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Your search keyword '"Nita, Dragos A."' showing total 38 results
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38 results on '"Nita, Dragos A."'

6. Inflammatory Brain Diseases

Author

Twilt, Marinka, Nita, Dragos A., Benseler, Susanne M., Wheeler, Derek S., editor, Wong, Hector R., editor, and Shanley, Thomas P., editor

Published
2014
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18. Interplay of brain structure and function in neonatal congenital heart disease

Author

Birca, Ala, primary, Vakorin, Vasily A., additional, Porayette, Prashob, additional, Madathil, Sujana, additional, Chau, Vann, additional, Seed, Mike, additional, Doesburg, Sam M., additional, Blaser, Susan, additional, Nita, Dragos A., additional, Sharma, Rohit, additional, Duerden, Emma G., additional, Hickey, Edward J., additional, Miller, Steven P., additional, and Hahn, Cecil D., additional

Published
2016
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27. Anti–Glutamic Acid Decarboxylase Antibody Associated Limbic Encephalitis in a Child: Expanding the Spectrum of Pediatric Inflammatory Brain Diseases.

Author

Mishra, Navin, Rodan, Lance H., Nita, Dragos A., Gresa-Arribas, Nuria, Kobayashi, Jeff, and Benseler, Susanne M.

Subjects
GLUTAMIC acid, AMNESIA, DECARBOXYLASE genetics, ENCEPHALITIS, AUTOIMMUNE diseases, THERAPEUTICS, DISEASE risk factors
Abstract

Anti–glutamic acid decarboxylase directed antibodies are a rare cause of autoimmune limbic encephalitis that is relatively resistant to immunotherapy. Here we report a 15-year-old boy with nonparaneoplastic, anti–glutamic acid decarboxylase limbic encephalitis presenting with subacute headache, memory disturbance, psychiatric symptoms, and seizures. At onset, his memory disturbance manifested as transient global amnesia-like episodes. Clinical remission was achieved with rituximab, intravenous immunoglobulin, and corticosteroids. [ABSTRACT FROM PUBLISHER]

Published
2014
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28. Posttraumatic Epilepsy: The Roles of Synaptic Plasticity.

Author

Timofeev, Igor, Bazhenov, Maksim, Avramescu, Sinziana, and Nita, Dragos A.

Subjects
EPILEPSY, NEUROPLASTICITY, PAROXYSMAL hemoglobinuria, SEIZURES (Medicine), BRAIN diseases
Abstract

Acute cerebral cortical trauma often leads to paroxysmal activities that terminate in a few hours, but several months later, patients can develop epilepsy. The process occurring between the initial acute triggered seizures and the onset of spontaneous unprovoked seizures is termed epileptogenesis. Here the authors summarize recent morphological, electrophysiological, and computational studies demonstrating that partial cortical isolation increases the number and duration of silent states in the cortical network, boosting neuronal connectivity and network excitability. These changes develop progressively, and after several weeks their synergetic action leads to epilepsy. [ABSTRACT FROM AUTHOR]

Published
2010
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