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35 results on '"Nathalie Philippe"'

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1. Involvement of a common progenitor cell in core binding factor acute myeloid leukaemia associated with mastocytosis

2. Écrivains migrants, littératures d’immigration, écritures diasporiques

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3. Duplication at chromosome 2q31.1-q31.2 in a family presenting syndactyly and nystagmus

4. Incidence and prognostic impact of c-Kit, FLT3, and Ras gene mutations in core binding factor acute myeloid leukemia (CBF-AML)

5. Outcome of Childhood Acute Promyelocytic Leukemia With All-Trans-Retinoic Acid and Chemotherapy

6. Several types of mutations of the Abl gene can be found in chronic myeloid leukemia patients resistant to STI571, and they can pre-exist to the onset of treatment

7. Long-term results of three randomized trials (58831, 58832, 58881) in childhood acute lymphoblastic leukemia: a CLCG-EORTC report

8. Another pedigree with familial acute myeloid leukemia and germline CEBPA mutation

9. Recurrent in-frame insertion in C/EBPα TAD2 region is a polymorphism without prognostic value in AML

10. Prevalence, clinical profile, and prognosis of NPM mutations in AML with normal karyotype

12. Incidence and prognostic value of TET2 alterations in de novo acute myeloid leukemia achieving complete remission

13. Wilms tumor 1 gene mutations are associated with a higher risk of recurrence in young adults with acute myeloid leukemia: a study from the Acute Leukemia French Association

14. High frequency of RUNX1 biallelic alteration in acute myeloid leukemia secondary to familial platelet disorder

15. BCR-ABL mutants spread resistance to non-mutated cells through a paracrine mechanism

16. RUNX1 DNA-binding mutations and RUNX1-PRDM16 cryptic fusions in BCR-ABL+ leukemias are frequently associated with secondary trisomy 21 and may contribute to clonal evolution and imatinib resistance

17. Cooperation of activating Ras/rtk signal transduction pathway mutations and inactivating myeloid differentiation gene mutations in M0 AML: a study of 45 patients

18. Mutations of PTPN11 are rare in adult myeloid malignancies

19. Molecular characterization of the idiopathic hypereosinophilic syndrome (HES) in 35 French patients with normal conventional cytogenetics

20. Incidence of childhood leukaemia and non-Hodgkin's lymphoma in France: National Registry of Childhood Leukaemia and Lymphoma, 1990-1999

21. Analysis of p16 gene deletion and point mutation in breast carcinoma

22. Coexistence of AML1/RUNX1 and BCR-ABL point mutations in an imatinib-resistant form of CML

23. Evaluation of Minimal Residual Disease Based on NPM1 Mutations in AML with Intermediate Risk Cytogenetics: A Prospective Study of 36 Patients

24. RUNX1 DNA-Binding Mutations and RUNX1-PRDM16 Cryptic Fusion in BCR/ABL+ Leukemias Are Frequently Associated with Secondary Trisomy 21 and May Contribute to Clonal Evolution and Imatinib Resistance

25. Trisomy 13, AML1 Mutations and M0 Subtype Are Remarkably Intricate Events in Acute Myeloid Leukemia (AML)

26. NPM Mutations in Adult AML with Normal Karyotype: A Retrospective Study of the Acute Leukemia French Association (ALFA)

27. Few Genes Expression Predicts Outcome in Adult Acute Myeloid Leukemia (AML) with Normal Karyotype

28. Incidence and Prognosis of RTKs and RAS Mutations in CBF AML. A Retrospective Study of French Adult ALFA and Pediatric LAME Trials

29. Ras Pathway Activation in Childhood B-Lineage Acute Lymphoblastic Leukemia: Ras, Ptpn11, and Flt3 Mutations

30. Incidence and Prognostic Impact of Gene Mutations in Older Patients with AML Treated in the ALFA-9801 Study

31. Association of TET2 Alterations with NPM1 Mutations and Prognostic Value in De Novo Acute Myeloid Leukemia (AML)

32. Incidence and Prognostic Impact of SNPs Regulating PU.1 Gene Expression in AML

33. Which AML Subsets Benefit from Leukemic Cell Priming during Chemotherapy? Long-Term Analysis of the ALFA-9802 GM-CSF Study

34. Molecular characterization of the idiopathic hypereosinophilic syndrome (HES) in 35 french patients with normal conventional cytogenetics

35. p16 gene homozygous deletions in acute lymphoblastic leukemia