuroimaging studies. In reviewing the more recent findings, we propose the less parsimonious hypothesis that the disorders co-occur, both because they share some common biological features and because the psychosocial sequella of each can contribute to the development of the other. [ABSTRACT FROM AUTHOR]

Published
1999
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