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642 results on '"Pagonabarraga, Javier"'

1. Tau seeding activity in skin biopsy differentiates tauopathies from synucleinopathies

Author

Dellarole, Ilaria Linda, Vacchi, Elena, Ruiz-Barrio, Inigo, Pinton, Sandra, Raimondi, Andrea, Rossi, Stefania, Morandi, Sara, Bianco, Giovanni, Begum Bacinoglu, Merve, Lombardo, Annalisa, Celauro, Luigi, Staedler, Claudio, Galati, Salvatore, Pagonabarraga, Javier, Kulisevsky, Jaime, Legname, Giuseppe, Gobbi, Claudio, Kaelin-Lang, Alain, Moda, Fabio, and Melli, Giorgia

Published
2024
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4. Measuring cognitive impairment and monitoring cognitive decline in Huntington’s disease: a comparison of assessment instruments

Author

Horta-Barba, Andrea, Martinez-Horta, Saul, Pérez-Pérez, Jesús, Puig-Davi, Arnau, de Lucia, Natascia, de Michele, Giuseppe, Salvatore, Elena, Kehrer, Stefanie, Priller, Josef, Migliore, Simone, Squitieri, Ferdinando, Castaldo, Anna, Mariotti, Caterina, Mañanes, Veronica, Lopez-Sendon, Jose Luis, Rodriguez, Noelia, Martinez-Descals, Asunción, Júlio, Filipa, Januário, Cristina, Delussi, Marianna, de Tommaso, Marina, Noguera, Sandra, Ruiz-Idiago, Jesús, Sitek, Emilia J., Wallner, Renata, Nuzzi, Angela, Pagonabarraga, Javier, and Kulisevsky, Jaime

Published
2023
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5. Level I PD‐MCI Using Global Cognitive Tests and the Risk for Parkinson's Disease Dementia

Author

Boel, Judith A, de Bie, Rob MA, Schmand, Ben A, Dalrymple‐Alford, John C, Marras, Connie, Adler, Charles H, Goldman, Jennifer G, Tröster, Alexander I, Burn, David J, Litvan, Irene, Geurtsen, Gert J, Bernard, Bryan, Stebbins, Glenn, Filoteo, J Vincent, Weintraub, Daniel, Caviness, John N, Belden, Christine, Zabetian, Cyrus P, Cholerton, Brenna A, Huang, Xuemei, Eslinger, Paul J, Leverenz, James B, Duff‐Canning, Sarah, Farrer, Matt, Anderson, Tim J, Myall, Daniel J, Naismith, Sharon L, Lewis, Simon JG, Halliday, Glenda M, Wu, Ruey‐Meei, Williams‐Gray, Caroline H, Breen, David P, Barker, Roger A, Yarnall, Alison J, Klein, Martin, Mollenhauer, Brit, Trenkwalder, Claudia, Kulisevsky, Jaime, Pagonabarraga, Javier, Gasca‐Salas, Carmen, Rodriguez‐Oroz, Maria C, Junque, Carme, Segura, Barbara, Barone, Paolo, Santangelo, Gabriella, Cammisuli, Davide M, Biundo, Roberta, Antonini, Angelo, Weis, Luca, Pedersen, Kenn Freddy, and Alves, Guido

Subjects
Acquired Cognitive Impairment, Neurodegenerative, Neurosciences, Parkinson's Disease, Dementia, Clinical Research, Aging, Brain Disorders, Neurological, dementia, mild cognitive impairment, global cognitive tests, Parkinson's disease, diagnostic accuracy, MDS Study Group Mild Cognitive Impairment in Parkinson's Disease
Abstract

BackgroundThe criteria for PD-MCI allow the use of global cognitive tests. Their predictive value for conversion from PD-MCI to PDD, especially compared to comprehensive neuropsychological assessment, is unknown.MethodsThe MDS PD-MCI Study Group combined four datasets containing global cognitive tests as well as a comprehensive neuropsychological assessment to define PD-MCI (n = 467). Risk for developing PDD was examined using a Cox model. Global cognitive tests were compared to neuropsychological test batteries (Level I&II) in determining risk for PDD.ResultsPD-MCI based on a global cognitive test (MMSE or MoCA) increases the hazard for developing PDD (respectively HR = 2.57, P = 0.001; HR = 4.14, P =

Published
2022

7. Neuroanatomical and cognitive correlates of visual hallucinations in Parkinson’s disease and dementia with Lewy bodies: Voxel-based morphometry and neuropsychological meta-analysis

Author

Pezzoli, Stefania, Sánchez-Valle, Raquel, Solanes, Aleix, Kempton, Matthew J, Bandmann, Oliver, Shin, Jae Il, Cagnin, Annachiara, Goldman, Jennifer G, Merkitch, Doug, Firbank, Michael J, Taylor, John-Paul, Pagonabarraga, Javier, Kulisevsky, Jaime, Blanc, Frederic, Verdolini, Norma, Venneri, Annalena, and Radua, Joaquim

Subjects
Biomedical and Clinical Sciences, Health Sciences, Mental Health, Neurodegenerative, Alzheimer's Disease including Alzheimer's Disease Related Dementias (AD/ADRD), Dementia, Acquired Cognitive Impairment, Brain Disorders, Parkinson's Disease, Neurosciences, Aging, 2.1 Biological and endogenous factors, Aetiology, Neurological, Cognition, Hallucinations, Humans, Lewy Body Disease, Magnetic Resonance Imaging, Neuropsychological Tests, Parkinson Disease, Lewy body disease, Visual hallucinations, Dementia with Lewy bodies, Meta-analysis, VBM, MRI, Grey matter volume, Neuropsychology, Memory, Parkinson's disease, Parkinson’s disease, Medical and Health Sciences, Psychology and Cognitive Sciences, Behavioral Science & Comparative Psychology, Biomedical and clinical sciences, Health sciences
Abstract

Visual hallucinations (VH) are common in Parkinson's disease and dementia with Lewy bodies, two forms of Lewy body disease (LBD), but the neural substrates and mechanisms involved are still unclear. We conducted meta-analyses of voxel-based morphometry (VBM) and neuropsychological studies investigating the neuroanatomical and cognitive correlates of VH in LBD. For VBM (12 studies), we used Seed-based d Mapping with Permutation of Subject Images (SDM-PSI), including statistical parametric maps for 50% of the studies. For neuropsychology (35 studies), we used MetaNSUE to consider non-statistically significant unreported effects. VH were associated with smaller grey matter volume in occipital, frontal, occipitotemporal, and parietal areas (peak Hedges' g -0.34 to -0.49). In patients with Parkinson's disease without dementia, VH were associated with lower verbal immediate memory performance (Hedges' g -0.52). Both results survived correction for multiple comparisons. Abnormalities in these brain regions might reflect dysfunctions in brain networks sustaining visuoperceptive, attention, and executive abilities, with the latter also being at the basis of poor immediate memory performance.

Published
2021

9. Understanding visual hallucinations: A new synthesis

Author

Collerton, Daniel, Barnes, James, Diederich, Nico J., Dudley, Rob, ffytche, Dominic, Friston, Karl, Goetz, Christopher G., Goldman, Jennifer G., Jardri, Renaud, Kulisevsky, Jaime, Lewis, Simon J.G., Nara, Shigetoshi, O’Callaghan, Claire, Onofrj, Marco, Pagonabarraga, Javier, Parr, Thomas, Shine, James M., Stebbins, Glenn, Taylor, John-Paul, Tsuda, Ichiro, and Weil, Rimona S.

Published
2023
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17. Tipping the scales: how clinical assessment shapes the neural correlates of Parkinson’s disease mild cognitive impairment

Author

Aracil-Bolaños, Ignacio, Sampedro, Frederic, Marín-Lahoz, Juan, Horta-Barba, Andrea, Martínez-Horta, Saül, Gónzalez-de-Echávarri, José María, Pérez-Pérez, Jesús, Bejr-Kasem, Helena, Pascual-Sedano, Berta, Botí, Mariángeles, Campolongo, Antonia, Izquierdo, Cristina, Gironell, Alexandre, Gómez-Ansón, Beatriz, Kulisevsky, Jaime, and Pagonabarraga, Javier

Published
2022
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19. Smoking is associated with age at disease onset in Parkinson's disease

Author

Adarmes-Gómez, Astrid D., Aguilar, Miquel, Alvarez, Ignacio, Barrero, Francisco Javier, Bergareche Yarza, Jesús Alberto, Bonilla-Toribio, Marta, Botía, Juan A., Boungiorno, María Teresa, Buiza-Rueda, Dolores, Cámara, Ana, Carrillo, Fátima, Cerdan, Debora, Clarimón, Jordi, Compta, Yaroslau, Diez-Fairen, Monica, Dols-Icardo, Oriol, de Fabregues, Oriol, Cartagena, Pilar Sanz, Duarte, Jacinto, Duran, Raquel, Escamilla-Sevilla, Francisco, Ezquerra, Mario, Feliz, Cici, Fernández-Santiago, Rubén, Fernández, Manel, García-Ruiz, Pedro, Gómez-Garre, Pilar, Gomez Heredia, Maria Jose, Gonzalez-Aramburu, Isabel, Gorostidi, Ana, Hoenicka, Janet, Infante, Jon, Jesús, Silvia, Jimenez-Escrig, Adriano, Kulisevsky, Jaime, Labrador-Espinosa, Miguel A., Lopez-Sendon, Jose Luis, López de Munain, Adolfo, Macias-Garcia, Daniel, Martínez-Torres, Irene, Marín, Juan, Marti, Maria Jose, Martínez-Castrillo, Juan Carlos, Álvarez-Santullano, Marina Mata, Mínguez-Castellanos, Adolfo, Mir, Pablo, Rezola, Elisabet Mondragon, Muñoz, Esteban, Pagonabarraga, Javier, Pastor, Pau, Errazquin, Francisco Perez, Periñán, Maria Teresa, Ruiz-Martínez, Javier, Ruz, Clara, Rodriguez, Antonio Sanchez, Sierra, María, Tabernero, Cesar, Tartari, Juan Pablo, Tolosa, Eduard, Valldeoriola, Francesc, Vela, Lydia, Vives, Francisco, Pascual-Sedano, Berta, Hernández-Vara, Jorge, Rolán, Dolores Vilas, Bandrés-Ciga, Sara, Rosas, Irene, Morís, Germán, Coto, Eliecer, Blázquez-Estrada, Marta, Suárez, Esther, García-Fernández, Ciara, Martínez, Carmen, Herrera, Israel Duarte, Pérez-Oliveira, Sergio, Álvarez, Victoria, and Menéndez-González, Manuel

Published
2022
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20. Prediction of Cognitive Heterogeneity in Parkinson's Disease: A 4‐Year Longitudinal Study Using Clinical, Neuroimaging, Biological and Electrophysiological Biomarkers.

Author

Puig‐Davi, Arnau, Martinez‐Horta, Saul, Pérez‐Carasol, Laura, Horta‐Barba, Andrea, Ruiz‐Barrio, Iñigo, Aracil‐Bolaños, Ignacio, Pérez‐González, Rocío, Rivas‐Asensio, Elisa, Sampedro, Frederic, Campolongo, Antonia, Pagonabarraga, Javier, and Kulisevsky, Jaime

Subjects
MILD cognitive impairment, MAGNETIC resonance imaging, PARKINSON'S disease, POWER density, COGNITION disorders
Abstract

Objective: Cognitive impairment in Parkinson's disease (PD) can show a very heterogeneous trajectory among patients. Here, we explored the mechanisms involved in the expression and prediction of different cognitive phenotypes over 4 years. Methods: In 2 independent cohorts (total n = 475), we performed a cluster analysis to identify trajectories of cognitive progression. Baseline and longitudinal level II neuropsychological assessments were conducted, and baseline structural magnetic resonance imaging, resting electroencephalogram and neurofilament light chain plasma quantification were carried out. Linear mixed‐effects models were used to study longitudinal changes. Risk of mild cognitive impairment and dementia were estimated using multivariable hazard regression. Spectral power density from the electroencephalogram at baseline and source localization were computed. Results: Two cognitive trajectories were identified. Cluster 1 presented stability (PD‐Stable) over time, whereas cluster 2 showed progressive cognitive decline (PD‐Progressors). The PD‐Progressors group showed an increased risk for evolving to PD mild cognitive impairment (HR 2.09; 95% CI 1.11–3.95) and a marked risk for dementia (HR 4.87; 95% CI 1.34–17.76), associated with progressive worsening in posterior‐cortical‐dependent cognitive processes. Both clusters showed equivalent clinical and sociodemographic characteristics, structural magnetic resonance imaging, and neurofilament light chain levels at baseline. Conversely, the PD‐Progressors group showed a fronto‐temporo‐occipital and parietal slow‐wave power density increase, that was in turn related to worsening at 2 and 4 years of follow‐up in different cognitive measures. Interpretation: In the absence of differences in baseline cognitive function and typical markers of neurodegeneration, the further development of an aggressive cognitive decline in PD is associated with increased slow‐wave power density and with a different profile of worsening in several posterior‐cortical‐dependent tasks. ANN NEUROL 2024;96:981–993 [ABSTRACT FROM AUTHOR]

Published
2024
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30. Baseline Large‐Scale Network Dynamics Associated with Disease Progression in Huntington's Disease

Author

Aracil‐Bolaños, Ignacio, primary, Pérez‐Pérez, Jesús, additional, Martínez‐Horta, Saül, additional, Horta‐Barba, Andrea, additional, Puig‐Davi, Arnau, additional, García‐Cornet, Júlia, additional, Olmedo‐Saura, Gonzalo, additional, Campolongo, Antonia, additional, Pagonabarraga, Javier, additional, and Kulisevsky, Jaime, additional

Published
2023
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32. Subclinical affective and cognitive fluctuations in Parkinson's disease: a randomized double-blind double-dummy study of Oral vs. Intrajejunal Levodopa

Author

Kulisevsky, Jaime, Bejr-Kasem, Helena, Martinez-Horta, Saul, Horta-Barba, Andrea, Pascual-Sedano, Berta, Campolongo, Antonia, Marín-Lahoz, Juan, Aracil-Bolaños, Ignacio, Pérez-Pérez, Jesús, Izquierdo-Barrionuevo, Cristina, de Fàbregues, Oriol, Puente, Victor, Crespo-Cuevas, Ane, Calopa, Matilde, and Pagonabarraga, Javier

Published
2020
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35. Cerebrospinal fluid cytokines in multiple system atrophy: A cross-sectional Catalan MSA registry study

Author

Pagonabarraga, Javier, Valldeoriola, Francesc, Hernández-Vara, Jorge, Classen, Serge Jauma, Puente, Victor, Pont, Claustre, Caballol, Núria, Tolosa, Eduardo, Bayes, Angels, Campdelacreu, Jaume, de Fàbregues, Oriol, Ávila, Asunción, Calopa, Matilde, Gaig, Carles, Fabregat, Neus, Pastor, Pau, Aguilar, Miquel, Pujol, Montserrat, Sánchez, Almudena, Planellas, Lluís, Ezquerra, Mario, Fernández-Santiago, Rubén, Botta, Teresa, Tartaglia, Gian, Compta, Yaroslau, Dias, Sara P., Giraldo, Darly M., Pérez-Soriano, Alexandra, Muñoz, Esteban, Saura, Josep, Fernández, Manel, Bravo, Paloma, Cámara, Ana, Pulido-Salgado, Marta, Painous, Cèlia, Ríos, José, and Martí, María José

Published
2019
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37. The Sant Pau Initiative on Neurodegeneration (SPIN) cohort: A data set for biomarker discovery and validation in neurodegenerative disorders

Author

Alcolea, Daniel, Clarimón, Jordi, Carmona-Iragui, María, Illán-Gala, Ignacio, Morenas-Rodríguez, Estrella, Barroeta, Isabel, Ribosa-Nogué, Roser, Sala, Isabel, Sánchez-Saudinós, M. Belén, Videla, Laura, Subirana, Andrea, Benejam, Bessy, Valldeneu, Sílvia, Fernández, Susana, Estellés, Teresa, Altuna, Miren, Santos-Santos, Miguel, García-Losada, Lídia, Bejanin, Alexandre, Pegueroles, Jordi, Montal, Víctor, Vilaplana, Eduard, Belbin, Olivia, Dols-Icardo, Oriol, Sirisi, Sònia, Querol-Vilaseca, Marta, Cervera-Carles, Laura, Muñoz, Laia, Núñez, Raúl, Torres, Soraya, Camacho, M. Valle, Carrió, Ignasi, Giménez, Sandra, Delaby, Constance, Rojas-Garcia, Ricard, Turon-Sans, Janina, Pagonabarraga, Javier, Jiménez, Amanda, Blesa, Rafael, Fortea, Juan, and Lleó, Alberto

Published
2019
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38. Predicting Disability in Progressive Supranuclear Palsy Using Bedside Frontal‐Lobe Signs.

Author

Ruiz‐Barrio, Iñigo, Horta‐Barba, Andrea, Aracil‐Bolaños, Ignacio, Martinez‐Horta, Saül, Kulisevsky, Jaime, and Pagonabarraga, Javier

Subjects
PROGNOSIS, DISABILITIES, BRAIN cortical thickness, SYMPTOMS, PROGRESSIVE supranuclear palsy, FRONTAL lobe, FRONTAL lobe diseases
Abstract

Background: Frontal lobe signs in progressive supranuclear palsy (PSP) are prevalent and occur early in the disease. Although they are recognized in clinical practice, studies are needed to systematically investigate them for an in‐depth understanding of the neurological substrate and their potential prognostic implications in the disease. Objectives: To study the predictive role of frontal lobe signs in PSP, as well as to describe their neuropsychological and anatomical correlations. Methods: Nine recognized signs of frontal lobe dysfunction were assessed in 61 patients with PSP. Those signs able to predict PSP Rating Scale (PSPRS) score at baseline were selected, a survival analysis was performed and associations with neuropsychological tests and cortical thickness parameters in brain MRI were studied. Results: Grasping, anosognosia and orobuccal apraxia predicted the PSPRS score independently of age, gender, clinical subtype and disease duration. The occurrence of groping in the first 4 years could be a predictor of survival. Grasping and anosognosia were associated with frontal cognitive dysfunction, whereas orobuccal apraxia and groping were related to a more widespread cognitive impairment, involving temporal–parietal areas. Presence of groping showed an extensive cortical atrophy, with predominant prefrontal, temporal and superior parietal cortical thinning. Conclusions: Grasping, groping, anosognosia and orobuccal apraxia are easily evaluable bedside clinical signs that reflect distinct stages of disease progression. Grasping, anosognosia and orobuccal apraxia predict disease disability in patients with PSP, and early onset groping could be a survival predictor. [ABSTRACT FROM AUTHOR]

Published
2024
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39. Cerebrospinal fluid levels of coenzyme Q10 are reduced in multiple system atrophy

Author

Ávila, Asunción, Bayés, Àngels, Botta-Orfila, Teresa, Caballol, Núria, Calopa, Matilde, Campdelacreu, Jaume, Ezquerra, Mario, de Fàbregues, Oriol, Fernández-Santiago, Rubén, Hernández-Vara, Jorge, Jaumà, Serge, Marchese, Domenica, Pagonabarraga, Javier, Pastor, Pau, Planellas, Lluís, Pont-Sunyer, Claustre, Puente, Víctor, Pujol, Montserrat, Saura, Josep, Tartaglia, Gian Gaetano, Tolosa, Eduard, Valldeoriola, Francesc, Compta, Yaroslau, Giraldo, Darly M., Muñoz, Esteban, Antonelli, Francesca, Fernández, Manel, Bravo, Paloma, Soto, Marta, Cámara, Ana, Torres, Ferran, and Martí, María José

Published
2018
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42. Baseline Large‐Scale Network Dynamics Associated with Disease Progression in Huntington's Disease.

Author

Aracil‐Bolaños, Ignacio, Pérez‐Pérez, Jesús, Martínez‐Horta, Saül, Horta‐Barba, Andrea, Puig‐Davi, Arnau, García‐Cornet, Júlia, Olmedo‐Saura, Gonzalo, Campolongo, Antonia, Pagonabarraga, Javier, and Kulisevsky, Jaime

Abstract

Background: Huntington's disease (HD) is a genetically determined disease with motor, cognitive, and neuropsychiatric disorders. However, the links between clinical progression and disruptions to dynamics in motor and cognitive large‐scale networks are not well established. Objective: To investigate changes in dynamic and static large‐scale networks using an established tool of disease progression in Huntington's disease, the composite Unified Huntington's Disease Rating Scale (cUHDRS). Methods: Sixty‐four mutation carriers were included. Static and dynamic baseline functional connectivity as well as topological features were correlated to 2‐year follow‐up clinical assessments using the cUHDRS. Results: Decline in cUHDRS scores was associated with higher connectivity between frontal default‐mode and motor networks, whereas higher connectivity in posterior, mainly visuospatial regions was associated with a smaller decline in cUHDRS scores. Conclusions: Structural disruptions in HD were evident both in posterior parietal/occipital and frontal motor regions, with reciprocal increases in functional connectivity. However, although higher visuospatial network connectivity was tied to a smaller cUHDRS decline, increased motor and frontal default‐mode connections were linked to a larger cUHDRS decreases. Therefore, divergent functional compensation mechanisms might be at play in the clinical evolution of HD. [ABSTRACT FROM AUTHOR]

Published
2024
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45. Functional neurological disorders after COVID-19 and SARS-CoV-2 vaccines: a national multicentre observational study

Author

Alonso-Canovas, Araceli, primary, Kurtis, Monica M, additional, Gomez-Mayordomo, Victor, additional, Macías-García, Daniel, additional, Gutiérrez Viedma, Álvaro, additional, Mondragón Rezola, Elisabet, additional, Pagonabarraga, Javier, additional, Aranzabal Orgaz, Lidia, additional, Masjuan, Jaime, additional, Martinez-Castrillo, Juan Carlos, additional, and Pareés, Isabel, additional

Published
2023
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46. Functional neurological disorders after COVID-19 and SARS-CoV-2 vaccines: a national multicentre observational study

Author

Alonso-Canovas, Araceli, Kurtis, Monica M, Gomez-Mayordomo, Victor, Macías García, Daniel, Gutiérrez Viedma, Álvaro, Mondragón Rezola, Elisabet, Pagonabarraga, Javier, Aranzabal Orgaz, Lidia, Masjuan, Jaime, Martinez-Castrillo, Juan Carlos, Pareés, Isabel, Alonso-Canovas, Araceli, Kurtis, Monica M, Gomez-Mayordomo, Victor, Macías García, Daniel, Gutiérrez Viedma, Álvaro, Mondragón Rezola, Elisabet, Pagonabarraga, Javier, Aranzabal Orgaz, Lidia, Masjuan, Jaime, Martinez-Castrillo, Juan Carlos, and Pareés, Isabel

Abstract

Functional neurological disorders (FNDs) are a common cause of neurology consultations.1 Dissociative seizures, motor and cognitive disorders are the main phenotypes. Diagnosis is made on positive terms: signs of inconsistency, incongruence and variability of physical signs with attention on clinical examination.1 Abnormal emotional processing and expectations are involved in the genesis and perpetuation of FND.1 From the onset of SARS-CoV-2 pandemic, world population has been affected by high levels of stress, uncertainty and misleading information, with a potential impact on mental health. Different studies have ascertained an increase of FND consultations (threefold in an emergency department).2 On the development of SARS-CoV-2 vaccines, several cases of FND following vaccination were published, as well as an official warning from the Functional Neurological Society.3 Post-COVID-19 symptoms (known as Long-COVID-19) have also become a frequent reason for neurology consultation.4 Somatic symptom disorder may be common in these patients, and socioeconomic implications are vast.5 Our experience is that a proportion of patients with FND describe an association with COVID-19 infection/vaccination. Here, we report a cohort of patients with FND for whom COVID-19 or SARS-CoV-2 vaccines were the main precipitant factors.

Published
2023

50. Parkinson's Disease‐Cognitive Rating Scale ( PD‐CRS ): normative data and mild cognitive impairment assessment in Brazil

Author

Brandão, Pedro Renato de Paula, primary, Pereira, Danilo Assis, additional, Grippe, Talyta Cortez, additional, Bispo, Diógenes Diego, additional, Maluf, Fernando Bisinoto, additional, Yunes, Márcia Pereira, additional, Nunes Filho, Gilberto, additional, Alves, Cândida Helena Lopes, additional, Pagonabarraga, Javier, additional, Kulisevsky, Jaime, additional, da Costa, Andressa Mátos Luiz, additional, Serafim, Cristiano Franco do Val, additional, Ferreira, Ana Carolina da Bouza, additional, Bastos, Arthur de Melo Monteiro, additional, Belchior, Ana Clara Fernandes, additional, de Almeida, Beatriz Luna Coutinho, additional, de Almeida e Castro, Brenda Macedo, additional, Matos, Matheus Sampaio, additional, de Matos, Rodrigo Cardoso, additional, Rios, Gabriel de Almeida, additional, Carneiro, Lorena Oliveira, additional, da Mota, Bianca Christie Costa, additional, Castro, Lucas Ernesto do Rêgo, additional, Rocha, Vitória Luísa Silveira, additional, Tavares, Maria Clotilde Henriques, additional, and Cardoso, Francisco, additional

Published
2023
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