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1. Cardiovascular Magnetic Resonance-derived Fontan Hemodynamics Are Associated with Reduced Kidney Function but Not Albuminuria in Single Ventricle Patients

Author

Jef Van den Eynde, MD, Jos Westenberg, PhD, Mark Hazekamp, MD, PhD, Hildo Lamb, MD, PhD, Monique Jongbloed, MD, PhD, Jolanda Wentzel, MD, PhD, Sasa Kenjeres, PhD, Ilona Dekkers, MD, PhD, Alexander Van De Bruaene, MD, PhD, Friso Rijnberg, MD, and Arno Roest, MD, PhD

Subjects
Diseases of the circulatory (Cardiovascular) system, RC666-701
Published
2024
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4. Radiofrequency Catheter Ablation of Idiopathic Right Ventricular Outflow Tract Arrhythmias

Author

Naiara Calvo, MD, Monique Jongbloed, MD, PhD, and Katja Zeppenfeld, MD, PhD

Subjects
ventricular arrhythmias, outflow tract, premature ventricular contractions, ablation, Diseases of the circulatory (Cardiovascular) system, RC666-701
Abstract

Idiopathic ventricular arrhythmias (VA) consist of various subtypes of VA that occur in the absence of clinically apparent structural heart disease. Affected patients account for approximately 10% of all patients referred for evaluation of ventricular tachycardia (VT). Arrhythmias arising from the outflow tract (OT) are the most common subtype of idiopathic VA and more than 70–80% of idiopathic VTs or premature ventricular contractions (PVCs) originate from the right ventricular (RV) OT. Idiopathic OT arrhythmias are thought to be caused by adenosine-sensitive, cyclic adenosine monophosphate (cAMP) mediated triggered activity and, in general, manifest at a relatively early age. Usually they present as salvos of paroxysmal ventricular ectopic beats and are rarely life-threatening. When highly symptomatic and refractory to antiarrhythmic therapy or causative for ventricular dysfunction, ablation is a recommended treatment with a high success rate and a low risk of complications.

Published
2013
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7. The coronary arteries in adults after arterial switch: a systematic review

Author

Engele, L. J., Mulder, B. J. M., Schoones, J. W., Kies, P., Egorova, A. D., Vliegen, H. W., Hazekamp, M. G., Bouma, B. J., and Monique Jongbloed

Subjects
Cardiology and Cardiovascular Medicine
Abstract

Background Coronary artery status in adults longterm after the arterial switch operation (ASO) is unclear. As a consequence, current follow-up strategies for coronary assessment remain controversial. We conducted a systemic review to provide an overview of coronary complications during adulthood and to evaluate the value of coronary imaging in adults after ASO, in light of current guidelines. Material and method Studies describing coronary complications or coronary imaging after ASO in adults were considered eligible for review and analysis. Articles were screened for the inclusion of adult ASO patients and data on coronary complications and findings of coronary imaging were collected. In cohort studies with both adults (≥18 years) and non-adults ( Results A total of 993 adults were followed with a median follow-up of 2.0 years after reaching adulthood. Myocardial ischemia was suspected in 16/192 patients (6.8%). The number of coronary interventions was 4 (0.4%) and coronary death was reported in 4 (0.4%) patients. The following coronary abnormalities were found by routine coronary computer tomography CT (cCT): stenosis (4%), acute angle (40%), kinking (24%) and interaterial course (11%). No coronary events were reported during pregnancy (n=45). Conclusion The reported number of coronary interventions (0.4%) and of coronary death (0.4%) during a median follow-up of 2 years in 993 ASO adults is low. Coronary abnormalities including acute angle, kinking and interarterial course were commonly found by cCT. The 2020 European Society of Cardiology (ESC) guidelines state that routine screening for coronary pathologies is questionable. However, based on current findings and in line with the 2018 American ACC/AHA guidelines we suggest a baseline assessment of the coronary arteries in all adult ASO patients. Thereafter, an individualized coronary follow-up strategy, based upon coronary findings, is advisable. Funding Acknowledgement Type of funding sources: Foundation. Main funding source(s): Hartstichting Freedom from coronary complications

Published
2021
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8. Clinical course long after atrial switch: a novel risk score for serious clinical events

Author

Woudstra, O., Zandstra, T. E., Vogel, R. F., Dijk, A. P. J., Vliegen, H. W., Kies, P., Monique Jongbloed, Egorova, A. D., Doevendans, P. A., Konings, T. C., Mulder, B. J. M., Tanck, M. W. T., Meijboom, F. J., Bouma, B. J., General practice, Pathology, Cardiology, ACS - Heart failure & arrhythmias, and Amsterdam Reproduction & Development (AR&D)

Published
2020

10. Apoptosis and epicardial contributions act as complementary factors in remodeling of the atrioventricular canal myocardium and atrioventricular conduction patterns in the embryonic chick heart

Author

Rebecca, Vicente Steijn, David, Sedmera, Nico A, Blom, Monique, Jongbloed, Alena, Kvasilova, and Ondrej, Nanka

Subjects
Pre-Excitation Syndromes, Ventricular Remodeling, Heart Conduction System, cardiovascular system, Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels, Animals, Apoptosis, cardiovascular diseases, Atrial Remodeling, Chick Embryo, Epithelial Cell Adhesion Molecule, Pericardium
Abstract

Background: During heart development, it has been hypothesized that apoptosis of atrioventricular canal myocardium and replacement by fibrous tissue derived from the epicardium are imperative to develop a mature atrioventricular conduction. To test this, apoptosis was blocked using an established caspase inhibitor and epicardial growth was delayed using the experimental epicardial inhibition model, both in chick embryonic hearts. Results: Chicken embryonic hearts were either treated with the peptide caspase inhibitor zVAD-fmk by intrapericardial injection in ovo (ED4) or underwent epicardial inhibition (ED2.5). Spontaneously beating embryonic hearts isolated (ED7–ED8) were then stained with voltage-sensitive dye Di-4-ANEPPS and imaged at 0.5–1 kHz. Apoptotic cells were quantified (ED5–ED7) by whole-mount LysoTracker Red and anti-active caspase 3 staining. zVAD-treated hearts showed a significantly increased proportion of immature (base to apex) activation patterns at ED8, including ventricular activation originating from the right atrioventricular junction, a pattern never observed in control hearts. zVAD-treated hearts showed decreased numbers of apoptotic cells in the atrioventricular canal myocardium at ED7. Hearts with delayed epicardial outgrowth showed also increased immature activation patterns at ED7.5 and ED8.5. However, the ventricular activation always originated from the left atrioventricular junction. Histological examination showed no changes in apoptosis rates, but a diminished presence of atrioventricular sulcus tissue compared with controls. Conclusions: Apoptosis in the atrioventricular canal myocardium and controlled replacement of this myocardium by epicardially derived HCN4-/Trop1- sulcus tissue are essential determinants of mature ventricular activation pattern. Disruption can lead to persistence of accessory atrioventricular connections, forming a morphological substrate for ventricular pre-excitation. Developmental Dynamics 247:1033-1042, 2018. © 2018 Wiley Periodicals, Inc.

Published
2018

11. Contributors

Author

Philip Aagaard, Dominic James Abrams, Hugues Abriel, Wayne O. Adkisson, Esperanza Agullo-Pascual, Francisco J. Alvarado, Ahmad S. Amin, Charles Antzelevitch, Justus M.B. Anumonwo, Luciana Armaganijan, Arash Arya, Samuel Asirvatham, Felipe Atienza, Peter H. Backx, Lisa M. Ballou, Elise Balse, Sujata Balulad, Andrea Barbuti, Gust H. Bardy, Guillaume Bassil, David G. Benditt, Omer Berenfeld, Donald M. Bers, Ofer Binah, Frank Bogun, Rossana Bongianino, Noel G. Boyle, Patrick M. Boyle, Günter Breithardt, Marisa Brini, Peter R. Brink, Pedro Brugada, Eric Buch, Feliksas F. Bukauskas, Hugh Calkins, David J. Callans, Sean M. Caples, Ernesto Carafoli, William A. Catterall, Marina Cerrone, Arnaud Chaumeil, Caressa Chen, Lan S. Chen, Peng-Sheng Chen, Jianding Cheng, Nipavan Chiamvimonvat, David J. Christini, Aman Chugh, Andreu M. Climent, Ira S. Cohen, Stuart J. Connolly, Lebron Cooper, Eric M. Crespo, Lia Crotti, Thomas A. Csepe, Frank Cuoco, Anne B. Curtis, Ralph J. Damiano, Dawood Darbar, Mithilesh K. Das, Andre d’Avila, Mario Delmar, Eva Delpón, Marco Denegri, Arnaud Denis, Nicolas Derval, Isabelle Deschênes, Abhishek Deshmukh, Luigi Di Biase, Timm M. Dickfeld, Hans Dierckx, Borislav Dinov, Sanjay Dixit, Dobromir Dobrev, Remi Dubois, Lars Eckardt, Andrew G. Edwards, Kenneth A. Ellenbogen, Patrick T. Ellinor, N.A. Mark Estes, Larissa Fabritz, Vadim V. Fedorov, Antonio B. Fernandez, Elvis Teijeira Fernández, David Filgueiras-Rama, Michael C. Fishbein, Glenn I. Fishman, David S. Frankel, Paul Friedman, Antonio Frontera, Apoor S. Gami, Paul Garabelli, Alfred L. George, Edward P. Gerstenfeld, Sigfus Gizurarson, Michael R. Gold, Jeffrey J. Goldberger, Andrew Grace, Guido Grassi, Ruth Ann Greenfield, Wendy L. Gross, Blair P. Grubb, María S. Guillem, Sándor Györke, Michel Haïssaguerre, Johan Hake, Henry R. Halperin, Brian J. Hansen, Stéphane Hatem, David L. Hayes, Jordi Heijman, Todd J. Herron, Gerhard Hindricks, Mélèze Hocini, Stefan H. Hohnloser, David R. Holmes, Masahiko Hoshijima, Thomas J. Hund, Mathew D. Hutchinson, Leonard Ilkhanoff, Jodie Ingles, James E. Ip, Warren M. Jackman, Nicholas Jackson, Pierre Jaïs, José Jalife, Bong Sook Jhun, Roy M. John, Monique Jongbloed, Luc Jordaens, Jonathan M. Kalman, Timothy J. Kamp, Mohamed H. Kanj, Suraj Kapa, Beverly Karabin, Ioannis Karakikes, Demosthenes G. Katritsis, Kuljeet Kaur, Paulus Kirchhof, André G. Kléber, George J. Klein, Peter Kohl, Jayanthi N. Koneru, Jacob S. Koruth, Andrew D. Krahn, Trine Krogh-Madsen, Karl Heinz Kuck, Saurabh Kumar, Alexander Kushnir, Neal K. Lakdawala, Zachary W.M. Laksman, Rakesh Latchamsetty, Dennis H. Lau, Bruce B. Lerman, Richard Z. Lin, Shien-Fong Lin, Mark S. Link, Bin Liu, Christopher F. Liu, Deborah J. Lockwood, Anatoli N. Lopatin, Steven A. Lubitz, Rajiv Mahajan, Jonathan C. Makielski, Marek Malik, Francis E. Marchlinski, Steven M. Markowitz, Barry J. Maron, Martin S. Maron, Steven O. Marx, Stéphane Massé, Andrew D. McCulloch, Pippa McKelvie-Sebileau, Spencer J. Melby, Andreas Metzner, Anushka P. Michailova, Gregory F. Michaud, John M. Miller, Jyotsna Mishra, Raul D. Mitrani, Peter J. Mohler, Fred Morady, Robert J. Myerburg, Hiroshi Nakagawa, Chrishan Joseph Nalliah, Kumaraswamy Nanthakumar, Carlo Napolitano, Sanjiv M. Narayan, Andrea Natale, Stanley Nattel, Saman Nazarian, Thao P. Nguyen, Akihiko Nogami, Sami F. Noujaim, Karine Nubret Le Coniat, Brian Olshansky, Jin O-Uchi, Gavin Y. Oudit, Feifan Ouyang, Cevher Ozcan, Douglas L. Packer, Sandeep V. Pandit, Alexander V. Panfilov, David S. Park, Bence Patocskai, Dainius H. Pauza, Neringa Pauziene, Jonathan P. Piccini, Geoffrey S. Pitt, Sunny S. Po, Abhiram Prasad, Silvia G. Priori, Przemysław B. Radwański, Wouter-Jan Rappel, Michelle Reiser, Alejandro Jimenez Restrepo, Richard B. Robinson, Dan M. Roden, Michael R. Rosen, Raphael Rosso, Yoram Rudy, Kristina Rysevaite-Kyguoliene, Hani N. Sabbah, Frederic Sacher, Frank B. Sachse, Ardan M. Saguner, Prashanthan Sanders, Michael C. Sanguinetti, Pasquale Santangeli, Mohammad Sarraf, Jonathan Satin, Martin Jan Schalij, Benjamin J. Scherlag, Matthew R. Schill, J. William Schleifer, Richard B. Schuessler, Peter J. Schwartz, Timon Seeger, Christopher Semsarian, Gino Seravalle, Ashok J. Shah, Robin M. Shaw, Mark J. Shen, Win–Kuang Shen, Shey-Shing Sheu, Kalyanam Shivkumar, Jennifer N.A. Silva, Allan C. Skanes, Kyoko Soejima, Virend K. Somers, Dan Sorajja, Stavros Stavrakis, Christian Steinberg, Lynne Warner Stevenson, William G. Stevenson, Michael O. Sweeney, Charles Swerdlow, Masateru Takigawa, Juan Tamargo, Harikrishna Tandri, Usha B. Tedrow, Nathaniel Thompson, Paul D. Thompson, Gordon F. Tomaselli, Jeffrey A. Towbin, Natalia A. Trayanova, Martin Tristani-Firouzi, Zian H. Tseng, Akiko Ueda, Héctor H. Valdivia, Virginijus Valiunas, Christian van der Werf, George F. Van Hare, David Vidmar, Sami Viskin, Niels Voigt, Edward P. Walsh, Paul J. Wang, Xander H.T. Wehrens, Mark S. Weiss, Arthur A.M. Wilde, Bruce L. Wilkoff, Y. Joseph Woo, Joseph C. Wu, Raymond Yee, Junaid A.B. Zaman, Manuel Zarzoso, Emily P. Zeitler, Katja Zeppenfeld, Tarek Zghaib, Xiao-Dong Zhang, and Douglas P. Zipes

Published
2018
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14. Contributors

Author

Hugues Abriel, Wayne O. Adkisson, Esperanza Agullo-Pascual, Olujimi A. Ajijola, Amin Al-Ahmad, Oluseun Alli, Robert K. Altman, Elad Anter, Charles Antzelevitch, Justus M.B. Anumonwo, Luciana Armaganijan, Hiroshi Ashikaga, Felipe Atienza, Uma Mahesh R. Avula, Peter H. Backx, Elise Balse, Conor D. Barrett, David G. Benditt, Omer Berenfeld, Donald M. Bers, Charles I. Berul, A. Christian Blank, Raffaella Bloise, Frank Matthias Bogun, Martin Borggrefe, Noel G. Boyle, Günter Breithardt, Marisa Brini, Peter R. Brink, Josep Brugada, Pau Brugada, Pedro Brugada, Ramon Brugada, Victoria Brugada, Eric Buch, Feliksas F. Bukauskas, J. David Burkhardt, Nenad Bursac, Hugh Calkins, David J. Callans, Oscar Campuzano, Sean M. Caples, Ernesto Carafoli, Augustin Castellanos, William Catterall, Marina Cerrone, Lan S. Chen, Lei Chen, Peng-Sheng Chen, Ashley Chin, Aman Chugh, Ira S. Cohen, Stuart J. Connolly, Jason Constantino, Lia Crotti, Frank A. Cuoco, Anne B. Curtis, Ralph J. Damiano, Dawood Darbar, Mithilesh K. Das, Mario Delmar, Eva Delpón, Luigi Di Biase, Sanjay Dixit, Dobromir Dobrev, Derek J. Dosdall, John W. Dyer, Lars Eckardt, Andrew G. Edwards, Igor R. Efimov, Kenneth A. Ellenbogen, Patrick T. Ellinor, Emilia Entcheva, N.A. Mark Estes, Rodolphe Fischmeister, John D. Fisher, Glenn I. Fishman, David S. Frankel, Michael R. Franz, Paul A. Friedman, Victor F. Froelicher, Apoor S. Gami, Alfred L. George, Edward P. Gerstenfeld, Michael R. Gold, Jeffrey J. Goldberger, Eleonora Grandi, Richard A. Gray, William J. Groh, Blair P. Grubb, Michel Haissaguerre, Johan Hake, Henry R. Halperin, Louise Harris, Stéphane Hatem, David L. Hayes, Meleze Hocini, Stefan H. Hohnloser, David Richard Holmes, Masahiko Hoshijima, Yuxuan Hu, Thomas J. Hund, Mathew D. Hutchinson, Hye Jin Hwang, Raymond E. Ideker, Leonard Ilkhanoff, Jodie Ingles, Warren M. Jackman, Pierre Jais, José Jalife, Bong Sook Jhun, Roy M. John, Monique Jongbloed, Mark E. Josephson, Alan H. Kadish, Jérôme Kalifa, Jonathan M. Kalman, Timothy J. Kamp, Mohamed Hani Kanj, Beverly Karabin, Robert S. Kass, Demosthenes G. Katritsis, Kuljeet Kaur, Jong J. Kim, Paulus Kirchhof, André G. Kléber, George J. Klein, Peter Kohl, Aravindan Kolandaivelu, Andrew D. Krahn, Andrew Krumerman, Saurabh Kumar, Karl-Heinz Kuck, Edward G. Lakatta, Rakesh Latchamsetty, Dennis H. Lau, Bruce B. Lerman, Jérôme Leroy, William R. Lewis, Shien-Fong Lin, Mark S. Link, Christopher F. Liu, Deborah J. Lockwood, Peter Loh, Anatoli N. Lopatin, John C. Lopshire, Steven A. Lubitz, Christopher Madias, Aman Mahajan, Jonathan C. Makielski, Marek Malik, Victor A. Maltsev, Francis E. Marchlinski, Ariane J. Marelli, Steven M. Markowitz, Barry J. Maron, Jeffrey R. Martens, Steven O. Marx, Andrew D. McCulloch, Andreas Metzner, Anuska P. Michailova, John Michael Miller, Michelle Lynne Milstein, Peter Mohler, Fred Morady, Robert J. Myerburg, Hiroshi Nakagawa, Carlo Napolitano, Sanjiv M. Narayan, Andrea Natale, Stanley Nattel, Saman Nazarian, Jeanne M. Nerbonne, Fu Siong Ng, Akihiko Nogami, Sami F. Noujaim, Brian Olshansky, Hakan Oral, Jin O-Uchi, Feifan Ouyang, Cevher Ozcan, Douglas L. Packer, Olle Pahlm, Sandeep V. Pandit, David S. Park, Geoffrey S. Pitt, Sunny S. Po, Silvia G. Priori, Wouter-Jan Rappel, Vivek Y. Reddy, Jason O. Robertson, Richard B. Robinson, Dan M. Roden, Robert A. Rose, Michael R. Rosen, Raphael Rosso, Yoram Rudy, Jeremy N. Ruskin, Hani N. Sabbah, Frank B. Sachse, Lindsey L. Saint, Javier Saiz, José A. Sánchez-Chapula, Prashanthan Sanders, Michael C. Sanguinetti, Pasquale Santangeli, Georgia Sarquella-Brugada, Jonathan Satin, Martin Jan Schalij, Benjamin J. Scherlag, Rainer Schimpf, Georg Schmidt, Peter J. Schwartz, Christopher Semsarian, Ashok J. Shah, Robin Shaw, Shey Shing Sheu, Kalyanam Shivkumar, Allan C. Skanes, Virend K. Somers, Bruce S. Stambler, Adam B. Stein, Lynne Warner Stevenson, William G. Stevenson, Jian Sun, Richard Sutton, Michael O. Sweeney, Charles Swerdlow, Juan Tamargo, Harikrishna Tandri, Rabi Tawil, Usha Tedrow, Cecile Terrenoire, Catalina Tobón, Jeffrey A. Towbin, Natalia A. Trayanova, Martin Tristani-Firouzi, Richard G. Trohman, Zian H. Tseng, Mintu P. Turakhia, Ravi Vaidyanathan, Héctor H. Valdivia, Virginijus Valiunas, Marcel A.G. van der Heyden, Christian van der Werf, George F. Van, Marmar Vaseghi, Christian Veltmann, Victoria L. Vetter, Sami Viskin, Niels Voigt, Marc A. Vos, Galen S. Wagner, Paul J. Wang, Rukshen Weerasooriya, Arthur A.M. Wilde, Bruce L. Wilkoff, Erik Wissner, Y. Joseph Woo, Masatoshi Yamazaki, Felix Yang, Yael Yaniv, Sing-Chien Yap, Raymond Yee, Manuel Zarzoso, Katja Zeppenfeld, and Douglas P. Zipes

Published
2014
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18. Aortic stenosis: correlation of prenatal echocardiography to postmortem histology

Author

Zwanenburg, F., Munsteren, J. C., Wisse, L. J., Ruiter, M. C., Haak, M. C., and Monique Jongbloed

Subjects
Cardiology and Cardiovascular Medicine
Abstract

Background Foetal aortic stenosis (AS) is a challenging congenital heart disease considering its potential to progress during the course of pregnancy. Especially at midgestation, it remains extremely difficult to distinguish the cases that end up biventricular from the cases that will develop into an hypoplastic left heart syndrome. Purpose To test the hypothesis that the degree of myocardial maturation is a possible predictor of biventricular outcome, we present 4 cases of foetal AS with a varying degree of severity and uniquely correlate differences in myocardial function based on prenatal echocardiography to their post-mortem histopathologic maturation. Methods We selected 4 cases with midgestational AS from our tertiary foetal cardiology service between 2018–2020. Speckle tracking recordings of the cardiac four-chamber view were performed during routine foetal echocardiography to quantify myocardial wall motion as a marker for myocardial function. Three cases decided to terminate the pregnancy and donated the cardiac specimen. Immunohistochemical labelling (ICH) against key markers for myocardial maturation (troponin-I, N-cadherin, connexin-43, MLC2A, MLC2V and α-SMA) and fibrosis (Sirius Red) were compared with 2 normal foetal cardiac specimens. Results Two cases with critical AS presented extremely decreased global and segmental longitudinal strain (GLS and SLS) values (GLS −2% and −0.9%) in the left ventricle (LV), indicating an impaired myocardial wall deformation. Post-mortem ICH showed overt endocardial fibro-elastosis (EFE) and pathological fibrosis patterns in the subendocardial layer which was remarkably spatially correlated to the EFE. The cardiomyocytes were disorganised with reduced expression of troponin-I and disturbed expression of connexin-43. The remaining 2 cases had normal LV appearance on foetal echocardiography, showing a mild reduction in left ventricular GLS and SLS (GLS −11.8% and −14.2%). Post-mortem ICH of 1 of these cases showed mild EFE with a milder fibrosis pattern. Cardiomyocytes were less disorganised but also showed a disturbed expression of connexin-43. The 4th case continued the pregnancy and had a biventricular outcome. Conclusions This is a unique case series showing that myocardial function correlates with high extent to histology. The degree of the reduction in myocardial function corresponded with the amount of pathological fibrosis patterns and disorganisation of the cardiomyocyte network. Myocardial wall motion on foetal echocardiography seems to hold promise as a possible marker for cardiac maturation. Funding Acknowledgement Type of funding sources: None. Speckle tracking and fibrosis patterns

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