134 results on '"Nagarajan, Uma M"'
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2. Chlamydia and Innate Immunity
3. Virulence Protein Pgp3 Is Insufficient To Mediate Plasmid-Dependent Infectivity of Chlamydia trachomatis
4. Induction and Function of Type I IFNs During Chlamydial Infection
5. IL-12 secretion by Langerhans cells stimulated with Candida skin test reagent is mediated by dectin-1 in some healthy individuals
6. Host–Pathogen Interactions of Chlamydia trachomatis in Porcine Oviduct Epithelial Cells
7. Reduced Uterine Tissue Damage during Chlamydia muridarum Infection in TREM-1,3-Deficient Mice
8. Tumor Necrosis Factor Alpha-Induced Interleukin-1 Alpha Synthesis and Cell Death Is Increased in Mouse Epithelial Cells Infected With Chlamydia muridarum
9. Immune Recognition and Host Cell Response duringChlamydiaInfection
10. IL-1α Is Essential for Oviduct Pathology during Genital Chlamydial Infection in Mice
11. Chlamydia and Innate Immunity
12. Human Fallopian Tube Epithelial Cell Culture Model To Study Host Responses to Chlamydia trachomatis Infection
13. The bare lymphocyte syndrome: molecular clues to the transcriptional regulation of major histocompatibility complex class II genes
14. Conserved residues of the bare lymphocyte syndrome transcription factor RFXAP determine coordinate MHC class II expression
15. Caspase-11 Contributes to Oviduct Pathology during Genital Chlamydia Infection in Mice
16. A Chlamydia-specific TCR Transgenic Mouse Demonstrates Th1 Polyfunctionality with Enhanced Effector Function 1
17. T Cell-Independent Gamma Interferon and B Cells Cooperate To Prevent Mortality Associated with Disseminated Chlamydia muridarum Genital Tract Infection
18. Discovery of Blood Transcriptional Endotypes in Women with Pelvic Inflammatory Disease
19. Differential signaling pathways are initiated in macrophages during infection depending on the intracellular fate ofChlamydiaspp.
20. A Chlamydia-Specific TCR-Transgenic Mouse Demonstrates Th1 Polyfunctionality with Enhanced Effector Function
21. A novel role for Connexin 43 in IFN beta expression during Chlamydia trachomatis infection and cytosolic DNA-sensing
22. Rationalized design of a mucosal vaccine protects against Mycobacterium tuberculosis challenge in mice
23. Guanylate Binding Proteins Enable Rapid Activation of Canonical and Noncanonical Inflammasomes in Chlamydia-Infected Macrophages
24. Guanylate Binding Proteins Enable Rapid Activation of Canonical and Noncanonical Inflammasomes in Chlamydia-Infected Macrophages
25. Significant role of IL-1 signaling, but limited role of inflammasome activation in oviduct pathology during Chlamydia muridarum genital infection1
26. The DNA Sensor, Cyclic GMP–AMP Synthase, Is Essential for Induction of IFN-β during Chlamydia trachomatis Infection
27. CD4+ T Cell Expression of MyD88 Is Essential for Normal Resolution of Chlamydia muridarum Genital Tract Infection
28. Significant Role of IL-1 Signaling, but Limited Role of Inflammasome Activation, in Oviduct Pathology during Chlamydia muridarum Genital Infection
29. Rationalized design of a mucosal vaccine protects against Mycobacterium tuberculosischallenge in mice
30. Interferon Regulatory Transcription Factor 3 Protects Mice from Uterine Horn Pathology during Chlamydia muridarum Genital Infection
31. Essential Role for Neutrophils in Pathogenesis and Adaptive Immunity in Chlamydia caviae Ocular Infections
32. MyD88 Deficiency Leads to Decreased NK Cell Gamma Interferon Production and T Cell Recruitment during Chlamydia muridarum Genital Tract Infection, but a Predominant Th1 Response and Enhanced Monocytic Inflammation Are Associated with Infection Resolution
33. Stimulator of IFN Gene Is Critical for Induction of IFN-β during Chlamydiamuridarum Infection
34. Host Chemokine and Cytokine Response in the Endocervix within the First Developmental Cycle of Chlamydia muridarum
35. Critical Role for Interleukin-1β (IL-1β) during Chlamydia muridarum Genital Infection and Bacterial Replication-Independent Secretion of IL-1β in Mouse Macrophages
36. NK cells inability to produce IFN gamma in MyD88-/- mice contributes to the delayed clearance of Chlamydia muridarum primary genital infection (133.2)
37. Effect of Chlamydiaphage φCPG1 on the Course of Conjunctival Infection with “ Chlamydia caviae ” in Guinea Pigs
38. Role for the Chlamydial Type III Secretion Apparatus in Host Cytokine Expression
39. Type I Interferon Signaling Exacerbates Chlamydia muridarum Genital Infection in a Murine Model
40. Inhibition of phago‐lysosome fusion by chlamydial elementary bodies is necessary for the early induction of type I IFNs in mouse macrophages
41. X Box-Like Sequences in the MHC Class II Region Maintain Regulatory Function
42. Chlamydia trachomatisInduces Expression of IFN-γ-Inducible Protein 10 and IFN-β Independent of TLR2 and TLR4, but Largely Dependent on MyD88
43. A Hierarchy of Nuclear Localization Signals Governs the Import of the Regulatory Factor X Complex Subunits and MHC Class II Expression
44. CHAPTER 10: IMMUNE RECOGNITION AND HOST CELL RESPONSE DURING CHLAMYDIA INFECTION.
45. Toll-Like Receptor-2, but Not Toll-Like Receptor-4, Is Essential for Development of Oviduct Pathology in Chlamydial Genital Tract Infection
46. Modulation of Gene Expression by the MHC Class II Transactivator
47. Class II Transactivator Is Required for Maximal Expression of HLA-DOB in B Cells
48. Associations and Interactions between Bare Lymphocyte Syndrome Factors
49. Novel Mutations Within the RFX-B Gene and Partial Rescue of MHC and Related Genes Through Exogenous Class II Transactivator in RFX-B-Deficient Cells
50. RFX-B Is the Gene Responsible for the Most Common Cause of the Bare Lymphocyte Syndrome, an MHC Class II Immunodeficiency
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