39 results on '"Naryzhnaya, Natalia V"'
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2. Peptides Are Cardioprotective Drugs of the Future: The Receptor and Signaling Mechanisms of the Cardioprotective Effect of Glucagon-like Peptide-1 Receptor Agonists
3. The role of β‐adrenergic receptors in the regulation of cardiac tolerance to ischemia/reperfusion. Why do β‐adrenergic receptor agonists and antagonists protect the heart?
4. The β2‐adrenergic receptor agonist formoterol attenuates necrosis and apoptosis in the rat myocardium under experimental stress‐induced cardiac injury.
5. Thymus in Cardiometabolic Impairments and Atherosclerosis: Not a Silent Player?
6. The role of adrenergic and muscarinic receptors in stress-induced cardiac injury
7. The Role of Microvascular Obstruction and Intra-Myocardial Hemorrhage in Reperfusion Cardiac Injury. Analysis of Clinical Data
8. Effect of Cold Adaptation on the State of Cardiovascular System and Cardiac Tolerance to Ischemia/Reperfusion Injury
9. Angiotensin 1–7 increases cardiac tolerance to ischemia/reperfusion and mitigates adverse remodeling of the heart—The signaling mechanism
10. δ-Opioid Receptor as a Molecular Target for Increasing Cardiac Resistance to Reperfusion in Drug Development
11. KATP channels are regulators of programmed cell death and targets for creation of novel drugs against ischemia/reperfusion cardiac injury
12. KATP channels are regulators of programmed cell death and targets for the creation of novel drugs against ischemia/reperfusion cardiac injury.
13. Association of Epicardial Adipose Tissue Adipocytes Hypertrophy with Biomarkers of Low-Grade Inflammation and Extracellular Matrix Remodeling in Patients with Coronary Artery Disease
14. The Infarct-Reducing Effect of the δ2 Opioid Receptor Agonist Deltorphin II: The Molecular Mechanism
15. Do reactive oxygen species damage or protect the heart in ischemia and reperfusion? Analysis on experimental and clinical data
16. A historical literature review of coronary microvascular obstruction and intra-myocardial hemorrhage as functional/structural phenomena
17. The effect of an adaptation to hypoxia on cardiac tolerance to ischemia/reperfusion.
18. Reperfusion Cardiac Injury: Receptors and the Signaling Mechanisms
19. Production of Reactive Oxygen Species by Epicardial Adipocytes Is Associated with an Increase in Postprandial Glycemia, Postprandial Insulin, and a Decrease in Serum Adiponectin in Patients with Severe Coronary Atherosclerosis
20. The Signaling Mechanism of Remote Postconditioning of the Heart: Prospects of the Use of Remote Postconditioning for the Treatment of Acute Myocardial Infarction.
21. The Infarct-Reducing Effect of the δ 2 Opioid Receptor Agonist Deltorphin II: The Molecular Mechanism.
22. Pyroptosis is a drug target for prevention of adverse cardiac remodeling: The crosstalk between pyroptosis, apoptosis, and autophagy
23. The effect of an adaptation to hypoxia on cardiac tolerance to ischemia/reperfusion
24. High carbohydrate high fat diet causes arterial hypertension and histological changes in the aortic wall in aged rats: The involvement of connective tissue growth factors and fibronectin
25. Cardioprotective and Vasoprotective Effects of Corticotropin-Releasing Hormone and Urocortins: Receptors and Signaling
26. The Role of Pyroptosis in Ischemic and Reperfusion Injury of the Heart
27. Preserved cardiac mitochondrial function and reduced ischaemia/reperfusion injury afforded by chronic continuous hypoxia: Role of opioid receptors
28. Takotsubo Syndrome: Clinical Manifestations, Etiology and Pathogenesis
29. Hypertrophy and Insulin Resistance of Epicardial Adipose Tissue Adipocytes: Association with the Coronary Artery Disease Severity
30. Comparative Analysis of the Cardioprotective Properties of Opioid Receptor Agonists in a Rat Model of Myocardial Infarction
31. Prospects for Creation of Cardioprotective and Antiarrhythmic Drugs Based on Opioid Receptor Agonists
32. Pharmacology of mitochondrial permeability transition pore inhibitors
33. Is oxidative stress of adipocytes a cause or a consequence of the metabolic syndrome?
34. The Involvement of Protein Kinases in the Cardioprotective Effect of Chronic Hypoxia.
35. Prospects for Creation of Cardioprotective Drugs Based on Cannabinoid Receptor Agonists
36. Prospects for Creation of Cardioprotective Drugs Based on Cannabinoid Receptor Agonists.
37. K ATP channels are regulators of programmed cell death and targets for the creation of novel drugs against ischemia/reperfusion cardiac injury.
38. Is oxidative stress of adipocytes a cause or a consequence of the metabolic syndrome?
39. Role of protein kinase C, PI3 kinase, tyrosine kinases, NO-synthase, KATP channels and MPT pore in the signaling pathway of the cardioprotective effect of chronic continuous hypoxia.
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