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4. Legumain is upregulated in acute cardiovascular events and associated with improved outcome - potentially related to anti-inflammatory effects on macrophages

5. Cholangiocytes modulate CD100 expression in the liver and facilitate pathogenic Th17 differentiation

12. LIGHT/TNFSF14 is increased in patients with type 2 diabetes mellitus and promotes islet cell dysfunction and endothelial cell inflammation in vitro

13. Cholangiocytes cleave surface CD100 from biliary infiltrating T cells and mediate pathogenic Th17 differentiation

18. Secreted Wnt antagonists in scrub typhus

22. Triglyceride-rich [HDL.sub.3] from patients with familial hypercholesterolemia are less able to inhibit cytokine release or to promote cholesterol efflux

23. Gut Microbiota-Dependent Trimethylamine N-Oxide Associates With Inflammation in Common Variable Immunodeficiency

28. Neutrophil extracellular traps drive inflammatory pathogenesis in malaria

34. Additional file 1: of Soluble markers of neutrophil, T-cell and monocyte activation are associated with disease severity and parasitemia in falciparum malaria

35. Increased Levels of Lectin‐Like Oxidized Low‐Density Lipoprotein Receptor‐1 in Ischemic Stroke and Transient Ischemic Attack

36. Interleukin 27 is increased in carotid atherosclerosis and promotes NLRP3 inflammasome activation

37. Rickettsia conorii is a potent complement activator in vivo and combined inhibition of complement and CD14 is required for attenuation of the cytokine response ex vivo

38. Complement Activation Correlates With Disease Severity and Contributes to Cytokine Responses inPlasmodium falciparumMalaria

40. Increased endothelial and macrophage markers are associated with disease severity and mortality in scrub typhus

43. Cytokine Network in Scrub Typhus: High Levels of Interleukin-8 Are Associated with Disease Severity and Mortality

44. A Complex Interaction between Rickettsia conorii and Dickkopf-1 – Potential Role in Immune Evasion Mechanisms in Endothelial Cells

50. Dickkopf-1 Enhances Inflammatory Interaction Between Platelets and Endothelial Cells and Shows Increased Expression in Atherosclerosis

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