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20 results on '"Nakashima, Taiji"'

15. Impaired IL-17 Signaling Pathway Contributes to the Increased Collagen Expression in Scleroderma Fibroblasts

Author

Nakashima, Taiji, primary, Jinnin, Masatoshi, additional, Yamane, Keitaro, additional, Honda, Noritoshi, additional, Kajihara, Ikko, additional, Makino, Takamitsu, additional, Masuguchi, Shinichi, additional, Fukushima, Satoshi, additional, Okamoto, Yoshinobu, additional, Hasegawa, Minoru, additional, Fujimoto, Manabu, additional, and Ihn, Hironobu, additional

Published
2012
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19. Sevelamer hydrochloride use in a toddler

Author

Nakashima, Taiji, primary, Honda, Masataka, additional, Hataya, Hiroshi, additional, Hamasaki, Yuko, additional, Goto, Miwa, additional, Osako, Yutaka, additional, Nakata, Haruko, additional, Ishikura, Kenji, additional, and Ikeda, Masahiro, additional

Published
2005
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20. [Two cases of autosomal dominant polycystic kidney diseases who presented bilateral enlarged kidneys and severe hypertension in the neonatal period].

Author

Wakaki H, Ikeda M, Hataya H, Ishikura K, Nakashima T, Oosako Y, and Honda M

Subjects
Diagnosis, Differential, Female, Humans, Infant, Infant, Newborn, Male, Polycystic Kidney, Autosomal Dominant diagnosis, Hypertension, Renal etiology, Polycystic Kidney, Autosomal Dominant complications
Abstract

Autosomal dominant polycystic kidney disease(ADPKD) is rarely observed in the neonatal period. We report 2 cases of ADPKD who showed bilateral enlarged, hyperechoic kidneys and severe hypertension. It is difficult to differentiate ADPKD from autosomal recessive polycystic kidney disease (ARPKD) based on the initial clinical presentations in this period. In both cases, bilateral enlarged kidneys and severe hypertension were detected without oligohydramnion and respiratory distress. The mother of case 1 has polycystic kidneys. The father of case 2 was diagnosed as ADPKD. Case 2 had heart failure due to hypertension. Angiotensin converting enzyme inhibitor (ACE-I) was administered to both patients and resulted in good control of blood pressure. ADPKD in the neonatal and very early infantile period has diverse clinical courses. In general, although severe cases are rare, some cases have renal failure and/or hypertension as we reported. We emphasize that both the prompt diagnosis of ADPKD and the start of medication are of great importance in the neonatal and very early infantile period. We recommend that neonates and infants with a family history of ADPKD undergo screening including physical examinations, blood pressure measurements and urinalysis.

Published
2004

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