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87 results on '"Park, Geun-Ho"'

4. Evolution Behavior of Non-metallic Inclusions in High-Al or Si-Alloyed Steel Reacting With CaO–Al2O3–FeO–SiO2–MgO Slag.

Author

Kim, Dong Woon, Kim, Tae Sung, Park, Geun Ho, and Park, Joo Hyun

Subjects
INTERFACIAL reactions, STEEL, SLAG, ALUMINUM-magnesium alloys, PARTICLE analysis, POPULATION density, SPINEL
Abstract

The effect of the reaction between alloy elements and slag on the evolution behavior of non-metallic inclusions in high-Al or Si-alloyed steel melts was investigated at 1873 K (1600 °C). Aluminum (1.5 mass pct) added to the molten steel produces an alumina-rich inclusion by reaction with oxygen, and it reduces FeO by the interfacial reaction with the CaO–Al2O3–FeO–SiO2–MgO slag, resulting in an increase of the alumina content of the slag. On the other hand, silicon (4.0 mass pct) added to the molten steel increases the silica content of the slag by the interfacial reactions with the slag and causing aluminum pick-up from the liquid slag into the molten steel, which changed the existing silica-rich inclusion into aluminum-rich inclusions. The addition of each of the two alloying elements produced alumina-rich inclusions, and the two series of existing alumina inclusions were modified into MgAl2O4 spinel inclusions by picking up magnesium from the slag into the molten steel. Through a population density function (PDF) analysis of inclusion particles, we confirmed that inclusion growth occurred by diffusion of magnesium and aluminum in steel with 1.5 pct Al or 4.0 pct Si. The supersaturation degree for alumina inclusions in the 1.5 pct Al-added steel and 4.0 pct Si-added steel continued to decrease as the reaction time increased. As a result, dendritic or spherical alumina inclusions were observed in both series at 1 minute of alloying, and small, independently present polygonal-shaped MgAl2O4 spinel inclusions were mainly observed at 30 minutes of alloying. [ABSTRACT FROM AUTHOR]

Published
2024
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20. Anti-interleukin-33 Reduces Ovalbumin-Induced Nephrotoxicity and Expression of Kidney Injury Molecule-1

Author

Park, Geun Ho, Shinn, Helen Ki, Kang, Ju-Hee, Na, Won Ju, Kim, Young Hyo, and Park, Chang-Shin

Subjects
0301 basic medicine, medicine.medical_specialty, Ovalbumin, Urology, lcsh:RC870-923, Endothelial NOS, Nephrotoxicity, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, Kidney Injury Molecule-1, Internal medicine, medicine, Blood urea nitrogen, Creatinine, biology, Fundamental Science for Neurourology, business.industry, Acute kidney injury, Acute Kidney Injury, Interleukin-33, lcsh:Diseases of the genitourinary system. Urology, medicine.disease, Interleukin 33, Nitric oxide synthase, 030104 developmental biology, Endocrinology, Neurology, chemistry, Immunology, biology.protein, Original Article, Neurology (clinical), business, 030215 immunology
Abstract

Purpose To evaluate the effect of anti-interleukin-33 (anti-IL-33) on a mouse model of ovalbumin (OVA)-induced acute kidney injury (AKI). Methods Twenty-four female BALB/c mice were assigned to 4 groups: group A (control, n=6) was administered sterile saline intraperitoneally (i.p.) and intranasally (i.n.); group B (allergic, n=6) was administered i.p./i.n. OVA challenge; group C (null treatment, n=6) was administered control IgG i.p. before OVA challenge; and group D (anti-IL-33, n=6) was pretreated with 3.6 µg of anti-IL-33 i.p. before every OVA challenge. The following were evaluated after sacrifice: serum blood urea nitrogen and creatinine levels, Kidney injury molecule-1 gene (Kim-1) and protein (KIM-1) expression in renal parenchyma, and expression of cyclooxygenase-2 (COX-2), inducible nitric oxide synthase (iNOS), phosphorylated endothelial NOS (p-eNOS), and phosphorylated AMP kinase (p-AMPK) proteins in renal parenchyma. Results After OVA injection and intranasal challenge, mice in groups B and C showed significant increases in the expression of Kim-1 at both the mRNA and protein levels. After anti-IL-33 treatment, mice in group D showed significant Kim-1 down-regulation at the mRNA and protein levels. Group D also showed significantly lower COX-2 protein expression, marginally lesser iNOS expression than groups B and C, and p-eNOS and p-AMPK expression at baseline levels. Conclusions Kim-1 could be a useful marker for detecting early-stage renal injury in mouse models of OVA-induced AKI. Further, anti-IL-33 might have beneficial effects on these mouse models.

Published
2016

36. Evaluation of the acaricidal activity of 63 commercialized pesticides against Haemaphysalis longicornis (Acari: Ixodidae).

Author

Park, Geun‐Ho, Kim, Hyun Kyung, Lee, Wook‐Gyo, Cho, Shin‐Hyeong, and Kim, Gil‐Hah

Subjects
*ACARICIDES, *PYRETHROIDS, *PESTICIDES, *IXODIDAE, *MITES, *CARBAMATES, *GRASSLANDS
Abstract

The acaricidal activity and residual effects of 63 commercialized pesticides against the Cheongju strain of Haemaphysalis longicornis in Korea were examined. Twenty‐two pesticides (4 carbamates, 5 organophosphates, 10 pyrethroids, 1 amitraz, 1 diamide, and 1 unclassified pesticide) caused greater than 80% mortality in H. longicornis adults under laboratory conditions using the spray method. These 22 pesticides were used to treat grassland under field conditions for investigation of the residual effects, and 100% mortality in H. longicornis adults was observed with all the carbamates (carbosulfan, benfuracarb, fenobucarb, and carbaryl) and γ‐cyhalothrin after 3 days of pesticide treatment. γ‐Cyhalothrin exhibited 56.7% mortality after 10 days of treatment, which was the longest residual effect of treated grass on H. longicornis adults. With regard to the residual effects on H. longicornis under field conditions, most of the 22 pesticides exhibited higher mortality in nymphs than in adults. In particular, benfuracarb exhibited 96.7% acaricidal activity until 10 days after treatment. These results indicate that carbamates are highly likely to be available under field conditions and, based on this preliminary data, could be used for the control of H. longicornis adults and nymphs. [ABSTRACT FROM AUTHOR]

Published
2019
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