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1. Patent Signals of IPO Performance: Evidence from High- and Low-Tech Industries in Japan

Author

Le Thuy Ngoc An, Yoshiyuki Matsuura, Norliza Che Yahya, and Nguyen Huu Phuc

Subjects
patent, signal, initial public offering (ipo), high-tech industries, low-tech industries, Technology (General), T1-995, Management. Industrial management, HD28-70
Abstract

Prior studies pointed to evidence that startups and venture capital (VC) companies tended to use different measures to provide signals to outsiders. This study adds to those previous insights by focusing on established firms’ patenting behaviors and their effect on the amount of money raised at the initial public offering (IPO). Since technology intensity may differ considerably between high and low-tech companies, our main interest in this paper lies on whether the significance of pre-IPO patenting activities as a predictor of IPO performance also varies between these two industry categories. Using cross-sectional data representing 308 Japanese industrial firms’ IPO commitments between 2000 and 2015, we find a robust positive correlation between patent applications and IPO performance. Contrary to the conventional wisdom proposing that high-tech firms with more patenting activities achieve better IPO performance, we show that the signaling power of patenting is stronger for the low-tech companies in our sample: While the high-tech firms do not seem to have significantly benefited from a patent signal, the low-tech firms seem to have attracted external investors more easily due to patenting at the IPO.

Published
2022
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14. Productive infection of primary human hepatocytes with SARS-CoV-2 induces antiviral and proinflammatory responses

Author

Heinen, Natalie, primary, Khanal, Rajendra, additional, Westhoven, Saskia, additional, Klöhn, Mara, additional, Herrmann, Simon T, additional, Herrmann, Maike, additional, Tuoc, Tran, additional, Ulmke, Pauline Antonie, additional, Nguyen, Hoang Duy, additional, Nguyen, Huu Phuc, additional, Steinmann, Eike, additional, Todt, Daniel, additional, Brown, Richard J P, additional, Sharma, Amar Deep, additional, and Pfaender, Stephanie, additional

Published
2023
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16. Association of the neonatal Fc receptor promoter variable number of tandem repeat polymorphism with immunoglobulin response in patients with chronic inflammatory demyelinating polyneuropathy.

Author

Fisse, Anna Lena, Schäfer, Emelie, Hieke, Alina, Schröder, Maximilian, Klimas, Rafael, Brünger, Jil, Huckemann, Sophie, Grüter, Thomas, Sgodzai, Melissa, Schneider‐Gold, Christiane, Gold, Ralf, Nguyen, Huu Phuc, Pitarokoili, Kalliopi, Motte, Jeremias, and Arning, Larissa

Subjects
POLYNEUROPATHIES, CHRONIC inflammatory demyelinating polyradiculoneuropathy, TANDEM repeats, FC receptors, GENE expression, INTRAVENOUS immunoglobulins
Abstract

Background and purpose: Chronic inflammatory demyelinating polyneuropathy (CIDP) is an autoimmune disease with humoral and cellular autoimmunity causing demyelination of peripheral nerves, commonly treated with intravenous immunoglobulins (IVIg). The neonatal Fc receptor (FcRn), encoded by the FCGRT gene, prevents the degradation of immunoglobulin G (IgG) by recycling circulating IgG. A variable number of tandem repeat (VNTR) polymorphism in the promoter region of the FCGRT gene is associated with different expression levels of mRNA and protein. Thus, patients with genotypes associated with relatively low FcRn expression may show a poorer treatment response to IVIg due to increased IVIg degradation. Methods: VNTR genotypes were analyzed in 144 patients with CIDP. Patients' clinical data, including neurological scores and treatment data, were collected as part of the Immune‐Mediated Neuropathies Biobank registry. Results: Most patients (n = 124, 86%) were VNTR 3/3 homozygotes, and 20 patients (14%) were VNTR 2/3 heterozygotes. Both VNTR 3/3 and VNTR 2/3 genotype groups showed no difference in clinical disability and immunoglobulin dosage. However, patients with a VNTR 2 allele were more likely to receive subcutaneous immunoglobulins (SCIg) than patients homozygous for the VNTR 3 allele (25% vs. 9.7%, p = 0.02) and were more likely to receive second‐line therapy (75% vs. 54%, p = 0.05). Conclusions: The VNTR 2/3 genotype is associated with the administration of SCIg, possibly reflecting a greater benefit from SCIg due to more constant immunoglobulin levels without lower IVIg levels between the treatment circles. Also, the greater need for second‐line treatment in VNTR 2/3 patients could be an indirect sign of a lower response to immunoglobulins. [ABSTRACT FROM AUTHOR]

Published
2024
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17. Early postnatal behavioral, cellular, and molecular changes in models of Huntington disease are reversible by HDAC inhibition

Author

Siebzehnrübl, Florian A., Raber, Kerstin A., Urbach, Yvonne K., Schulze-Krebs, Anja, Canneva, Fabio, Moceri, Sandra, Habermeyer, Johanna, Achoui, Dalila, Gupta, Bhavana, Steindler, Dennis A., Stephan, Michael, Nguyen, Huu Phuc, Bonin, Michael, Riess, Olaf, Bauer, Andreas, Aigner, Ludwig, Couillard-Despres, Sebastien, Paucar, Martin Arce, Svenningsson, Per, Osmand, Alexander, Andreew, Alexander, Zabel, Claus, Weiss, Andreas, Kuhn, Rainer, Moussaoui, Saliha, Blockx, Ines, Van der Linden, Annemie, Cheong, Rachel Y., Roybon, Laurent, Petersén, Åsa, and von Hörsten, Stephan

Published
2018

21. Heterozygous UCHL1 loss-of-function variants cause a neurodegenerative disorder with spasticity, ataxia, neuropathy, and optic atrophy

Author

Park, Joohyun, primary, Tucci, Arianna, additional, Cipriani, Valentina, additional, Demidov, German, additional, Rocca, Clarissa, additional, Senderek, Jan, additional, Butryn, Michaela, additional, Velic, Ana, additional, Lam, Tanya, additional, Galanaki, Evangelia, additional, Cali, Elisa, additional, Vestito, Letizia, additional, Maroofian, Reza, additional, Deininger, Natalie, additional, Rautenberg, Maren, additional, Admard, Jakob, additional, Hahn, Gesa-Astrid, additional, Bartels, Claudius, additional, van Os, Nienke J.H., additional, Horvath, Rita, additional, Chinnery, Patrick F., additional, Tiet, May Yung, additional, Hewamadduma, Channa, additional, Hadjivassiliou, Marios, additional, Downes, Susan M., additional, Németh, Andrea H., additional, Tofaris, George K., additional, Wood, Nicholas W., additional, Hayer, Stefanie N., additional, Bender, Friedemann, additional, Menden, Benita, additional, Cordts, Isabell, additional, Klein, Katrin, additional, Nguyen, Huu Phuc, additional, Krauss, Joachim K., additional, Blahak, Christian, additional, Strom, Tim M., additional, Sturm, Marc, additional, van de Warrenburg, Bart, additional, Lerche, Holger, additional, Maček, Boris, additional, Synofzik, Matthis, additional, Ossowski, Stephan, additional, Timmann, Dagmar, additional, Wolf, Marc E., additional, Smedley, Damian, additional, Riess, Olaf, additional, Schöls, Ludger, additional, Houlden, Henry, additional, Haack, Tobias B., additional, and Hengel, Holger, additional

Published
2023
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27. Implications of specific lysine residues within ataxin-3 for the molecular pathogenesis of Machado-Joseph disease

Author

Pereira Sena, Priscila, primary, Weber, Jonasz Jeremiasz, additional, Bayezit, Sercan, additional, Saup, Rafael, additional, Incebacak Eltemur, Rana Dilara, additional, Li, Xiaoling, additional, Velic, Ana, additional, Jung, Jaqueline, additional, Macek, Boris, additional, Nguyen, Huu Phuc, additional, Riess, Olaf, additional, and Schmidt, Thorsten, additional

Published
2023
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31. Structural architecture and brain network efficiency link polygenic scores to intelligence.

Author

Genç, Erhan, Metzen, Dorothea, Fraenz, Christoph, Schlüter, Caroline, Voelkle, Manuel C., Arning, Larissa, Streit, Fabian, Nguyen, Huu Phuc, Güntürkün, Onur, Ocklenburg, Sebastian, Kumsta, Robert, Genç, Erhan, Metzen, Dorothea, Fraenz, Christoph, Schlüter, Caroline, Voelkle, Manuel C., Arning, Larissa, Streit, Fabian, Nguyen, Huu Phuc, Güntürkün, Onur, Ocklenburg, Sebastian, and Kumsta, Robert

Abstract

Intelligence is highly heritable. Genome-wide association studies (GWAS) have shown that thousands of alleles contribute to variation in intelligence with small effect sizes. Polygenic scores (PGS), which combine these effects into one genetic summary measure, are increasingly used to investigate polygenic effects in independent samples. Whereas PGS explain a considerable amount of variance in intelligence, it is largely unknown how brain structure and function mediate this relationship. Here, we show that individuals with higher PGS for educational attainment and intelligence had higher scores on cognitive tests, larger surface area, and more efficient fiber connectivity derived by graph theory. Fiber network efficiency as well as the surface of brain areas partly located in parieto-frontal regions were found to mediate the relationship between PGS and cognitive performance. These findings are a crucial step forward in decoding the neurogenetic underpinnings of intelligence, as they identify specific regional networks that link polygenic predisposition to intelligence.

Published
2023

32. Differential diagnosis of chorea (guidelines of the German Neurological Society)

Author

Saft, Carsten; https://orcid.org/0000-0002-3983-7292, Burgunder, Jean-Marc, Dose, Matthias, Jung, Hans Heinrich, Katzenschlager, Regina, Priller, Josef, Nguyen, Huu Phuc, Reetz, Kathrin, Reilmann, Ralf, Seppi, Klaus, Landwehrmeyer, Georg Bernhard, Saft, Carsten; https://orcid.org/0000-0002-3983-7292, Burgunder, Jean-Marc, Dose, Matthias, Jung, Hans Heinrich, Katzenschlager, Regina, Priller, Josef, Nguyen, Huu Phuc, Reetz, Kathrin, Reilmann, Ralf, Seppi, Klaus, and Landwehrmeyer, Georg Bernhard

Abstract

Introduction Choreiform movement disorders are characterized by involuntary, rapid, irregular, and unpredictable movements of the limbs, face, neck, and trunk. These movements often initially go unnoticed by the affected individuals and may blend together with seemingly intended, random motions. Choreiform movements can occur both at rest and during voluntary movements. They typically increase in intensity with stress and physical activity and essentially cease during deep sleep stages. In particularly in advanced stages of Huntington disease (HD), choreiform hyperkinesia occurs alongside with dystonic postures of the limbs or trunk before they typically decrease in intensity. Summary or definition of the topic The differential diagnosis of HD can be complex. Here, the authors aim to provide guidance for the diagnostic process. This guidance was prepared for the German Neurological Society (DGN) for German-speaking countries. Recommendations Hereditary (inherited) and non-hereditary (non-inherited) forms of chorea can be distinguished. Therefore, the family history is crucial. However, even in conditions with autosomal-dominant transmission such as HD, unremarkable family histories do not necessarily rule out a hereditary form (e.g., in cases of early deceased or unknown parents, uncertainties in familial relationships, as well as in offspring of parents with CAG repeats in the expandable range (27–35 CAG repeats) which may display expansions into the pathogenic range). Conclusions The differential diagnosis of chorea can be challenging. This guidance prepared for the German Neurological Society (DGN) reflects the state of the art as of 2023.

Published
2023

33. Symptomatic treatment options for Huntington’s disease (guidelines of the German Neurological Society)

Author

Saft, Carsten; https://orcid.org/0000-0002-3983-7292, Burgunder, Jean-Marc, Dose, Matthias, Jung, Hans Heinrich, Katzenschlager, Regina, Priller, Josef, Nguyen, Huu Phuc, Reetz, Kathrin, Reilmann, Ralf, Seppi, Klaus, Landwehrmeyer, Georg Bernhard, Saft, Carsten; https://orcid.org/0000-0002-3983-7292, Burgunder, Jean-Marc, Dose, Matthias, Jung, Hans Heinrich, Katzenschlager, Regina, Priller, Josef, Nguyen, Huu Phuc, Reetz, Kathrin, Reilmann, Ralf, Seppi, Klaus, and Landwehrmeyer, Georg Bernhard

Abstract

Introduction Ameliorating symptoms and signs of Huntington’s disease (HD) is essential to care but can be challenging and hard to achieve. The pharmacological treatment of motor signs (e.g. chorea) may favorably or unfavorably impact other facets of the disease phenotype (such as mood and cognition). Similarly, pharmacotherapy for behavioral issues may modify the motor phenotype. Sometimes synergistic effects can be achieved. In patients undergoing pragmatic polypharmacological therapy, emerging complaints may stem from the employed medications' side effects, a possibility that needs to be considered. It is recommended to clearly and precisely delineate the targeted signs and symptoms (e.g., chorea, myoclonus, bradykinesia, Parkinsonism, or dystonia). Evidence from randomized controlled trials (RCTs) is limited. Summary or definition of the topic Therefore, the guidelines prepared for the German Neurological Society (DGN) for German-speaking countries intentionally extend beyond evidence from RCTs and aim to synthesize evidence from RCTs and recommendations of experienced clinicians. Recommendations First-line treatment for chorea is critically discussed, and a preference in prescription practice for using tiapride instead of tetrabenazine is noted. In severe chorea, combining two antidopaminergic drugs with a postsynaptic (e.g., tiapride) and presynaptic mode of action (e.g., tetrabenazine) is discussed as a potentially helpful strategy. Sedative side effects of both classes of compounds can be used to improve sleep if the highest dosage of the day is given at night. Risperidone, in some cases, may ameliorate irritability but also chorea and sleep disorders. Olanzapine can be helpful in the treatment of weight loss and chorea, and quetiapine as a mood stabilizer with an antidepressant effect. Conclusions Since most HD patients simultaneously suffer from distinct motor signs and distinct psychiatric/behavioral symptoms, treatment should be individually adapted.

Published
2023

34. Law on Compensation for Loss of Life and Health in the Field of Road Traffic

Author

Nguyen Huu Phuc

Subjects
General Engineering
Abstract

The article focuses on researching theoretical issues, and legal provisions on compensation for loss of life and health due to infringement in the field of road traffic. Besides, the article also studies the practical application of the law and the court's trial with some typical cases in recent times. Since then, providing solutions to improve the provisions of the law on compensation for loss of life and health due to infringement in the field of road traffic. To achieve those goals, the project must deal with specific issues such as a General overview of compensation for loss of life and health due to abuse in the field of road traffic; Analysis, assessment, and regulations on compensation for loss of life and health due to abuse in the field of road traffic according to current legal regulations; Analyze the current situation of applying regulations on compensation for loss of life and health due to abuse in the field of road traffic by the law with some recent typical cases. On that basis, propose solutions and proposals to improve the provisions of Vietnamese law on compensation for loss of life and health due to abuse in the field of road traffic.

Published
2022

35. Investigating the effects of additional truncating variants in DNA-repair genes on breast cancer risk in BRCA1-positive women

Author

Sepahi, Ilnaz, Faust, Ulrike, Sturm, Marc, Bosse, Kristin, Kehrer, Martin, Heinrich, Tilman, Grundman-Hauser, Kathrin, Bauer, Peter, Ossowski, Stephan, Susak, Hana, Varon, Raymonda, Schröck, Evelin, Niederacher, Dieter, Auber, Bernd, Sutter, Christian, Arnold, Norbert, Hahnen, Eric, Dworniczak, Bernd, Wang-Gorke, Shan, Gehrig, Andrea, Weber, Bernhard H. F., Engel, Christoph, Lemke, Johannes R., Hartkopf, Andreas, Nguyen, Huu Phuc, Riess, Olaf, and Schroeder, Christopher

Published
2019
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37. S3-Leitlinie: Diagnostik und Therapie des hepatozellulären Karzinoms

Author

Voesch Sabrina, Bitzer Michael, Albert Jörg, Bartenstein Peter, Bechstein Wolf, Blödt Susanne, Brunner Thomas, Dombrowski Frank, Evert Matthias, Follmann Markus, La Fougère Christian, Freudenberger Paul, Geier Andreas, Gkika Eleni, Götz Martin, Hammes Elke, Helmberger Thomas, Hoffmann Ralf-Thorsten, Hofmann Wolf-Peter, Huppert Peter, Kautz Achim, Knötgen Gabi, Körber Jürgen, Krug David, Lammert Frank, Lang Hauke, Langer Thomas, Lenz Philipp, Mahnken Andreas, Meining Alexander, Micke Oliver, Nadalin Silvio, Nguyen Huu Phuc, Ockenga Johann, Oldhafer Karl-Jürgen, Paprottka Philipp, Paradies Kerstin, Pereira Philippe, Persigehl Thorsten, Plauth Mathias, Plentz Ruben, Pohl Jürgen, Riemer Jutta, Reimer Peter, Ringwald Johanna, Ritterbusch Ulrike, Roeb Elke, Schellhaas Barbara, Schirmacher Peter, Schmid Irene, Schuler Andreas, von Schweinitz Dietrich, Seehofer Daniel, Sinn Marianne, Stein Alexander, Stengel Andreas, Steubesand Nadine, Stoll Christian, Tannapfel Andrea, Taubert Anne, Trojan Jörg, van Thiel Ingo, Tholen Reina, Vogel Arndt, Vogl Thomas, Vorwerk Hilke, Wacker Frank, Waidmann Oliver, Wedemeyer Heiner, Wege Henning, Wildner Dane, Wittekind Christian, Wörns Marcus-Alexander, Galle Peter, and Malek Nisar

Subjects
Gastroenterology
Published
2022

40. Evidences for Mutant Huntingtin Inducing Musculoskeletal and Brain Growth Impairments via Disturbing Testosterone Biosynthesis in Male Huntington Disease Animals

Author

Yu-Taeger, Libo, primary, Novati, Arianna, additional, Weber, Jonasz Jeremiasz, additional, Singer-Mikosch, Elisabeth, additional, Pabst, Ann-Sophie, additional, Cheng, Fubo, additional, Saft, Carsten, additional, Koenig, Jennifer, additional, Ellrichmann, Gisa, additional, Heikkinen, Taneli, additional, Pouladi, Mahmoud A., additional, Riess, Olaf, additional, and Nguyen, Huu Phuc, additional

Published
2022
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41. Optical Genome Mapping in MDS and AML as tool for structural variant profiling—comment and data update on Yang et al.: “High-resolution structural variant profiling of myelodysplastic syndromes by optical genome mapping uncovers cryptic aberrations of prognostic and therapeutic significance”

Author

Vangala, Deepak B., primary, Nilius-Eliliwi, Verena, additional, Gerding, Wanda M., additional, Schroers, Roland, additional, and Nguyen, Huu Phuc, additional

Published
2022
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44. BAF (mSWI/SNF) complex regulates mediolateral cortical patterning in the developing forebrain

Author

Nguyen, Huong, primary, Sokpor, Godwin, additional, Parichha, Arpan, additional, Pham, Linh, additional, Saikhedkar, Nidhi, additional, Xie, Yuanbin, additional, Ulmke, Pauline Antonie, additional, Rosenbusch, Joachim, additional, Pirouz, Mehdi, additional, Behr, Rüdiger, additional, Stoykova, Anastassia, additional, Brand-Saberi, Beate, additional, Nguyen, Huu Phuc, additional, Staiger, Jochen F., additional, Tole, Shubha, additional, and Tuoc, Tran, additional

Published
2022
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45. Heterozygous UCHL1 loss-of-function variants cause a neurodegenerative disorder with spasticity, ataxia, neuropathy, and optic atrophy

Author

Park, Joohyun, primary, Tucci, Arianna, additional, Cipriani, Valentina, additional, Demidov, German, additional, Rocca, Clarissa, additional, Senderek, Jan, additional, Butryn, Michaela, additional, Velic, Ana, additional, Lam, Tanya, additional, Galanaki, Evangelia, additional, Cali, Elisa, additional, Vestito, Letizia, additional, Maroofian, Reza, additional, Deininger, Natalie, additional, Rautenberg, Maren, additional, Admard, Jakob, additional, Hahn, Gesa-Astrid, additional, Bartels, Claudius, additional, van Os, Nienke J.H., additional, Horvath, Rita, additional, Chinnery, Patrick F., additional, Tiet, May Yung, additional, Hewamadduma, Channa, additional, Hadjivassiliou, Marios, additional, Tofaris, George K., additional, Wood, Nicholas W., additional, Hayer, Stefanie N., additional, Bender, Friedemann, additional, Menden, Benita, additional, Cordts, Isabell, additional, Klein, Katrin, additional, Nguyen, Huu Phuc, additional, Krauss, Joachim K., additional, Blahak, Christian, additional, Strom, Tim M., additional, Sturm, Marc, additional, van de Warrenburg, Bart, additional, Lerche, Holger, additional, Maček, Boris, additional, Synofzik, Matthis, additional, Ossowski, Stephan, additional, Timmann, Dagmar, additional, Wolf, Marc E., additional, Smedley, Damian, additional, Riess, Olaf, additional, Schöls, Ludger, additional, Houlden, Henry, additional, Haack, Tobias B., additional, Hengel, Holger, additional, Ambrose, J.C., additional, Arumugam, P., additional, Baple, E.L., additional, Bleda, M., additional, Boardman-Pretty, F., additional, Boissiere, J.M., additional, Boustred, C.R., additional, Brittain, H., additional, Caulfield, M.J., additional, Chan, G.C., additional, Craig, C.E.H., additional, Daugherty, L.C., additional, de Burca, A., additional, Devereau, A., additional, Elgar, G., additional, Foulger, R.E., additional, Fowler, T., additional, Furió-Tarí, P., additional, Hackett, J.M., additional, Halai, D., additional, Hamblin, A., additional, Henderson, S., additional, Holman, J.E., additional, Hubbard, T.J.P., additional, Ibáñez, K., additional, Jackson, R., additional, Jones, L.J., additional, Kasperaviciute, D., additional, Kayikci, M., additional, Lahnstein, L., additional, Lawson, K., additional, Leigh, S.E.A., additional, Leong, I.U.S., additional, Lopez, F.J., additional, Maleady-Crowe, F., additional, Mason, J., additional, McDonagh, E.M., additional, Moutsianas, L., additional, Mueller, M., additional, Murugaesu, N., additional, Need, A.C., additional, Odhams, C.A., additional, Patch, C., additional, Perez-Gil, D., additional, Polychronopoulos, D., additional, Pullinger, J., additional, Rahim, T., additional, Rendon, A., additional, Riesgo-Ferreiro, P., additional, Rogers, T., additional, Ryten, M., additional, Savage, K., additional, Sawant, K., additional, Scott, R.H., additional, Siddiq, A., additional, Sieghart, A., additional, Smedley, D., additional, Smith, K.R., additional, Sosinsky, A., additional, Spooner, W., additional, Stevens, H.E., additional, Stuckey, A., additional, Sultana, R., additional, Thomas, E.R.A., additional, Thompson, S.R., additional, Tregidgo, C., additional, Tucci, A., additional, Walsh, E., additional, Watters, S.A., additional, Welland, M.J., additional, Williams, E., additional, Witkowska, K., additional, Wood, S.M., additional, and Zarowiecki, M., additional

Published
2022
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47. A24 Mutant huntingtin impairs musculoskeletal and brain growth via disturbing testosterone biosynthesis in male huntington disease animals

Author

Yu-Taeger, Libo, primary, Novati, Arianna, additional, Weber, Jonasz Jeremiasz, additional, Singer-Mikosch, Elisabeth, additional, Pabst, Ann-Sophie, additional, Saft, Carsten, additional, König, Jennifer, additional, Ellrichmann, Gisa, additional, Heikkinen, Taneli, additional, Pouladi, Mahmoud A, additional, Riess, Olaf, additional, and Nguyen, Huu Phuc, additional

Published
2022
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50. I03 CAG-targeted brain-permeable protein lowering strategy tested in polyQ mouse models

Author

Surdyka, Magdalena, primary, Niewiadomska-Cimicka, Anna, additional, Kalinowska-Poska, Zaneta, additional, Jesion, Ewelina, additional, Singer-Mikosch, Elisabeth, additional, Fievet, Lorraine, additional, Fiszer, Agnieszka, additional, Caron, Nicholas S, additional, Hayden, Michael R, additional, Nguyen, Huu Phuc, additional, Trottier, Yvon, additional, and Figiel, Maciej, additional

Published
2022
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