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1. Quantifying regional α ‐synuclein, amyloid β, and tau accumulation in lewy body dementia

2. Sex Modulates the Pathological Aging Effect on Caudate Functional Connectivity in Mild Cognitive Impairment

3. Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease

4. Parkinson disease clinical subtypes: key features & clinical milestones

5. Analysis of neurodegenerative disease-causing genes in dementia with Lewy bodies

6. A Comprehensive Resource for Induced Pluripotent Stem Cells from Patients with Primary Tauopathies

7. Heritability and genetic variance of dementia with Lewy bodies

8. TREM2 brain transcript-specific studies in AD and TREM2 mutation carriers

9. Distinct cytokine profiles in human brains resilient to Alzheimer's pathology

10. Genetic variants associated with Alzheimer’s disease confer different cerebral cortex cell-type population structure

11. Utility of perfusion PET measures to assess neuronal injury in Alzheimer's disease

12. TDP-43 expression influences amyloidβ plaque deposition and tau aggregation

13. Prediction and Classification of Alzheimer’s Disease Based on Combined Features From Apolipoprotein-E Genotype, Cerebrospinal Fluid, MR, and FDG-PET Imaging Biomarkers

14. The Influence of Cerebrospinal Fluid Abnormalities and APOE 4 on PHF-Tau Protein: Evidence From Voxel Analysis and Graph Theory

15. New Perspective for Non-invasive Brain Stimulation Site Selection in Mild Cognitive Impairment: Based on Meta- and Functional Connectivity Analyses

16. Performing Sparse Regularization and Dimension Reduction Simultaneously in Multimodal Data Fusion

17. Quantitative 18F-AV1451 Brain Tau PET Imaging in Cognitively Normal Older Adults, Mild Cognitive Impairment, and Alzheimer's Disease Patients

18. Quantification of white matter cellularity and damage in preclinical and early symptomatic Alzheimer's disease

19. Amyloid-β Plaques in Clinical Alzheimer’s Disease Brain Incorporate Stable Isotope Tracer In Vivo and Exhibit Nanoscale Heterogeneity

20. Corrigendum: Conversion Discriminative Analysis on Mild Cognitive Impairment Using Multiple Cortical Features from MR Images

21. Conversion Discriminative Analysis on Mild Cognitive Impairment Using Multiple Cortical Features from MR Images

22. Shortfalls in the peptidyl-prolyl cis–trans isomerase protein Pin1 in neurons are associated with frontotemporal dementias

23. Neocortical Lewy Body Pathology Parallels Parkinson's Dementia, but Not Always

24. Differential Vulnerability of Hippocampal Subfields in Primary Age-Related Tauopathy and Chronic Traumatic Encephalopathy

25. New insights into the genetic etiology of Alzheimer's disease and related dementias

26. Clinical and genetic features of Lewy body pathology in autosomal dominant and sporadic Alzheimer disease

27. Genome-wide association study and functional validation implicates JADE1 in tauopathy

28. Identification of a sex-specific genetic signature in dementia with Lewy bodies: a meta-analysis of genome-wide association studies

29. The Second NINDS/NIBIB Consensus Meeting to Define Neuropathological Criteria for the Diagnosis of Chronic Traumatic Encephalopathy

30. Is Levodopa Response a Valid Indicator of Parkinson's Disease?

31. Comparing amyloid-β plaque burden with antemortem PiB PET in autosomal dominant and late-onset Alzheimer disease

32. TREM2 brain transcript-specific studies in AD and TREM2 mutation carriers

33. Tau PET in autosomal dominant Alzheimer’s disease: relationship with cognition, dementia and other biomarkers

34. Parkinson’s disease and multiple system atrophy have distinct α-synuclein seed characteristics

35. Widespread distribution of tauopathy in preclinical Alzheimer's disease

36. Gene Expression Imputation Across Multiple Tissue Types Provides Insight Into the Genetic Architecture of Frontotemporal Dementia and Its Clinical Subtypes

37. KL-VS heterozygosity is associated with lower amyloid-dependent tau accumulation and memory impairment in Alzheimer’s disease

38. Early Selective Vulnerability of the CA2 Hippocampal Subfield in Primary Age-Related Tauopathy

39. Tauopathy in autosomal dominant and late‐onset Alzheimer disease

40. Assessment of the contribution of common genetic variants associated with Alzheimer’s disease on neuropathological burden and clinical characteristics in the Brains for Dementia Research cohort

41. Mendelian randomization implies no direct causal association between leukocyte telomere length and amyotrophic lateral sclerosis

42. Genetic risk for Alzheimer’s disease influences neuropathology and cognition via multiple biological pathways

43. Genome sequencing analysis identifies new loci associated with Lewy body dementia and provides insights into the complex genetic architecture

44. Ante- and postmortem tau in autosomal dominant and late-onset Alzheimer's disease

45. Neuropathological findings in a South Korean patient with Perry syndrome

46. Parkinson disease clinical subtypes: key featuresclinical milestones

47. Analysis of neurodegenerative disease-causing genes in dementia with Lewy bodies

48. The utility of the National Alzheimer’s Coordinating Center’s database for the rapid assessment of evolving neuropathologic conditions

49. From basic research to the clinic: innovative therapies for ALS and FTD in the pipeline

50. The Revised National Alzheimer’s Coordinating Center’s Neuropathology Form—Available Data and New Analyses

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