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3. Loss of function in NSD2 causes DNA methylation signature similar to that in Wolf-Hirschhorn syndrome

7. Whole-exome analysis of 177 pediatric patients with undiagnosed diseases

9. Clinical delineation, sex differences, and genotype–phenotype correlation in pathogenic KDM6A variants causing X-linked Kabuki syndrome type 2

10. CDK19-related disorder results from both loss-of-function and gain-of-function de novo missense variants

17. Integrated diagnosis based on transcriptome analysis in suspected pediatric sarcomas

18. The ARID1B spectrum in 143 patients: from nonsyndromic intellectual disability to Coffin–Siris syndrome

19. A Syndromic Neurodevelopmental Disorder Caused by Mutations in SMARCD1, a Core SWI/SNF Subunit Needed for Context-Dependent Neuronal Gene Regulation in Flies

21. Genetic abnormalities in a large cohort of Coffin–Siris syndrome patients

23. Mowat-Wilson syndrome: growth charts

25. Clinical diversity and molecular mechanism of VPS35L-associated Ritscher-Schinzel syndrome

28. DNA methylation signature inNSD2loss-of-function variants appeared similar to that in Wolf-Hirschhorn syndrome

31. Correction: The ARID1B spectrum in 143 patients: from nonsyndromic intellectual disability to Coffin–Siris syndrome

32. SNP array screening of cryptic genomic imbalances in 450 Japanese subjects with intellectual disability and multiple congenital anomalies previously negative for large rearrangements

34. Clinical diversity and molecular mechanism of VPS35L-associated Ritscher-Schinzel syndrome

36. Spectrum of mutations and genotype–phenotype analysis in Noonan syndrome patients with RIT1 mutations

38. Detailed analysis of 26 cases of 1q partial duplication/triplication syndrome

44. The spectrum of ZEB2 mutations causing the Mowat–Wilson syndrome in Japanese populations

46. Molecular and clinical findings and their correlations in Silver-Russell syndrome: implications for a positive role of IGF2 in growth determination and differential imprinting regulation of the IGF2–H19 domain in bodies and placentas

47. Clinical manifestations in patients with SOS1 mutations range from Noonan syndrome to CFC syndrome

48. Establishment of in-hospital clinical network for patients with neurofibromatosis type 1 in Nagoya University Hospital

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