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2. The European reference network for metabolic diseases (MetabERN) clinical pathway recommendations for Pompe disease (acid maltase deficiency, glycogen storage disease type II)

5. Loss of the batten disease protein CLN3 leads to mis-trafficking of M6PR and defective autophagic-lysosomal reformation

6. RagD auto-activating mutations impair MiT/TFE activity in kidney tubulopathy and cardiomyopathy syndrome

7. How dietary advanced glycation end products could facilitate the occurrence of food allergy

8. De novo missense variants in phosphatidylinositol kinase PIP5KIγ underlie a neurodevelopmental syndrome associated with altered phosphoinositide signaling

9. The Mucopolysaccharidoses

11. Cipaglucosidase alfa plus miglustat: linking mechanism of action to clinical outcomes in late-onset Pompe disease.

12. Start, switch and stop (triple‐S) criteria for enzyme replacement therapy of late‐onset Pompe disease: European Pompe Consortium recommendation update 2024.

13. Safety and efficacy of cipaglucosidase alfa plus miglustat versus alglucosidase alfa plus placebo in late-onset Pompe disease (PROPEL): an international, randomised, double-blind, parallel-group, phase 3 trial

15. Milder presentation of TELO2-related syndrome in two sisters homozygous for the p.Arg609His pathogenic variant

16. Start, switch and stop (triple-S) criteria for enzyme replacement therapy of late-onset Pompe disease:European Pompe Consortium recommendation update 2024

17. Adolescent gender dysphoria management: position paper from the Italian Academy of Pediatrics, the Italian Society of Pediatrics, the Italian Society for Pediatric Endocrinology and Diabetes, the Italian Society of Adolescent Medicine and the Italian Society of Child and Adolescent Neuropsychiatry

18. PRDX1 gene-related epi-cblC disease is a common type of inborn error of cobalamin metabolism with mono- or bi-allelic MMACHC epimutations

23. Wilson disease protein ATP7B utilizes lysosomal exocytosis to maintain copper homeostasis.

26. microRNAs as biomarkers in Pompe disease

28. De novo missense variants in phosphatidylinositol kinase PIP5KIγ underlie a neurodevelopmental syndrome associated with altered phosphoinositide signaling

30. Vitamin E Improves Clinical Outcome of Patients Affected by Glycogen Storage Disease Type Ib

32. Common and Novel TMEM70 Mutations in a Cohort of Italian Patients with Mitochondrial Encephalocardiomyopathy

35. Contribution of Genetic Test to Early Diagnosis of Methylenetetrahydrofolate Reductase (MTHFR) Deficiency: The Experience of a Reference Center in Southern Italy

39. Additional file 2 of Mitochondrial reprogramming in peripheral blood mononuclear cells of patients with glycogen storage disease type Ia

40. Additional file 5 of Mitochondrial reprogramming in peripheral blood mononuclear cells of patients with glycogen storage disease type Ia

41. Additional file 1 of Mitochondrial reprogramming in peripheral blood mononuclear cells of patients with glycogen storage disease type Ia

42. Additional file 4 of Mitochondrial reprogramming in peripheral blood mononuclear cells of patients with glycogen storage disease type Ia

43. Additional file 3 of Mitochondrial reprogramming in peripheral blood mononuclear cells of patients with glycogen storage disease type Ia

44. Additional file 7 of Mitochondrial reprogramming in peripheral blood mononuclear cells of patients with glycogen storage disease type Ia

45. Inborn Errors of Metabolism

46. Long-term monitoring for short/branched-chain acyl-CoA dehydrogenase deficiency: A single-center 4-year experience and open issues

49. Loss of the batten disease protein CLN3 leads to mis-trafficking of M6PR and defective autophagic-lysosomal reformation.

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