149 results on '"Mukohda, Masashi"'
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2. Emerging evidence for a cardiovascular protective effect of concentrated Japanese plum juice
3. Long-term treatment with the streptococcal exotoxin streptolysin O inhibits vascular smooth muscle contraction by inducing iNOS expression in endothelial cells
4. Increased Blood Pressure Causes Lymphatic Endothelial Dysfunction via Oxidative Stress in Spontaneously Hypertensive Rats
5. RhoBTB1 protects against hypertension and arterial stiffness by restraining phosphodiesterase 5 activity
6. Interference With Endothelial PPAR (Peroxisome Proliferator–Activated Receptor)-γ Causes Accelerated Cerebral Vascular Dysfunction in Response to Endogenous Renin-Angiotensin System Activation
7. Endothelial PPARγ (Peroxisome Proliferator–Activated Receptor-γ) Protects From Angiotensin II–Induced Endothelial Dysfunction in Adult Offspring Born From Pregnancies Complicated by Hypertension
8. Vascular contraction in response to protein kinase C activator was induced via protein kinase Cβ in rat carotid artery
9. Hypertension is linked to enhanced lymphatic contractile response via RGS16/RhoA/ROCK pathway
10. Abstract 003: Treatment With Ligilactobacillus Improves Gut Permeability And Hypertension In Spontaneously Hypertensive Rats
11. Abstract P125: Long-term Exposure Of Angiotensin II Enhanced Vascular Contraction Via Activation Of Protein Kinase C Beta
12. PPARγ Regulation in Hypertension and Metabolic Syndrome
13. Methylglyoxal Accumulation in Arterial Walls Causes Vascular Contractile Dysfunction in Spontaneously Hypertensive Rats
14. Exploring Mechanisms of Diabetes-Related Macrovascular Complications: Role of Methylglyoxal, a Metabolite of Glucose on Regulation of Vascular Contractility
15. PKCβ inhibitor blocked angiotensin II-induced vascular smooth muscle proliferation and oxidative stress
16. Gut microflora and metabolic syndrome: new insight into the pathogenesis of hypertension
17. Methylglyoxal Augments Angiotensin II–Induced Contraction in Rat Isolated Carotid Artery
18. Methylglyoxal Enhances Sodium Nitroprusside–Induced Relaxation in Rat Aorta
19. Methylglyoxal Inhibits Smooth Muscle Contraction in Isolated Blood Vessels
20. Streptococcal Exotoxin Streptolysin O Causes Vascular Endothelial Dysfunction Through PKCβ Activation
21. Mechanisms Underlying Pioglitazone-Mediated Relaxation in Isolated Blood Vessel
22. Streptolysin O: a novel mediator of endothelial dysfunction
23. Anti-inflammatory mechanisms of the vascular smooth muscle PPARγ
24. Long-term methylglyoxal treatment impairs smooth muscle contractility in organ-cultured rat mesenteric artery
25. Abstract P079: Lymphatic Contraction Was Enhanced In Spontaneously Hypertensive Rats
26. Failure to vasodilate in response to salt loading blunts renal blood flow and causes salt-sensitive hypertension
27. Bacterial toxin, streptolysin O caused vascular endothelial dysfunction: Relationship between dysbiosis and hypertension
28. Interference with Endothelial PPARγ Causes Accelerated Cerebral Vascular Dysfunction in Response to Endogenous Renin-Angiotensin System Activation
29. Abstract 065: Endothelial CULLIN3 Mutation Causes Vascular Dysfunction, Arterial Stiffening, and Hypertension
30. Abstract 120: Protective Role of Vascular Smooth Muscle Rho-Related BTB Domain Containing Protein 1 in Hypertension and Arterial Stiffness
31. Smooth Muscle PPARgamma Mutation Causes Impaired Renal Blood Flow and Salt‐Sensitive Hypertension
32. Protective Role of Vascular Smooth Muscle RhoBTB1 in Hypertension
33. Endothelial‐Specific Interference with PPARγ Causes Endothelial Dysfunction with Sex‐ Specific Mechanisms in Offspring Born from AVP‐infused Pregnancies
34. Role of PPARG, a Transcriptional Factor on Hypertension
35. Role of PPARγ, a transcription factor in cardiovascular disease
36. Thoracic duct function was impaired in spontaneously hypertensive rat
37. Abstract 094: Smooth Muscle PPARγ Mutation Causes Impaired Renal Blood Flow and Salt-Sensitive Hypertension
38. Abstract 133: Endothelial-Specific Interference With PPARγ Increases the Susceptibility to Angiotensin II-Induced Endothelial Dysfunction in Adult Offspring Born from AVP-Infused Pregnancies
39. Abstract 110: Vascular Smooth Muscle RhoBTB1 Protects From Hypertension and Arterial Stiffness by Cullin-3 Dependent Ubiquitination of Phosphodiesterase 5
40. Abstract 036: Interference With PPARγ in the Endothelium Produces Endothelial Dysfunction in the Cerebral Circulation in Response to Activation of the Endogenous Renin-Angiotensin System
41. Abstract 121: Endothelial Cullin3 Mutation Causes Vascular Dysfunction, Arterial Stiffening, and Hypertension
42. Endothelial Cullin3 Mutation Causes Vascular Dysfunction, Arterial Stiffening, and Hypertension
43. Cardiovascular Effects of Endothelial‐Specific Interference with PPARγ Activity in Offspring Born from AVP‐induced Preeclamptic Pregnancies
44. Smooth Muscle PPARγ Mutation Causes Impaired Renal Blood Flow and Salt‐Sensitive Hypertension
45. Endogenous Renin‐Angiotensin System Activation Causes Accelerated Cerebral Vascular Dysfunction in Mice Expressing Dominant‐Negative Mutations in PPARγ in Endothelium
46. Abstract 140: Endogenous Renin-angiotensin System Activation Causes Accelerated Cerebral Vascular Dysfunction in Mice Expressing Dominant-negative Mutations in PPARγ in Endothelium
47. Abstract 062: Vascular Dysfunction and Hypertension are Prevented by a Novel PPARγ Target Gene, RhoBTB1
48. Abstract 099: Smooth Muscle PPARγ Mutation Causes Salt-sensitive Hypertension
49. Abstract P189: Endothelial Cullin3 Mutation Causes Vascular Dysfunction, Arterial Stiffening, and Hypertension
50. Abstract 124: RhoBTB1, a Novel PPARγ Target Gene, Rescues Hypertension and Vascular Dysfunction Caused by PPARγ Dysfunction
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