203 results on '"Mortera-Blanco, Teresa"'
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2. Germline UBA1 Variant With Somatic Amplification in a Woman With Inflammatory Diseases and Myelodysplastic Syndrome.
3. Patient-specific MDS-RS iPSCs define the mis-spliced transcript repertoire and chromatin landscape of SF3B1-mutant HSPCs
4. Pseudouridine-modified tRNA fragments repress aberrant protein synthesis and predict leukaemic progression in myelodysplastic syndrome
5. Competition of dual SF3B1mt clones in MDS-RS is associated with distinct RNA mis-splicing in hematopoietic stem cells
6. Early transfusion patterns improve the Molecular International Prognostic Scoring System (IPSS-M) prediction in myelodysplastic syndromes
7. Data from Erythroid Differentiation Enhances RNA Mis-Splicing in SF3B1-Mutant Myelodysplastic Syndromes with Ring Sideroblasts
8. Main supplemental file from Erythroid Differentiation Enhances RNA Mis-Splicing in SF3B1-Mutant Myelodysplastic Syndromes with Ring Sideroblasts
9. Data S5 from Erythroid Differentiation Enhances RNA Mis-Splicing in SF3B1-Mutant Myelodysplastic Syndromes with Ring Sideroblasts
10. A three-dimensional in vitro model of erythropoiesis recapitulates erythroid failure in myelodysplastic syndromes
11. Development of an ex vivo three dimensional (3-D) model of acute myeloid leukaemia (AML)
12. Erythroid differentiation enhances RNA mis-splicing in SF3B1-mutant myelodysplastic syndromes with ring sideroblasts
13. Supplementary Figure S7 from Integrated Genomic and Transcriptomic Analysis Improves Disease Classification and Risk Stratification of MDS with Ring Sideroblasts
14. Supplementary Results S1 from Integrated Genomic and Transcriptomic Analysis Improves Disease Classification and Risk Stratification of MDS with Ring Sideroblasts
15. Supplementary Table S1 from Integrated Genomic and Transcriptomic Analysis Improves Disease Classification and Risk Stratification of MDS with Ring Sideroblasts
16. Data from Integrated Genomic and Transcriptomic Analysis Improves Disease Classification and Risk Stratification of MDS with Ring Sideroblasts
17. SF3B1-initiating mutations in MDS-RSs target lymphomyeloid hematopoietic stem cells
18. P713: EXPLORING CLONAL COMPETITION THROUGH 12-YEAR FOLLOW-UP OF AN MDS-RS PATIENT WITH DUAL SF3B1 MUTATIONS
19. Integrated Genomic and Transcriptomic Analysis Improves Disease Classification and Risk Stratification of MDS with Ring Sideroblasts
20. Erythroid differentiation intensifies RNA mis-splicing inSF3B1-mutant myelodysplastic syndromes with ring sideroblasts
21. Myelodysplastic Syndromes Are Propagated by Rare and Distinct Human Cancer Stem Cells In Vivo
22. Long-Term Clonal Inversion in an MDS-RS Case with Dual SF3B1 Mutations
23. Transfusion Patterns during Early Follow-up Predict Overall Survival Independently of IPSS-M in Patients with Myelodysplastic Syndromes
24. The extent of residual WT HSPCs is associated with the degree of anemia in patients with SF3B1-mutated MDS-RS
25. Transfusion Patterns during Early Follow-up Predict Overall Survival Independently of IPSS-M in Patients with Myelodysplastic Syndromes
26. Long-term cytokine-free expansion of cord blood mononuclear cells in three-dimensional scaffolds
27. Aberrant splicing of genes involved in haemoglobin synthesis and impaired terminal erythroid maturation in SF3B1 mutated refractory anaemia with ring sideroblasts
28. Whole Transcriptome Analysis Identifies Distinct Gene Expression Profiles between SF3B1mut and SF3B1 wt Myelodysplastic Syndrome with Ring Sideroblasts
29. Isogenic MDS-RS Patient-Derived iPSCs Define the Mis-Spliced Transcript Repertoire and Chromatin Landscape of SF3B1-Mutant Hematopoietic Stem/Progenitor Cells
30. Integrative Analysis of Primary SF3B1 mt Ring Sideroblasts Provides Fundamental Insights into MDS-RS Pathogenesis and Dyserythropoiesis
31. Identification of an Expanded Immature Cell Population in Human Bone Marrow during Erythroid Responses to ESA Treatment
32. A three-dimensional in vitro model of erythropoiesis recapitulates erythroid failure in myelodysplastic syndromes
33. Myelodysplastic Syndromes Are Propagated by Rare and Distinct Human Cancer Stem Cells In Vivo
34. Perturbed hematopoietic stem and progenitor cell hierarchy in myelodysplastic syndromes patients with monosomy 7 as the sole cytogenetic abnormality
35. Aberrant splicing of genes involved in haemoglobin synthesis and impaired terminal erythroid maturation inSF 3B1mutated refractory anaemia with ring sideroblasts
36. Development of an Ex Vivo Three Dimensional (3-D) Model of Acute Myeloid Leukaemia (AML)
37. Myelodysplastic syndromes are propagated by rare and distinct human cancer stem cells in vivo.
38. Characterization of the Hematopoietic Stem and Progenitor Cell Hierarchy in Myelodysplastic Syndromes Patients with Monosomy 7 As the Sole Cytogenetic Abnormality
39. Diverse Genetic Lesions In Myelodysplastic Syndromes Originate Exclusively In Rare MDS Stem Cells
40. Hierarchical Analysis Of Recurrent Point Mutations In SF3B1 and TET2 In RARS Stem Cells
41. Erythropoiesis In SF3B1 Mutated RARS Is Disrupted During Terminal Erythroid Maturation
42. Ex Vivo Three-Dimensional (3D) Functional biomimicry for the Long-Term Cytokine-Free Culture of Human Primary Acute Myeloid Leukemia (AML)
43. Physiologic Erythropoiesis with Enhanced Globin Switching and Endogenous Cytokine Production in a Long-Term Serum-free in Vitro human Bone Marrow Biomimicry
44. Ex vivo Mimicry of Normal and Abnormal Human Hematopoiesis
45. Long-Term in Vitro Cytokine-Free and Serum-Free Culture of Human Cord Blood Mononuclear Cells in a Three-Dimensional Scaffold.
46. Stem cell bioprocessing: fundamentals and principles
47. Stem cell bioprocessing: fundamentals and principles
48. THE EXTENT OF RESIDUAL WT HEMATOPOIETIC STEM AND PROGENITOR CELLS IS ASSOCIATED WITH THE DEGREE OF ANEMIA IN SF3B1-MUTATED MDS-RS PATIENTS
49. Integrative Analysis of Primary SF3B1 mtRing Sideroblasts Provides Fundamental Insights into MDS-RS Pathogenesis and Dyserythropoiesis
50. Whole Transcriptome Analysis Identifies Distinct Gene Expression Profiles between SF3B1mutand SF3B1 wtMyelodysplastic Syndrome with Ring Sideroblasts
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