209 results on '"Onofri, Franco"'
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2. Temperature-Dependent Olive Pomace Extraction for Obtaining Bioactive Compounds Preventing the Death of Murine Cortical Neurons.
3. Intersectin associates with synapsin and regulates its nanoscale localization and function
4. Phosphatidylinositol 4-Kinase Type IIα Is Responsible for the Phosphatidylinositol 4-Kinase Activity Associated with Synaptic Vesicles
5. The LRRK2 N-terminal domain influences vesicle trafficking: impact of the E193K variant
6. Protein-Protein Interactions and Protein Modules in the Control of Neurotransmitter Release
7. Synapsin III Regulates Dopaminergic Neuron Development in Vertebrates
8. LRRK2 regulates dendritic spine dynamics through interaction with post-synaptic actin cytoskeleton
9. Synapsins Are Downstream Players of the BDNF-Mediated Axonal Growth
10. LRRK2 modulates neuronal vesicles cycle through protein interactions
11. Erk Activation in Axonal Varicosities Modulates Presynaptic Plasticity in the CA3 Region of the Hippocampus through Synapsin
12. Synapsin III Controls Early Phases of Dopaminergic Neurons Development in Fishes and Mammals by Acting Upstream of BDNF and Cdk5 Signaling
13. REST/NRSF drives homeostatic plasticity of inhibitory synapses in a target-dependent fashion
14. Author response: REST/NRSF drives homeostatic plasticity of inhibitory synapses in a target-dependent fashion
15. Interaction of Grb2 via its Src Homology 3 Domains with Synaptic Proteins Including Synapsin I
16. Interactions of Synapsin I with Phospholipids: Possible Role in Synaptic Vesicle Clustering and in the Maintenance of Bilayer Structures
17. PRRT2 modulates presynaptic Ca2+ influx by interacting with P/Q-type channels
18. SYN1 loss-of-function mutations in autism and partial epilepsy cause impaired synaptic function
19. Abnormal exocytotic release of glutamate in a mouse model of amyotrophic lateral sclerosis
20. A Bioactive Olive Pomace Extract Prevents the Death of Murine Cortical Neurons Triggered by NMDAR Over-Activation
21. Presynaptic L-Type Ca2+ Channels Increase Glutamate Release Probability and Excitatory Strength in the Hippocampus during Chronic Neuroinflammation
22. PRRT2 Modulates Presynaptic Ca 2+ Influx by Interacting with P/Q-Type Channels
23. Erratum: A novel topology of proline-rich transmembrane protein 2 (PRRT2): Hints for an intracellular function at the synapse (Journal of Biological Chemistry (2016) 291 (6111-6123) DOI: 10.1074/jbc.M115.683888)
24. REST/NRSF drives homeostatic plasticity of inhibitory synapses in a targetdependent fashion.
25. Synapsin I interacts with c-Src and stimulates its tyrosine kinase activity
26. Synapsin associates with cyclophilin B in an ATP- and cyclosporin A-dependent manner
27. S100A1 codistributes with synapsin I in discrete brain areas and inhibits the F-actin-bundling activity of synapsin I
28. Specific interactions of neuronal focal adhesion kinase isoforms with Src kinases and amphiphysin
29. The translocation of focal adhesion kinase in brain synaptosomes is regulated by phosphorylation and actin assembly
30. The Inhibitory Effects of Interleukin-6 on Synaptic Plasticity in the Rat Hippocampus Are Associated with an Inhibition of Mitogen-Activated Protein Kinase ERK
31. Responses of Neostriatal Dopaminoceptive Cells to the Ischemic Insult
32. Altered synaptic mechanisms and excessive glutamate release in amyotrophic lateral sclerosis
33. Synaptic mechanisms of the abnormal release of glutamate in the spinal cord of pre-symptomatic SOD1G93A mice
34. Leucine‐rich repeat kinase 2 phosphorylation on synapsin I regulates glutamate release at pre‐synaptic sites
35. Presynaptic L-Type Ca2+ Channels Increase Glutamate Release Probability and Excitatory Strength in the Hippocampus during Chronic Neuroinflammation.
36. Synapsin I and Synapsin II regulate neurogenesis in the dentate gyrus of adult mice
37. A novel topology of proline-rich transmembrane protein 2 (PRRT2): Hints for an intracellular function at the synapse.
38. Additional file 1: of LRRK2 phosphorylates pre-synaptic N-ethylmaleimide sensitive fusion (NSF) protein enhancing its ATPase activity and SNARE complex disassembling rate
39. The LRRK2 G2385R variant is a partial loss-of-function mutation that affects synaptic vesicle trafficking through altered protein interactions
40. Altered mechanisms underlying the abnormal glutamate release in amyotrophic lateral sclerosis at a pre-symptomatic stage of the disease
41. Synaptic mechanisms underlying the excessive and precocious glutamate release in the spinal cord of sod1g93a mice
42. The protein kinase LRRK2 is involved in neurotransmitter release by interacting and phosphorylating synapsin I in nerve terminals
43. LRRK2 INTERACTS WITH SYNAPTIC VESICLES AT THE PRESYNAPTIC SITE
44. Synaptic mechanisms underlying the excessive and precocious glutamate release in the spinal cord of SOD1G93A experimental mice
45. PHOSPHORYLATION OF SYNAPSIN I BY CYCLIN-DEPENDENT KINASE-5 REGULATES SYNAPTIC VESICLE SPATIAL DISTRIBUTION AND RECRUITMENT TO RECYCLING POOL AT HIPPOCAMPAL SYNAPSES
46. LRRK2 BINDS TO NEURONAL VESICLES THROUGH PROTEIN INTERACTIONS MEDIATED BY ITS C-TERMINAL WD40 DOMAIN
47. PRRT2 Is a Key Component of the Ca 2+ -Dependent Neurotransmitter Release Machinery
48. A Novel Topology of Proline-rich Transmembrane Protein 2 (PRRT2)
49. LRRK2 phosphorylates pre-synaptic N-ethylmaleimide sensitive fusion (NSF) protein enhancing its ATPase activity and SNARE complex disassembling rate
50. Synapsins Are Downstream Players of the BDNF-Mediated Axonal Growth
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